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High MTCO1 expression is associated with cognitive impairment in lung cancer.
patients with primary ovarian insufficiency exhibit an increased incidence of mitochondrial cytochrome c oxidase 1 gene mutations;MT-CO1 gene mutation may be causal in the disease
studies have provided mechanistic insights into crosstalk between assembly intermediates, import processes and the synthesis of COX (显示 COX8A ELISA试剂盒) subunits in mitochondria, thus linking conceptually separated functions.
The m.9267G>C MT-COIII mutation was present with a nonsynonymous inherited mitochondrial homoplasmic variation MT-COI m.5913 G>A.
A novel heteroplasmic mutation was identified in MTCO1, m.7402delC, causing frameshift and a premature termination codon in a mitochondrial encephalomyopathy patient with cytochrome c (显示 CYCS ELISA试剂盒) oxidase deficiency.
mitochondrial DNA mutations in COI resulting in increased reactive oxygen and reactive nitrogen generation may be involved in prostate cancer biology
Possible association of a novel missense mutation A6375G in the mitochondrial cytochrome C oxidase I gene with asthenospermia in the Tunisian population
Abeta (显示 APP ELISA试剂盒) 1-42 bound to a peptide comprising the amino-terminal region of cytochrome c oxidase subunit 1
Both full-length and truncated COX1 proteins physically interact with AFG3L2 (显示 AFG3L2 ELISA试剂盒).
Data show that homoplasmic G6709A (MT-CO1) and G14804A (MT-CYB (显示 MT-CYB ELISA试剂盒)) alterations cause amino acid changes in the highly conserved residues.
a dependence of COX1 AND capital ES, CyrillicOX2 expression on the presence of MMP-9 (显示 MMP9 ELISA试剂盒) was shown in neutrophil infiltration during inflammation.
CCOI expression is inhibited by osteopontin (显示 SPP1 ELISA试剂盒) as the result of a novel CD44 (显示 CD44 ELISA试剂盒)-dependent transcriptional regulatory mechanism of the mitochondrial H strand
The gene expression of Cox1 in adipocytes was studied.
CcOX (显示 COX5A ELISA试剂盒) I protein levels significantly decreased following CO exposure while enzyme turnover number and CcOX (显示 COX5A ELISA试剂盒) I mRNA levels remained unchanged.
IOP elevation may directly damage mitochondria in the ONH axons by promoting reduction of COX (显示 CPOX ELISA试剂盒), mitochondrial fission and cristae depletion, alterations of OPA1 (显示 MED12 ELISA试剂盒) and Dnm1 (显示 DNM1 ELISA试剂盒) expression, and induction of OPA1 (显示 MED12 ELISA试剂盒) release.
Tumor necrosis factor alpha (显示 TNF ELISA试剂盒) inhibits oxidative phosphorylation through tyrosine phosphorylation at subunit I of cytochrome c (显示 CYCS ELISA试剂盒) oxidase
Cytochrome c oxidase is the component of the respiratory chain that catalyzes the reduction of oxygen to water. Subunits 1- 3 form the functional core of the enzyme complex. CO I is the catalytic subunit of the enzyme. Electrons originating in cytochrome c are transferred via the copper A center of subunit 2 and heme A of subunit 1 to the bimetallic center formed by heme A3 and copper B.
cytochrome c oxidase subunit I