Use your antibodies-online credentials, if available.
Select your species
Based on the ADEPT study data set, we report that airway mucosal expression of CCL26 was a robust discriminator of type 2 inflammation from healthy nonatopic subjects. Furthermore, airway mucosal CCL26 expression was best identified by using a panel of clinical biomarkers, including Feno values, bEOS counts, and expression of 2 novel markers, sCCL17 and sCCL26
hypoxia-inducible factors activate the transcription of chemokine (显示 CCL1 ELISA试剂盒) ligand 26 in cancer cells to recruit chemokine receptor 1 (显示 CMKLR1 ELISA试剂盒)-expressing myeloid-derived suppressor cells to the primary tumor
this study shows that expression of CCL26 is increased in eosinophilic myocarditis patients compared to chronic lymphocytic myocarditis patients
The reduction of circulating levels of MCP-4 (显示 CCL13 ELISA试剂盒), eotaxin-3, and MIP-1beta (显示 CCL4 ELISA试剂盒) could be one of the mechanisms by which bariatric surgery contributes to the reduction of cardiovascular risk in these patients.
Review: eotaxins (CCL11 (显示 CCL11 ELISA试剂盒), CCL24 (显示 CCL24 ELISA试剂盒), and CCL26) play key role(s) during symptomatic inflammatory responses raised in response to allergic crisis of allergic asthma and atopic dermatitis
Data show that the interleukins IL-4 (显示 IL4 ELISA试剂盒) and IL-13 (显示 IL13 ELISA试剂盒) are critical factors for the induction of eotaxin-3/CCL26 in the pancreas.
These results show a relation between CCL26 production by IL-13 (显示 IL13 ELISA试剂盒)-stimulated bronchial epithelial cells , sputum eosinophil counts, and asthma severity. They also suggest a role for CCL26 in the sustained inflammation observed in patients with severe eosinophilic asthma and reveal CCL26 as a potential target for treating patients with eosinophilic asthma that are refractory
The most potent inhibition of RANTES (显示 CCL5 ELISA试剂盒), eotaxin (显示 CCL11 ELISA试剂盒) and eotaxin3 as well as MMP9 (显示 MMP9 ELISA试剂盒) activity.
These findings indicate that eotaxin-3 is expressed in Chronic subdural hematoma fluid
these data suggest that CCL26 and CCL24 (显示 CCL24 ELISA试剂盒) are likely involved in the pathogenesis of chronic nasal hypereosinophilia, with a complex cooperation and different involvement of the various members of eotaxin (显示 CCL11 ELISA试剂盒) family.
Data indicate that puerarin attenuates airway inflammation via eotaxin-3.
Distinct acidic and basic residues within CCR3 (显示 CCR3 ELISA试剂盒) determine both receptor expression and activation by the eotaxins.
This gene is one of two Cys-Cys (CC) cytokine genes clustered on the q arm of chromosome 7. Cytokines are a family of secreted proteins involved in immunoregulatory and inflammatory processes. The CC cytokines are proteins characterized by two adjacent cysteines. The cytokine encoded by this gene displays chemotactic activity for normal peripheral blood eosinophils and basophils. The product of this gene is one of three related chemokines that specifically activate chemokine receptor CCR3. This chemokine may contribute to the eosinophil accumulation in atopic diseases.
chemokine (C-C motif) ligand 26
, C-C motif chemokine 26
, CC chemokine IMAC
, chemokine N1
, macrophage inflammatory protein 4-alpha
, small inducible cytokine A26
, small inducible cytokine subfamily A (Cys-Cys), member 26
, small-inducible cytokine A26
, thymic stroma chemokine-1
, chemokine (C-C motif) ligand 26-like
, eotaxin 3
, small chemokine ligand 26