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anti-FLT4 (FLT4) 抗体产品概述

Full name:
anti-Fms-Related Tyrosine Kinase 4 抗体 (FLT4)
在www.antibodies-online.cn可供245 Fms-Related Tyrosine Kinase 4 (FLT4) 抗体的35不同的供货商。 再加上,我们可以发FLT4 试剂盒 (46)FLT4 蛋白 (34)和数多这个蛋白质的别的产品。 总共333 FLT4产品已列进来了。
别名:
6130401C07, AI323512, cb1059, Chy, Flk-1, Flk1, Flt-4, FLT41, Krd-1, LMPH1A, Ly73, PCL, sVEGFR-2, VEGFR-2, VEGFR-3, VEGFR2, Vegfr3

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引用最多的anti-FLT4 抗体

  1. Human Monoclonal FLT4 Primary Antibody for FACS - ABIN4896919 : Shawber, Funahashi, Francisco, Vorontchikhina, Kitamura, Stowell, Borisenko, Feirt, Podgrabinska, Shiraishi, Chawengsaksophak, Rossant, Accili, Skobe, Kitajewski: Notch alters VEGF responsiveness in human and murine endothelial cells by direct regulation of VEGFR-3 expression. in The Journal of clinical investigation 2007 (PubMed)
    Show all 5 references for ABIN4896919

  2. Human Monoclonal FLT4 Primary Antibody for FACS - ABIN4896918 : Roorda, Ter Elst, Scherpen, Meeuwsen-de Boer, Kamps, de Bont: VEGF-A promotes lymphoma tumour growth by activation of STAT proteins and inhibition of p27(KIP1) via paracrine mechanisms. in European journal of cancer (Oxford, England : 1990) 2010 (PubMed)
    Show all 5 references for ABIN4896918

  3. Human Polyclonal FLT4 Primary Antibody for WB - ABIN616061 : Paone, Galli, Gabellini, Lukashev, Starace, Gorlach, De Cesaris, Ziparo, Del Bufalo, Sitkovsky, Filippini, Riccioli: Toll-like receptor 3 regulates angiogenesis and apoptosis in prostate cancer cell lines through hypoxia-inducible factor 1 alpha. in Neoplasia (New York, N.Y.) 2010 (PubMed)
    Show all 3 references for ABIN616061

  4. Human Polyclonal FLT4 Primary Antibody for ELISA, WB - ABIN1997515 : Shushanov, Bronstein, Adélaïde, Jussila, Tchipysheva, Jacquemier, Stavrovskaya, Birnbaum, Karamysheva: VEGFc and VEGFR3 expression in human thyroid pathologies. in International journal of cancer. Journal international du cancer 2000 (PubMed)
    Show all 2 references for ABIN1997515

  5. Human Monoclonal FLT4 Primary Antibody for EIA, WB - ABIN115160 : Davids, Davids, Charlton, Ng, Chong, Laubscher, Dar, Hodge, Soong, Goh: Response to a novel multitargeted tyrosine kinase inhibitor pazopanib in metastatic Merkel cell carcinoma. in Journal of clinical oncology : official journal of the American Society of Clinical Oncology 2009 (PubMed)
    Show all 2 references for ABIN115160

  6. Human Monoclonal FLT4 Primary Antibody for EIA, IHC (fro) - ABIN115161 : Jussila, Valtola, Partanen, Salven, Heikkilä, Matikainen, Renkonen, Kaipainen, Detmar, Tschachler, Alitalo, Alitalo: Lymphatic endothelium and Kaposi's sarcoma spindle cells detected by antibodies against the vascular endothelial growth factor receptor-3. in Cancer research 1998 (PubMed)
    Show all 2 references for ABIN115161

  7. Human Polyclonal FLT4 Primary Antibody for IHC (fro), IHC (p) - ABIN115449 : Earnshaw, Halligan, Cooke, Heck, Liu: Topoisomerase II is a structural component of mitotic chromosome scaffolds. in The Journal of cell biology 1985 (PubMed)
    Show all 2 references for ABIN115449

  8. Mouse (Murine) Polyclonal FLT4 Primary Antibody for FACS - ABIN4896915 : Imoukhuede, Popel: Expression of VEGF receptors on endothelial cells in mouse skeletal muscle. in PLoS ONE 2012 (PubMed)

  9. Human Polyclonal FLT4 Primary Antibody for IHC (p) - ABIN115450 : Dietrich, Onderka, Bock, Kruse, Vossmeyer, Stragies, Zahn, Cursiefen: Inhibition of inflammatory lymphangiogenesis by integrin alpha5 blockade. in The American journal of pathology 2007 (PubMed)

更多抗FLT4的相互作用对抗体

Mouse (Murine) Fms-Related Tyrosine Kinase 4 (FLT4) interaction partners

  1. Deletion of microRNA miR (显示 MLXIP 抗体)-150 increased the retinal pathological angiogenesis in high-fat-diet (HFD) induced type 2 diabetic mice, which was in part through vascular endothelial growth factor receptor 2 (VEGFR2 (显示 KDR 抗体)).

  2. findings demonstrated that a multi-component Chinese medicine DHI effectively increased blood flow recovery after tissue ischemia in diabetic mice by promoting angiogenesis and improving glucose tolerance through a concomitant activation of VEGF-A (显示 VEGFA 抗体)/VEGFR-2 (显示 KDR 抗体) and PPARdelta (显示 PPARD 抗体) signaling pathways

  3. Intronic Flk1 (显示 KDR 抗体) genetic enhancer element directs arterial-specific expression via RBPJ (显示 RBPJ 抗体)-mediated venous repression.

  4. Endothelial SCUBE2 may be a novel coreceptor for VEGFR2 and potentiate VEGF-induced signaling in adult angiogenesis.

  5. VEGFR2 (显示 KDR 抗体) signaling is necessary to maintain the integrity of the intestinal mucosal microvasculature during the postnatal period and lack of VEGFR2 (显示 KDR 抗体) signaling predisposes to NEC in neonatal mice.

  6. First-in-class selective PET tracers for imaging VEGFR-1 (显示 FLT1 抗体) and VEGFR-2 (显示 KDR 抗体) were constructed and successfully validated in an orthotopic murine tumor model.

  7. Blockade of FLT4 suppresses metastasis of melanoma cells by impaired lymphatic vessels.

  8. Vegf-d (显示 Figf 抗体) and its receptor Vegfr-3 were more highly expressed in lungs of hyperoxic, versus normoxic, wild-type mice, indicating that components of the Vegf-d (显示 Figf 抗体) signalling pathway are up-regulated in hyperoxia.

  9. data for the first time demonstrate a calpain/PTP1B/VEGFR2 negative feedback loop in the regulation of VEGF-induced angiogenesis. Modulation of local PTP1B and/or calpain activities may prove beneficial in the treatment of impaired wound healing in diabetes.

  10. We found that bevacizumab reduced airway hyper-responsiveness (AHR (显示 AHR 抗体)) and airway inflammation induced by HDM (显示 HDAC3 抗体). In addition, HDM (显示 HDAC3 抗体) exposure promoted expression of VEGF (显示 VEGFA 抗体), and caused AHR (显示 AHR 抗体), disruptions of the epithelial barrier, and airway inflammation. Bevacizumab ameliorated AHR (显示 AHR 抗体) and the release of Th2 cytokines, thereby protecting the epithelial barrier. Our data suggest that bevacizumab may be a new therapeutic strategy for asthm

Human Fms-Related Tyrosine Kinase 4 (FLT4) interaction partners

  1. The normalized methylation values for the VEGFR1 (显示 FLT1 抗体), VEGFR2 (显示 KDR 抗体) and VEGFR3 promoters tended to be higher in the tumour cell lines than in normal tonsil samples, whereas amounts of VEGFR1 (显示 FLT1 抗体), VEGFR2 (显示 KDR 抗体) and VEGFR3 messenger RNA were significantly higher

  2. These results indicate that VEGF-C (显示 VEGFC 抗体)-induced MSC (显示 MSC 抗体) osteogenesis is mediated through VEGFR2 (显示 KDR 抗体) and VEGFR3, and followed the activation of the ERK (显示 EPHB2 抗体)/RUNX2 (显示 RUNX2 抗体) signaling pathway.

  3. Assessment of VEGFR-2/VEGFR (显示 KDR 抗体)-3 on tumor samples might serve as a putative prognostic factor in renal cell carcinoma (显示 MOK 抗体) cases, identifying a subset of patients that may benefit from antiangiogenic treatments targeting VEGFR (显示 KDR 抗体) receptors.

  4. This study suggests that NRP1 (显示 NELL1 抗体) expression and LVD are independent factors that are likely to predict the risk of LN metastasis in squamous cell carcinoma (SCC (显示 CYP11A1 抗体))of the tongue, whereas the expression of VEGFC (显示 VEGFC 抗体), VEGFR3, CCR7 (显示 CCR7 抗体), and SEMA3E (显示 SEMA3E 抗体) are nonindependent predictive factors

  5. Data indicate that vascular endothelial growth factor D (VEGF-D (显示 Figf 抗体)) was the best indicator of metastasis and vascular endothelial growth factors and receptor-3 (VEGFR-3) may help to determine the prognosis and management of colorectal cancer (CRC (显示 CALR 抗体)).

  6. The summarizes the structure and function features of pathway-related molecules of VEGFC (显示 VEGFC 抗体)/D-VEGFR3/NRP2 (显示 NELL2 抗体) axis, stages of various tumors and their molecular mechanisms and significances in tuthe expression changes of these molecules in different anatomic organs or histopathologic types or development lymphatic metastasis.

  7. The most extensively accepted signaling pathways promoting lymphangiogenesis in tumors include the secreted lymphangiogenic proteins: VEGF-C (显示 VEGFC 抗体) and VEGF-D (显示 Figf 抗体), and their cognate receptor on lymphatic endothelium VEGF receptor (显示 FLT1 抗体)-3 (VEGFR-3).

  8. MTA1 (显示 MTA1 抗体) is up-regulated in CRC (显示 CALR 抗体); its expression is inversely associated with lymphatic metastases and the expression of VEGFC (显示 VEGFC 抗体), VEGFD (显示 Figf 抗体) and VEGFR3

  9. this study uncovers a unique molecular mechanism of lymphangiogenesis in which galectin-8 (显示 LGALS8 抗体)-dependent crosstalk among VEGF-C (显示 VEGFC 抗体), podoplanin (显示 PDPN 抗体) and integrin pathways plays a key role.

  10. Report FLT4 genetic alterations in angiosarcomas.

Zebrafish Fms-Related Tyrosine Kinase 4 (FLT4) interaction partners

  1. miR (显示 MYLIP 抗体)-126a directs lymphatic endothelial cell sprouting and extension by interacting with Cxcl12a-mediated chemokine (显示 CCL1 抗体) signaling and Vegfc (显示 VEGFC 抗体)-Flt4 signal axis.

  2. Ca(2 (显示 CA2 抗体)+) oscillations depended upon VEGF receptor-2 (Vegfr2) and Vegfr3 in endothelial cells budding from the dorsal aorta (DA) and posterior cardinal (显示 CARD8 抗体) vein, respectively.

  3. Experiments in mice and zebrafish demonstrate that changing levels of VEGFR3/Flt4 modulates aortic lumen diameter consistent with flow-dependent remodeling

  4. In the embryo, phenotypes driven by increased Vegfc (显示 VEGFC 抗体) are suppressed in the absence of Ccbe1 (显示 CCBE1 抗体), and Vegfc (显示 VEGFC 抗体)-driven sprouting is enhanced by local Ccbe1 (显示 CCBE1 抗体) overexpression. Moreover, Vegfc (显示 VEGFC 抗体)- and Vegfr3-dependent Erk (显示 MAPK1 抗体) signaling is impaired in the absence of Ccbe1 (显示 CCBE1 抗体).

  5. Flt4 plays an essential role in lymphangiogenesis [review]

  6. The parallel growth of motoneuron axons with the dorsal aorta depends on Vegfc (显示 VEGFC 抗体)/Vegfr3 signaling in zebrafish.

  7. Rspo1-Wnt-VegfC-Vegfr3 signaling plays a crucial role as an endothelial-autonomous permissive cue for developmental angiogenesis.

  8. flt4 signalling is suppressed by Dll4 (显示 DLL4 抗体) in developing zebrafish intersegmental arteries.

FLT4 抗原简介

Antigen Summary

This gene encodes a tyrosine kinase receptor for vascular endothelial growth factors C and D. The protein is thought to be involved in lymphangiogenesis and maintenance of the lymphatic endothelium. Mutations in this gene cause hereditary lymphedema type IA.

Alternative names and synonyms associated with FLT4

  • FMS-like tyrosine kinase 4 (Flt4) 抗体
  • fms-related tyrosine kinase 4 (FLT4) 抗体
  • fms-related tyrosine kinase 4 (Flt4) 抗体
  • fms-related tyrosine kinase 4 (flt4) 抗体
  • kinase insert domain protein receptor (Kdr) 抗体
  • 6130401C07 抗体
  • AI323512 抗体
  • cb1059 抗体
  • Chy 抗体
  • Flk-1 抗体
  • Flk1 抗体
  • Flt-4 抗体
  • FLT41 抗体
  • Krd-1 抗体
  • LMPH1A 抗体
  • Ly73 抗体
  • PCL 抗体
  • sVEGFR-2 抗体
  • VEGFR-2 抗体
  • VEGFR-3 抗体
  • VEGFR2 抗体
  • Vegfr3 抗体

Protein level used designations for FLT4

chylous ascites , receptor protein tyrosine kinase , tyrosine-protein kinase receptor FLT4 , vascular endothelial growth factor receptor 3 , vascular endothelial growth factor receptor-3 , FLT-4 , VEGFR-3 , fms-like tyrosine kinase 4 , soluble VEGFR3 variant 1 , soluble VEGFR3 variant 2 , soluble VEGFR3 variant 3 , FMS-like tyrosine kinase 4 , tyrosine kinase VEGFR-3 , VEGFR3 , expando , receptor tyrosine kinase Flt4 , VEGF receptor-2 , fetal liver kinase 1 , kinase NYK , protein-tyrosine kinase receptor flk-1 , soluble vascular endothelial growth factor receptor 2 , vascular endothelial growth factor receptor 2 , vascular endothelial growth factor receptor- 2 , vascular endothelial growth factor receptor-2

GENE ID SPECIES
14257 Mus musculus
2324 Homo sapiens
114110 Rattus norvegicus
338031 Bos taurus
100008829 Oryctolagus cuniculus
395742 Gallus gallus
30121 Danio rerio
16542 Mus musculus
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