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Mouse (Murine) Monoclonal IL17F Primary Antibody for ICS, Neut - ABIN2689743
Chang, Dong: IL-17F: regulation, signaling and function in inflammation. in Cytokine 2009
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Human Polyclonal IL17F Primary Antibody for IHC (p), WB - ABIN530198
Oda, Shimazu, Naoi, Morimoto, Shimomura, Shimoda, Kagara, Maruyama, Kim, Noguchi: Intratumoral regulatory T cells as an independent predictive factor for pathological complete response to neoadjuvant paclitaxel followed by 5-FU/epirubicin/cyclophosphamide in breast cancer patients. in Breast cancer research and treatment 2012
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Human Monoclonal IL17F Primary Antibody for CyTOF, FACS - ABIN4900181
Zrioual, Ecochard, Tournadre, Lenief, Cazalis, Miossec: Genome-wide comparison between IL-17A- and IL-17F-induced effects in human rheumatoid arthritis synoviocytes. in Journal of immunology (Baltimore, Md. : 1950) 2009
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Mouse (Murine) Monoclonal IL17F Primary Antibody for CyTOF, FACS - ABIN4900112
Yamaguchi, Fujio, Shoda, Okamoto, Tsuno, Takahashi, Yamamoto: IL-17B and IL-17C are associated with TNF-alpha production and contribute to the exacerbation of inflammatory arthritis. in Journal of immunology (Baltimore, Md. : 1950) 2007
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Human Monoclonal IL17F Primary Antibody for IHC, WB - ABIN1043779
Conti, Shen, Nayyar, Stocum, Sun, Lindemann, Ho, Hai, Yu, Jung, Filler, Masso-Welch, Edgerton, Gaffen: Th17 cells and IL-17 receptor signaling are essential for mucosal host defense against oral candidiasis. in The Journal of experimental medicine 2009
Mouse (Murine) Polyclonal IL17F Primary Antibody for ELISA, WB - ABIN1043827
Gomez-Rodriguez, Sahu, Handon, Davidson, Anderson, Kirby, August, Schwartzberg: Differential expression of interleukin-17A and -17F is coupled to T cell receptor signaling via inducible T cell kinase. in Immunity 2009
our data suggest that IL-20 (显示 IL20 抗体) subfamily cytokines, particularly IL-20 (显示 IL20 抗体), IL-22 (显示 IL22 抗体), and IL-24 (显示 IL24 抗体), might provide therapeutic benefit for patients with Diabetic foot ulcers (DFU) .
this study shows that IL-24 (显示 IL24 抗体) promotes Pseudomonas aeruginosa keratitis through the suppression of early protective mucosal immunity
Loss of IL-24 (显示 IL24 抗体) expression is associated with breast cancer.
our results indicate that Th17 cells contribute to the airway vascular remodeling in asthma by mediating EPC (显示 TCF21 抗体) chemotaxis, as well as PMVEC tube formation, via IL-17A (显示 IL17A 抗体) rather than IL-17F.
Data indicate that IL-17A (显示 IL17A 抗体) repressed IL-17F secretion in vitro and in vivo.
A psoriasis-like skin inflammation in mice with epidermis-specific inhibition of NF-kappaB (显示 NFKB1 抗体) was triggered by TNFR1 (显示 TNFRSF1A 抗体)-dependent upregulation of interleukin-24 (显示 IL24 抗体) & activation of STAT3 (显示 STAT3 抗体) signaling in keratinocytes.
The concentrations in plasma of IL-17A (显示 IL17A 抗体), IL-17F, and the IL-17AF heterodimer all were increased greatly in mice after endotoxemia.
IL-24 (显示 IL24 抗体) suppresses the growth of normal vascular smooth muscle cells by inhibiting hydrogen peroxide-induced reactive oxygen species (ROS (显示 ROS1 抗体)) production through the regulation of mitochondrial ROS (显示 ROS1 抗体) production and expression of antioxidant enzymes
IL-17A (显示 IL17A 抗体) and IL-17F secreted by CD4 (显示 CD4 抗体)+Th17 cells specific to lung self-antigens are critical mediators of autoimmunity leading to the pathogenesis of obliterative airway disease.
our study provides an inclusive meta-analysis of the function of IL-17A (显示 IL17A 抗体) rs2275913 and IL-17F rs763780 genetic variations in inflammatory diseases risk. The results indicated that these two genetic variations of IL-17A (显示 IL17A 抗体) and IL-17F were risk factors for inflammatory diseases, particularly RA.
This meta-analysis with seven articles in 697 patients diagnosed with chronic periodontitis , 188 patients diagnosed with aggressive periodontitis and 655 control patients totaling 1540 participants showed a non-significant association between the rs2275913 polymorphism in the IL-17A (显示 IL17A 抗体) gene and the rs763780 polymorphism in the IL-17F gene with the risk of chronic periodontitis, or with aggressive periodontitis
our study provides the evidence that functional IL-23R (显示 IL23R 抗体) rs1884444 G/T and IL-17F rs763780 A/G polymorphisms may be a new genetic susceptibility factor to SLE, especially in the Polish population.
IL23R (显示 IL23R 抗体) rs10889677 and IL17A (显示 IL17A 抗体) rs2275913 were not associated with the susceptibility to Necrotizing enterocolitis (NEC). In conclusion, data suggest that a variant of IL17F (rs763780) may contribute to the development of NEC.
Our results confirmed IL17A (显示 IL17A 抗体) and IL17F as potential candidate genes involved in RA. They play pivoting roles in the susceptibility and in clinical features of RA disease. Responses to RA treatments are differently conditioned by polymorphisms in IL17A (显示 IL17A 抗体) and IL17F genes.
IL17A (显示 IL17A 抗体) and IL17F gene polymorphism are not the important factors associated with susceptibility and some clinical parameters of rheumatoid arthritis in a Polish population.
we aimed to investigate the associations between the 7383A/G and 7488A/G polymorphisms of the interleukin (IL)-17F gene with disease activity and clinical outcomes in Turkish patients with ankylosing spondylitis
Using an in-vitro migration assay, B cells were shown to migrate towards both IL-17A (显示 IL17A 抗体) and IL-17F. These observations indicate a direct chemotactic effect of IL-17 (显示 IL17A 抗体) cytokines on primary peripheral blood B cells with higher effect being on asthmatic B cells.
IL-17 (显示 IL17A 抗体)-related cytokines expression was amplified in bronchial/nasal mucosa of neutrophilic asthma prone to exacerbation, suggesting a pathogenic role of IL-17F in frequent exacerbators.
IL-17A (-197G/A) and IL-17F (7488T/C) SNPs were not associated with susceptibility to rheumatoid arthritis or secondary Sjogrens syndrome (sSS, p > 0.05 for both SNPs). In addition, they did not influence RA activity or clinical markers of SS.
The protein encoded by this gene is a cytokine that shares sequence similarity with IL17. This cytokine is expressed by activated T cells, and has been shown to stimulate the production of several other cytokines, including IL6, IL8, and CSF2/GM_CSF. This cytokine is also found to inhibit the angiogenesis of endothelial cells and induce endothelial cells to produce IL2, TGFB1/TGFB, and monocyte chemoattractant protein-1.
, IL-4-induced secreted protein
, melanoma differentiation-associated gene 7 protein
, th2-specific cytokine FISP
, cytokine-like protein Mob-5
, interleukin 17F
, cytokine ML-1
, mutant IL-17F