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抗Human CISD2 抗体:
抗Mouse (Murine) CISD2 抗体:
抗Rat (Rattus) CISD2 抗体:
Human Polyclonal CISD2 Primary Antibody for ICC, IF - ABIN4298808
Stadler, Rexhepaj, Singan, Murphy, Pepperkok, Uhlén, Simpson, Lundberg: Immunofluorescence and fluorescent-protein tagging show high correlation for protein localization in mammalian cells. in Nature methods 2013
The c.103 + 1G > A mutation resulted in the loss of functional CISD2 protein in the two Italian Wolfram syndrome type 2 patients.
CISD2 was up-regulated in laryngeal squamous cell carcinoma
CISD2 could be an independent prognostic factor for PC and suggested that the CISD2/Wnt (显示 WNT2 抗体)/beta-catenin (显示 CTNNB1 抗体) pathway contributed to the proliferation of PC and EMT (显示 ITK 抗体)
the patient that we describe in this report with the c.215A > G missense CISD2 variant had the classical features of Wolfram syndrome type 1. The c.215A > G (p.Asn72Ser) variant does not induce CISD2 RNA mis (显示 AMH 抗体)-splicing or a reduction in CISD2 protein levels. Thus, we report a novel missense homozygous CISD2 mutation in a patient with clinical features that differ from previously reported Wolfram syndrome 2 case reports.
CISD2 exerts anti-apoptotic and anti-inflammatory effects in neural cells; and (2) curcumin can attenuate the downregulation of CISD2 in SCI and LPS (显示 IRF6 抗体)-treated astrocytes.
CISD2 protein may serve as a candidate prognostic marker and a novel therapeutic target for hepatocellular carcinoma (HCC (显示 FAM126A 抗体)) and play an important role in promoting proliferation and enhanced progression of HCC (显示 FAM126A 抗体).
The findings suggested that CISD2 haplotype-tagging single nucleotide polymorphisms are not associated with Alzheimer's disease risk.
NAF-1 (显示 TNIP1 抗体) is a BCL-2 (显示 BCL2 抗体)-associated co-factor that targets BCL-2 (显示 BCL2 抗体) for antagonism of the autophagy pathway at the endoplasmic reticulum.
NAF-1 (显示 TNIP1 抗体) as a previously unidentified cell target of anti-diabetes thiazolidinedione drugs.
provides the first structural information, to our knowledge, for future targeting of the NAF-1 (显示 TNIP1 抗体)-Bcl-2 (显示 BCL2 抗体) complex in the regulation of apoptosis/autophagy in cancer biology
the data establish Tnip1 (显示 TNIP1 抗体) as a critical regulator of IL-17 (显示 IL17A 抗体) biology and reveal a causal role of keratinocytes in the pathogenesis of psoriasis.
These results suggest that the thermosensitive FA-loaded hydrogel could rescue Cisd2(-/-) CM from oxidative stress-induced (显示 SQSTM1 抗体) damage and may have potential applications in the future treatment of CVD.
These studies define a novel role for ABIN1 (显示 TNIP1 抗体) dysfunction and NF-kappaB (显示 NFKB1 抗体) in mediating glomerulonephritis through proinflammatory activation of podocytes.
Suppression of IRAK1 (显示 IRAK1 抗体) or IRAK4 (显示 IRAK4 抗体) Catalytic Activity, but Not Type 1 IFN Signaling, Prevents Lupus Nephritis in Mice Expressing a Ubiquitin Binding-Defective Mutant of ABIN1 (显示 TNIP1 抗体)
NAF-1 (显示 TNIP1 抗体), a BCL-2 (显示 BCL2 抗体)-associated autophagy regulator, is required for homeostatic maintenance of skeletal muscle
Cisd2 deficiency impairs the activation of Wnt (显示 WNT2 抗体)/beta-catenin (显示 CTNNB1 抗体) signaling
These results suggest that the ABIN1 (显示 TNIP1 抗体)-mediated hyperactivation of IKKs and MAPKs might mediate chronic inflammation and CVD development.
Results suggest that that Cisd2 plays an important role in intracellular Ca21 homeostasis, which is required for the differentiation and functioning of adipocytes as well as the regulation of glucose homeostasis in mice.
Miner1 is a redox protein that resides in the ER and that regulates the UPR and mitochondrial function.
studies link ABIN-1 (显示 TNIP1 抗体) with IL-23 (显示 IL23A 抗体) and IL-17 (显示 IL17A 抗体), and they provide cellular and molecular mechanisms by which ABIN-1 (显示 TNIP1 抗体) regulates susceptibility to psoriasis.
The protein encoded by this gene is a zinc finger protein that localizes to the endoplasmic reticulum. The encoded protein binds an iron/sulfur cluster and may be involved in calcium homeostasis. Defects in this gene are a cause of Wolfram syndrome 2.
CDGSH iron-sulfur domain-containing protein 2A
, CDGSH iron sulfur domain-containing protein 2A
, CDGSH iron sulfur domain-containing protein 2-A
, zinc finger, CDGSH-type domain 2
, CDGSH iron-sulfur domain-containing protein 2
, CDGSH iron sulfur domain-containing protein 2
, CDGSH iron sulfur domain-containing protein 2-B
, CDGSH iron sulfur domain-containing protein 2B
, CDGSH iron-sulfur domain-containing protein 2B
, endoplasmic reticulum intermembrane small protein
, mitoNEET related 1
, mitoNEET-related 1 protein
, nutrient-deprivation autophagy factor-1
, CDGSH iron sulfur domain 2
, nervous system over-expressed protein 70
, nervous system overexpressed protein 70
, H/ACA ribonucleoprotein complex non-core subunit NAF1
, nuclear assembly factor 1 homolog
, A20-binding inhibitor of NF-kappa B activation
, Nef-associated factor 1
, TNFAIP3-interacting protein 1
, virion-associated nuclear shuttling protein