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抗Rat (Rattus) Tumor Protein p73 抗体:
抗Human Tumor Protein p73 抗体:
抗Mouse (Murine) Tumor Protein p73 抗体:
Human Monoclonal Tumor Protein p73 Primary Antibody for ChIP, CyTOF - ABIN4343232
Shimodaira, Yoshioka-Yamashita, Kolodner, Wang: Interaction of mismatch repair protein PMS2 and the p53-related transcription factor p73 in apoptosis response to cisplatin. in Proceedings of the National Academy of Sciences of the United States of America 2003
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Human Monoclonal Tumor Protein p73 Primary Antibody for ChIP, ICC - ABIN252619
Costanzo, Merlo, Pediconi, Fulco, Sartorelli, Cole, Fontemaggi, Fanciulli, Schiltz, Blandino, Balsano, Levrero: DNA damage-dependent acetylation of p73 dictates the selective activation of apoptotic target genes. in Molecular cell 2002
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Human Polyclonal Tumor Protein p73 Primary Antibody for IHC (p), IP - ABIN151880
Levy, Adamovich, Reuven, Shaul: The Yes-associated protein 1 stabilizes p73 by preventing Itch-mediated ubiquitination of p73. in Cell death and differentiation 2007
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Human Monoclonal Tumor Protein p73 Primary Antibody for IP, WB - ABIN967629
Jost, Marin, Kaelin: p73 is a simian [correction of human] p53-related protein that can induce apoptosis. in Nature 1997
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Human Monoclonal Tumor Protein p73 Primary Antibody for IP, WB - ABIN967628
Zhu, Jiang, Zhou, Chen: The potential tumor suppressor p73 differentially regulates cellular p53 target genes. in Cancer research 1998
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Human Monoclonal Tumor Protein p73 Primary Antibody for IP, WB - ABIN967630
De Laurenzi, Costanzo, Barcaroli, Terrinoni, Falco, Annicchiarico-Petruzzelli, Levrero, Melino: Two new p73 splice variants, gamma and delta, with different transcriptional activity. in The Journal of experimental medicine 1998
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Human Monoclonal Tumor Protein p73 Primary Antibody for GS, ICC - ABIN269466
King, Reddi, Ponnamperuma, Gerdes, Weinberg: Dysregulated ΔNp63α negatively regulates the maspin promoter in keratinocytes via blocking endogenous p73 binding. in Molecular carcinogenesis 2014
Human Monoclonal Tumor Protein p73 Primary Antibody for WB - ABIN252620
Shao, Tanaka, Gribi, Yu: Thioredoxin-related regulation of NO/NOS activities. in Annals of the New York Academy of Sciences 2002
Human Polyclonal Tumor Protein p73 Primary Antibody for IHC - ABIN966779
Yuan, Shioya, Ishiko, Sun, Gu, Huang, Lu, Kharbanda, Weichselbaum, Kufe: p73 is regulated by tyrosine kinase c-Abl in the apoptotic response to DNA damage. in Nature 1999
In the present study, we provide evidence that the tumor suppressor gene p73 is highly susceptible to Mn-induced neurotoxicity in the nigrostriatal system.
HECW2 (显示 HECW2 抗体) is an ubiquitin ligase that stabilises p73, a crucial mediator of neurodevelopment and neurogenesis. This study implicates pathogenic genetic variants in HECW2 (显示 HECW2 抗体) as potential causes of neurodevelopmental disorders in humans.
Imbalance of the apoptosis pathway, with dysregulation of p73 and TRAIL, seems to play a role in the oncogenesis of odontogenic tumors
High TP73 expression is associated with Metastasis of Hepatocellular Carcinoma.
The reduction of tumor protein p63 (显示 TP63 抗体) and tumor protein p73 isoforms, rather than alteration of DeltaN isoform expression, exerted a significant functional repercussion on cell death and proliferation in hepatitis B virus -expressing HepB cells.
p73 is epigenetically silenced in chondrosarcoma due to promoter methylation, which suggests the utility of p73 methylation as a biomarker.
A considerable number of lymphoma patients lacked the expression of either or both isoforms, while all lymphoid leukemia patients expressed both isoforms. The expression pattern differences of p73 isoforms may reflect differences in the biology of these malignancies.
TAp73beta upregulates pro-IL-1beta (显示 IL1B 抗体) mRNA and processed IL-1beta (显示 IL1B 抗体) protein. In addition, analysis of breast and lung cancer patient cohorts demonstrated that interaction between p73 and IL-1beta (显示 IL1B 抗体) predicts a negative survival outcome in these cancers.
analysis of how trifluoroethanol induces a conformational transition in the C-terminal sterile alpha motif (SAM (显示 TTN 抗体)) of human p73
The findings of this study suggested that the polymorphism G4C14-to-A4T14 in p73 gene might be associated with severe spermatogenesis impairment and could affect the susceptibility to male infertility with severe spermatogenesis impairment in Chinese population.
both p53 (显示 TP53 抗体) and p73 (显示 ARHGAP24 抗体) are critical in apoptosis induced by DNA damage and differentiation.
New function of p73 (显示 ARHGAP24 抗体), independent of p53 (显示 TP53 抗体), in the neurogenic architecture of the SVZ of rodent brain.
these results therefore highlight an unanticipated role for p53 (显示 TP53 抗体) family proteins in a regulatory network that integrates essential Wnt (显示 WNT2 抗体)-Tcf (显示 HNF4A 抗体) and nodal-Smad (显示 SMAD1 抗体) inputs.
TAp73 as necessary and sufficient for basal body docking, axonemal extension, and motility during the differentiation of Motile multiciliated cell progenitors.
p73 (显示 ARHGAP24 抗体) drives multiciliogenesis, both through transcriptional activation of a master ciliogenesis transcription factor FoxJ1 (显示 FOXJ1 抗体) and through regulation of multiple genes central to ciliogenesis.
The p73 (显示 ARHGAP24 抗体) acts as a critical regulator of multiciliogenesis in its capacity as a sequence-specific transcription factor, through genomic binding and regulation of genes.
Data show that the Mdm4 (显示 MDM4 抗体)-p73 (显示 ARHGAP24 抗体) axis cannot override the dominant role of p53 (显示 TP53 抗体) in development and tumorigenesis and that Mdm4 (显示 MDM4 抗体) and p73 (显示 ARHGAP24 抗体) interaction during development and tumorigenesis suggests new insight into the role of p53 (显示 TP53 抗体) family members.
In vivo inhibition of both p63 (显示 CKAP4 抗体) and p73 (显示 ARHGAP24 抗体) in combination accelerates tumor regression and increases survival of p53 (显示 TP53 抗体)-deficient mice.
Results indicate that p73 (显示 ARHGAP24 抗体) regulates basal and starvation-induced fuel metabolism in the liver, a finding that is likely to be highly relevant for metabolism-associated disorders, such as diabetes and cancer.
MDM2 (显示 MDM2 抗体) mediates p73 (显示 ARHGAP24 抗体) ubiquitination
This gene encodes a member of the p53 family of transcription factors involved in cellular responses to stress and development. It maps to a region on chromosome 1p36 that is frequently deleted in neuroblastoma and other tumors, and thought to contain multiple tumor suppressor genes. The demonstration that this gene is monoallelically expressed (likely from the maternal allele), supports the notion that it is a candidate gene for neuroblastoma. Many transcript variants resulting from alternative splicing and/or use of alternate promoters have been found for this gene, but the biological validity and the full-length nature of some variants have not been determined.
transformation related protein 73
, tumor protein p73
, tumor protein p73-like
, p53-like transcription factor
, p53-related protein