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CD69 encodes a member of the calcium dependent lectin superfamily of type II transmembrane receptors. 再加上，我们可以发CD69 试剂盒 (20) 和 CD69 蛋白 (16)和数多这个蛋白质的别的产品。
Showing 10 out of 431 products:
Human Monoclonal CD69 Primary Antibody for Func, FACS - ABIN263925
Nielsen, Afzelius, Ersbøll, Nielsen, Hansen: Expression of the activation antigen CD69 predicts functionality of in vitro expanded peripheral blood mononuclear cells (PBMC) from healthy donors and HIV-infected patients. in Clinical and experimental immunology 1998
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Human Monoclonal CD69 Primary Antibody for FACS - ABIN263924
Pitsios, Dimitrakopoulou, Tsalimalma, Kordossis, Choremi-Papadopoulou: Expression of CD69 on T-cell subsets in HIV-1 disease. in Scandinavian journal of clinical and laboratory investigation 2008
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Human Monoclonal CD69 Primary Antibody for FACS - ABIN263926
Drbal, Moertelmaier, Holzhauser, Muhammad, Fuertbauer, Howorka, Hinterberger, Stockinger, Schütz: Single-molecule microscopy reveals heterogeneous dynamics of lipid raft components upon TCR engagement. in International immunology 2007
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Human Monoclonal CD69 Primary Antibody for FACS - ABIN263923
Hrdinka, Dráber, Stepánek, Ormsby, Otáhal, Angelisová, Brdicka, Paces, Horejsí, Drbal: PRR7 is a transmembrane adaptor protein expressed in activated T cells involved in regulation of T cell receptor signaling and apoptosis. in The Journal of biological chemistry 2011
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Human Monoclonal CD69 Primary Antibody for FACS - ABIN4897718
Giuliani, Fleury, Vernochet, Ketroussi, Clay, Azzarone, Lataillade, Durrbach: Long-lasting inhibitory effects of fetal liver mesenchymal stem cells on T-lymphocyte proliferation. in PLoS ONE 2011
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Mouse (Murine) Monoclonal CD69 Primary Antibody for FACS - ABIN118239
Yokoyama, Koning, Kehn, Pereira, Stingl, Coligan, Shevach: Characterization of a cell surface-expressed disulfide-linked dimer involved in murine T cell activation. in Journal of immunology (Baltimore, Md. : 1950) 1988
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Human Monoclonal CD69 Primary Antibody for ELISA, FACS - ABIN4295495
Telang, Clem, Klarer, Clem, Trent, Bucala, Chesney: Small molecule inhibition of 6-phosphofructo-2-kinase suppresses t cell activation. in Journal of translational medicine 2012
Human Monoclonal CD69 Primary Antibody for IP, ELISA - ABIN1998359
Marzio, Mauël, Betz-Corradin: CD69 and regulation of the immune function. in Immunopharmacology and immunotoxicology 1999
Data show that a subset of inflammation-induced CD69(+) Kupffer cells which can feedback inhibit CD4 (显示 CD4 抗体) T cell response via cell surface TGF-beta1 (显示 TGFB1 抗体) at the late stage of immune response against infection.
Specific niches for lung-resident memory CD8 (显示 CD8A 抗体)+ T cells at the site of tissue regeneration enable CD69-independent maintenance.
this study shows that CD69 serves as a key mediator of the pathogenesis of psoriasis by controlling LAT1-CD98 (显示 SLC7A5 抗体)-mediated metabolic cues
Loss of CD69 is associated with enhanced protection against vaccinia virus infection.
The data highlight the contribution of CD69 as a nonredundant key regulator of BIC/miR (显示 MLXIP 抗体)-155-dependent regulatory T cell development and homeostasis.
Data show that after tumor necrosis factor alpha-induced protein 8 like-2 (TIPE2 (显示 TNFAIP8L2 抗体)) gene was down-regulated, the expression of the CD69 antigen was increased, and the proliferation of T lymphocytes and the secretion of cytokines IL-2 (显示 IL2 抗体) and IFN-gamma (显示 IFNG 抗体) were enhanced.
The generation of cancer-specific CD8 (显示 CD8A 抗体)(+) CD69(+)-expressing lymphocytes that inhibit colon cancer growth has been described.
Maintenance of immune tolerance by Foxp3 (显示 FOXP3 抗体)+ regulatory T cells requires CD69 expression.
AIM(-/-) mice were highly susceptible to steatosis-associated HCC (显示 FAM126A 抗体) development, whereas no AIM(+/+) mouse developed the disease despite comparable liver inflammation and fibrosis in response to a long-term high-fat diet.
CD69 interference with S1P1 (显示 S1PR1 抗体) function regulates peripheral T cell retention.
Higher CD69 expression were less sensitive to bendamustine and is associated with chronic lymphocytic leukemia.
In vitro functional assays showed that CD69(+) Treg cells exerted an important suppressive effect on the activation of T effector cells
results demonstrate the functional and mechanistic interplays between CD69 and S100A8 (显示 S100A8 抗体)/S100A9 (显示 S100A9 抗体) in supporting Treg-cell differentiation
Elevated expression of CD69 and CD161 (显示 KLRB1 抗体) on NK cells can be considered as immunological risk markers in RSA and IVF (显示 SCN5A 抗体) failure.
Human tumor-infiltrating CD4 (显示 CD4 抗体)+CD69+ T cells suppress T cell proliferation via membarene -bound TGF-beta1 (显示 TGFB1 抗体).
these findings identify CD69 and galectin-1 (显示 LGALS1 抗体) to be a novel regulatory receptor-ligand pair that modulates Th17 effector cell differentiation and function.
REVIEW: CD69 exerts a complex immunoregulatory role in humans, and that it could be considered as a target molecule for the therapy of immune-mediated diseases
Following coculture with GTKO/CD46 (显示 CD46 抗体) pig mesenchymal stromal cells, it is possible that upregulation of CD69 on human T cells initiates signaling events that would regulate CD4 (显示 CD4 抗体)+ and CD8 (显示 CD8A 抗体)+ T cell activation and differentiation.
In patients with allergic rhinitis CD69 antigen is overexpressed on human peripheral blood natural killer cells reflecting their activation status.
This is the first report of the regulation of CD69 expression by LMP-1 (显示 PDLIM7 抗体), and this novel finding may, thus, represent an important link between the EBV oncoprotein LMP-1 (显示 PDLIM7 抗体) and its critical role in the development of EBV-associated diseases.
This gene encodes a member of the calcium dependent lectin superfamily of type II transmembrane receptors. Expression of the encoded protein is induced upon activation of T lymphocytes, and may play a role in proliferation. Furthermore, the protein may act to transmit signals in natural killer cells and platelets.
, early activation antigen CD69
, Very Early Activation Antigen
, C-type lectin domain family 2, member C
, CD69 antigen (p60, early T-cell activation antigen)
, activation inducer molecule (AIM/CD69)
, early T-cell activation antigen p60
, early lymphocyte activation antigen
, leukocyte surface antigen Leu-23