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Cow (Bovine) Polyclonal PIGF Primary Antibody for IHC, WB - ABIN2778804
Mohammed, Nasreen, Tepper, Antony: Cyclic stretch induces PlGF expression in bronchial airway epithelial cells via nitric oxide release. in American journal of physiology. Lung cellular and molecular physiology 2007
The early variations of PIGF and soluble fms-like tyrosine kinase-1 (显示 FLT1 抗体) concentrations in newly pregnant obstetric antiphospholipid syndrome (oAPS) may help to detect patients at low risk of placenta-mediated complications (PMC).
The present study investigated the interplay of VEGF (显示 VEGFA 抗体)-A165a isoform, the anti-angiogenic VEGF (显示 VEGFA 抗体)-A165b, placental growth factor (PIGF) and their receptors, VEGFR1 (显示 FLT1 抗体) and VEGFR2 (显示 KDR 抗体).on junctional occupancy of VE-cadherin (显示 CDH5 抗体) and macromolecular leakage in human endothelial monolayers and the perfused placental microvascular bed.
knockdown of PlGF (显示 PGF 抗体) in spheroid body cells derived from two gastric cancer cell lines reduced in vitro tumorigenicity and stemness properties of spheroid body cells such as self-renewal ability, colony forming, migratory, and MMPs activities and decreased ability to differentiation and angiogenesis
Lower PlGF (显示 PGF 抗体) and higher PAPP-A (显示 PAPPA 抗体) and free beta-hCG (显示 CGA 抗体) levels were found in the fetal circulation of near-term severe preeclamptic pregnancies
VEGF (显示 VEGFA 抗体)/PlGF (显示 PGF 抗体) levels were higher in neonates exposed to pre-eclampsia, and there was a significant negative correlation between birth weight and VEGF (显示 VEGFA 抗体)/PlGF (显示 PGF 抗体) levels.
There is a significant negative correlation between the concentration of sFLt-1 and PlGF (显示 PGF 抗体) in normal pregnancy.
Data showed that sFlt-1/PlGF (显示 PGF 抗体) ratio increases with volume overload and persistent hypoxia after surgery with CHD (显示 CHDH 抗体).
Glioma cell-released PlGF (显示 PGF 抗体) can induce Bregs to suppress CD8 (显示 CD8A 抗体)(+) T cell activities.
In chronic kidney patients not yet on dialysis, higher serum level of PIGF are associated with increased mortality, but not cardiovascular events.
A highly conserved motif in PIGF is not involved in binding to PIGO or PIGG, but critical for its function.
Data indicate that VEGF-B (显示 VEGFB 抗体) is a high-affinity VEGFR-1 (显示 FLT1 抗体) ligand that, unlike PlGF (显示 PGF 抗体), cannot efficiently induce signaling downstream of VEGFR-1 (显示 FLT1 抗体).
the lack of PlGF (显示 PGF 抗体) may have contributed to the delayed angiogenic response and the prothrombotic phenotype observed in the PlGF (显示 PGF 抗体)(-/-)animals.
Overexpression of VEGF but not PlGF exacerbated the lipopolysaccharide-mediated toxic effects, supporting a pathophysiological role for VEGF in mediating the sepsis phenotype.
This gene encodes a protein involved in glycosylphosphatidylinositol (GPI)-anchor biosynthesis. The GPI-anchor, a glycolipid containing three mannose molecules in its core backbone, is found on many blood cells where it serves to anchor proteins to the cell surface. The encoded protein and another GPI synthesis protein, PIGO, function in the transfer of ethanolaminephosphate to the third mannose in GPI. Alternatively spliced transcript variants encoding different isoforms have been described.
, phosphatidylinositol-glycan biosynthesis class F protein
, phosphatidylinositol glycan, class F
, phosphatidylinositol glycan anchor biosynthesis class F S homeolog
, phosphatidylinositol glycan anchor biosynthesis, class F