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Human Neurotrophin 3 ELISA Kit for Sandwich ELISA - ABIN625065
Munkholm, Pedersen, Kessing, Vinberg: Elevated levels of plasma brain derived neurotrophic factor in rapid cycling bipolar disorder patients. in Psychoneuroendocrinology 2014
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Rat (Rattus) Neurotrophin 3 ELISA Kit for Sandwich ELISA - ABIN411340
Ozçelik, Rosenthal, Francke: Chromosomal mapping of brain-derived neurotrophic factor and neurotrophin-3 genes in man and mouse. in Genomics 1991
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Human Neurotrophin 3 ELISA Kit for Sandwich ELISA - ABIN411338
Tessarollo, Vogel, Palko, Reid, Parada: Targeted mutation in the neurotrophin-3 gene results in loss of muscle sensory neurons. in Proceedings of the National Academy of Sciences of the United States of America 1995
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Mouse (Murine) Neurotrophin 3 ELISA Kit for Sandwich ELISA - ABIN411339
Donovan, Hahn, Tessarollo, Hempstead: Identification of an essential nonneuronal function of neurotrophin 3 in mammalian cardiac development. in Nature genetics 1996
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Rat (Rattus) Neurotrophin 3 ELISA Kit for Sandwich ELISA - ABIN368093
Lu, Dong, Lu, Lv, Yang, Cheng, Li, Liang, Xu, Li, He: Effects of different sera conditions on olfactory ensheathing cells in vitro. in International journal of molecular sciences 2014
Mouse (Murine) Neurotrophin 3 ELISA Kit for Sandwich ELISA - ABIN424194
Khallaf, Messiha, Abo-Youssef, El-Sayed: Protective effects of telmisartan and tempol on lipopolysaccharide-induced cognitive impairment, neuroinflammation, and amyloidogenesis: possible role of brain-derived neurotrophic factor. in Canadian journal of physiology and pharmacology 2017
NT-3 has been shown to be both an osteogenic and angiogenic factor, and can also enhance expression of the key osteogenic factor, BMP-2, as well as the major angiogenic factor, VEGF, to promote bone formation, vascularization, and bone healing
High expression of NT-3 is associated with glioblastoma.
Meta-analysis found the levels of both NT-3 and NT-4/5 (显示 NTF4 ELISA试剂盒) were significantly increased in bipolar disorder patients. Through subgroup analysis, this increase persisted only in patients in depressed state, but not in manic or euthymic state. In addition, we found the differences in NT-3 and NT-4/5 (显示 NTF4 ELISA试剂盒) were significantly associated with the duration of illness, but not by the mean age or female proportion.
NT3 upregulates cellular proliferation, extracellular matrix protein production, and collagen deposition in human aortic valve interstitial cells through the Trk (显示 NTRK1 ELISA试剂盒)-Akt (显示 AKT1 ELISA试剂盒)-cyclin D1 (显示 CCND1 ELISA试剂盒) cascade.
The analysis of covariance (ANCOVA) indicated that the mean serum GDNF and NTF3 levels of ADHD patients were significantly higher than that of controls. However, serum BDNF (显示 BDNF ELISA试剂盒) and NGF (显示 NGFB ELISA试剂盒) levels did not show any significant differences between groups. These results suggest that elevated serum GDNF and NTF3 levels may be related to ADHD in children.
All patients had serum neurotrophin (显示 BDNF ELISA试剂盒) (NT-3, BDNF (显示 BDNF ELISA试剂盒), NGF (显示 NGFB ELISA试剂盒)) concentrations determined.
There were no differences in neurotrophin (显示 BDNF ELISA试剂盒) levels between patients with schizophrenia and controls. We found lower BDNF (显示 BDNF ELISA试剂盒) and higher NT-3 serum levels in depressed patients with schizophrenia.
The results suggest a gene dose-association between the A allele of rs6332 and the onset of AD in varepsilon4 non-carriers and the NTF-3 rs6489630 polymorphism being a relevant risk factor for AD in patients lacking the ApoE (显示 APOE ELISA试剂盒)-varepsilon4 allele in this Chinese sample.
Survival, differentiation, and neuroprotective mechanisms of human stem cells complexed with neurotrophin-3-releasing pharmacologically active microcarriers in an ex vivo model of Parkinson's disease.
No differences in plasma BDNF, NGF, NT3, NT4 and GDNF were found between autism spectrum disorders and control.
By immunohistochemistry, the localization of Neurotrophinss has been observed mainly in Purkinje cells; TrkA (显示 NTRK1 ELISA试剂盒) and TrkB (显示 NTRK2 ELISA试剂盒)-receptors in cells and fibers of granular and molecular layers. TrkC (显示 NTRK3 ELISA试剂盒) was faintly detected
Deletion of a kinesin I motor unmasks a mechanism of homeostatic axon-branching control by neurotrophin-3.
Here the authors demonstrate a novel function for p75(NTR (显示 NGFR ELISA试剂盒)) in regulating proper cell cycle exit of neuronal progenitors in the developing rat and mouse external granule layer, which is stimulated by proneurotrophin-3. In the absence of p75(NTR (显示 NGFR ELISA试剂盒)), cerebellar granule cell progenitors continue to proliferate beyond their normal period, resulting in a larger cerebellum that persists into adulthood, with consequent motor defici
NT-3-transduced bone marrow- derived neural stem cells differentiated into a greater number of cholinergic neurons.
Ntf3, but not Bdnf (显示 BDNF ELISA试剂盒), expression by postnatal supporting cells regulates cochlear function.
Study shows that dissociated cochlear nucleus neurons can be stimulated by neurotrophic factors BDNF (显示 BDNF ELISA试剂盒), NT-3, and FGF2 (显示 FGF2 ELISA试剂盒)
Data show that acteoside can up-regulate the expression of brain neurotrophin-3 (NT-3) in D-galactose combined with aluminum trichloride treated mice.
CAPS2 (显示 CADPS2 ELISA试剂盒) plays an important role in subcellular locality (axonal vs. somato (显示 SSTR5 ELISA试剂盒)-dendritic) of enhanced BDNF (显示 BDNF ELISA试剂盒) and NT-3 release, which is indispensable for proper development of postnatal cerebellum.
Uptake of NT-3 from irrigating vasculature and cerebrospinal fluid induces the rapid phosphorylation of endothelial nitric oxide
Loss of Ntf3, by contrast, causes an increase in layer VI neurons but does not rescue the Sip1 (显示 ZEB2 ELISA试剂盒) mutant phenotype, implying that other parallel pathways also control the timing of progenitor cell fate switch.
it was PDGF, NT-3 and IGF-2 in combination that conducted the transdifferentiation
This study aimed to examine the roles of mature neurotrophin-3 (NT3), pro-NT3 and p75 neurotrophin receptor (P75 (显示 NGFR ELISA试剂盒)(NTR (显示 NGFR ELISA试剂盒))) in photoreceptor degeneration
The protein encoded by this gene is a member of the neurotrophin family, that controls survival and differentiation of mammalian neurons. This protein is closely related to both nerve growth factor and brain-derived neurotrophic factor. It may be involved in the maintenance of the adult nervous system, and may affect development of neurons in the embryo when it is expressed in human placenta. NTF3-deficient mice generated by gene targeting display severe movement defects of the limbs. The mature peptide of this protein is identical in all mammals examined including human, pig, rat and mouse.
, nerve growth factor 2
, neurotrophic factor
, neurotrophin 3 - pending
, neurotrophin 3
, neurotrophin-3 (HDNF/NT-3)