Use your antibodies-online credentials, if available.
Select your species
Human BID ELISA Kit for Sandwich ELISA - ABIN365900
Lim, Loh, Ting, Bradshaw, Zeenathul: Antiproliferation and induction of caspase-8-dependent mitochondria-mediated apoptosis by ?-tocotrienol in human lung and brain cancer cell lines. in Biomedicine & pharmacotherapy 2014
Stage-specific expression of TNFalpha (显示 TNF ELISA试剂盒) regulates bad/bid-mediated apoptosis and RIP1 (显示 RALBP1 ELISA试剂盒)/ROS (显示 ROS1 ELISA试剂盒)-mediated secondary necrosis in Birnavirus-infected fish cells.
Hyperosmotic Shock Engages Two Positive Feedback Loops through Caspase-3 (显示 CASP3 ELISA试剂盒)-dependent Proteolysis of JNK1 (显示 MAPK8 ELISA试剂盒)-2 and Bid.
tail regression at metamorphosis implicates an apoptotic pathway inducible by T(3) hormone in an organ autonomous manner and involving the cell death executioners BID and Caspases-2 and -8
Here, we identified and characterized the Xenopus homologs of caspase-10 (显示 CASP10 ELISA试剂盒) (xCaspase-10beta), a novel initiator caspase (显示 CASP3 ELISA试剂盒), and Bid (xBid), a BH3-only (显示 BBC3 ELISA试剂盒) molecule of the Bcl-2 (显示 BCL2 ELISA试剂盒) family involved in both the extrinsic and intrinsic pathways.
diubiquitylation is a specific feature of xBid.
an oligomeric arrangement of Bid, Bax (显示 BAX ELISA试剂盒), and possibly other members of the Bcl-2 (显示 BCL2 ELISA试剂盒) family of proteins that form a self-propagating network that permeabilizes the outer-mitochondrial membrane.
Combined therapy with Seliciclib((R)) and Belinostat((R)) results in eradication of non-small cell lung cancer via apoptosis induction and BID activation.
Results from genome-wide DNA methylation (显示 HELLS PLURAL_@23240@), functional network analysis and pyrosequencing, show selective CpG sites (NOS1AP (显示 NOS1AP PLURAL_@23240@), BID, and GABRB1 (显示 GABRB1 PLURAL_@23240@)) differentially methylated in smokers and chronic obstructive pulmonary disease patients compared to nonsmokers.
The adenovirus-mediated truncated Bid overexpression induced by the Cre/LoxP system can effectively eliminate CD133+ ovarian cancer stem cells, representing a novel therapeutic strategy for the treatment of ovarian cancer.
MiR (显示 MLXIP ELISA试剂盒)-20a-directed regulation of BID in colorectal cancer.
the results establish that cleavage by caspase 8 (显示 CASP8 ELISA试剂盒) and the subsequent association with the outer mitochondrial membrane are two critical events that activate Bid during death receptor-mediated apoptosis.
The results indicate that BID-independent pathways are responsible for FAS (显示 FAS ELISA试剂盒)-dependent human islet cell death in Type 1 diabetes.
The BID-MTCH2 (显示 MTCH2 ELISA试剂盒) axis regulates the differentiation/apoptosis of stem cells and mitochondrial metabolism. (Review)
Data suggest, in models of spatial propagation of mitochondrial permeabilization during apoptosis, there appears to be requirement for cooperative signaling involving truncated-BID and ROS (显示 ROS1 ELISA试剂盒) (reactive oxygen species) for efficient/ robust propagation.
this study emphasizes that the coordinated action of hGzmB-activated p53 (显示 TP53 ELISA试剂盒) and GzmB (显示 Gzmb ELISA试剂盒)-cleaved Bid is important for GzmB (显示 Gzmb ELISA试剂盒)-induced cell death and for cytotoxic lymphocyte/Natural Killer Cell-mediated killing of target cells.
Cleaved Bid did not permeabilize the membrane, but lowered the membrane breakthrough force.
toxic reactive oxygen species levels in Mll5 (显示 MLL5 ELISA试剂盒)(-/-) mice are critically dependent on type 1 interferon (显示 IFNA ELISA试剂盒) (IFN-1) signaling, which triggers mitochondrial accumulation of full-length Bid.
In the present study, we identified the proapoptotic Bcl-2 (显示 BCL2 ELISA试剂盒) family protein Bid as a positive regulator of mutant SOD1 (显示 SOD1 ELISA试剂盒)-induced TLR-nuclear factor-kappaB (NF-kappaB (显示 NFKB1 ELISA试剂盒)) signaling in microglia.
ATM (显示 ATM ELISA试剂盒)-BID-MTCH2 (显示 MTCH2 ELISA试剂盒) pathway that we have identified plays a critical role in the DDR (显示 DDR1 ELISA试剂盒) via regulation of mitochondrial metabolism.
DRP1 (显示 CRMP1 ELISA试剂盒)-dependent apoptotic mitochondrial fission occurs independently of BAX (显示 BAX ELISA试剂盒), BAK (显示 BAK1 ELISA试剂盒) and APAF1 (显示 APAF1 ELISA试剂盒) to amplify cell death by BID and oxidative stress.
A phosphorylation-deficient mutant of BID, MTCH2 (显示 MTCH2 ELISA试剂盒)'s ligand, induces an increase in OXPHOS, but with higher ROS (显示 ROS1 ELISA试剂盒) and reduced ATP levels than Mtch2 (显示 MTCH2 ELISA试剂盒) loss, and is associated with hypersensitivity to irradiation.
Motifs of VDAC2 (显示 VDAC2 ELISA试剂盒) required for mitochondrial Bak (显示 BAK1 ELISA试剂盒) import and tBid-induced apoptosis.
BID protein suppresses p38 (显示 CRK ELISA试剂盒) activity and induce malignant cell transformation of hepatocytes.
compare membrane permeabilization by Bax (显示 BAX ELISA试剂盒) activated by either cBid [cleaved Bid (p7 and p15 (显示 CDKN2B ELISA试剂盒))] or Bim (显示 BCL2L11 ELISA试剂盒) and examine the role of membrane lipids in the recruitment and activation of these three Bcl-2 (显示 BCL2 ELISA试剂盒) pro-apoptotic proteins
Hepatocyte Bid suppression is critical for the resistance to the lethal effects of Fas (显示 FAS ELISA试剂盒) activation. Fas (显示 FAS ELISA试剂盒) signaling induces differential activation of non-canonical interleukin-1beta maturation, amplified in the absence of apoptotic Bid in hepatocytes.
Bid and Bax (显示 BAX ELISA试剂盒) are signal transduction factors in granulosa cells and play proapoptotic roles.
This gene encodes a death agonist that heterodimerizes with either agonist BAX or antagonist BCL2. The encoded protein is a member of the BCL-2 family of cell death regulators. It is a mediator of mitochondrial damage induced by caspase-8 (CASP8)\; CASP8 cleaves this encoded protein, and the COOH-terminal part translocates to mitochondria where it triggers cytochrome c release. Multiple alternatively spliced transcript variants have been found, but the full-length nature of some variants has not been defined.
BH3-interacting domain death agonist
, BID isoform ES(1b)
, BID isoform L(2)
, BID isoform Si6
, Human BID coding sequence
, apoptic death agonist
, desmocollin type 4
, p22 BID
, apoptotic death agonist BID
, BH3 interacting domain death agonist