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抗Human TLR9 抗体:
抗Mouse (Murine) TLR9 抗体:
抗Rat (Rattus) TLR9 抗体:
Dog (Canine) Monoclonal TLR9 Primary Antibody for CyTOF, ELISA - ABIN4360430
Gantner, Hermann, Nakashima, Matsukawa, Sakai, Bacon: CD40-dependent and -independent activation of human tonsil B cells by CpG oligodeoxynucleotides. in European journal of immunology 2003
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Dog (Canine) Monoclonal TLR9 Primary Antibody for FACS, ICC - ABIN4360434
Dillmann, Ringel, Ringleb, Mora, Olesch, Fink, Roberts, Brüne, Weigert: S1PR4 Signaling Attenuates ILT 7 Internalization To Limit IFN-α Production by Human Plasmacytoid Dendritic Cells. in Journal of immunology (Baltimore, Md. : 1950) 2016
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Dog (Canine) Monoclonal TLR9 Primary Antibody for CyTOF, ELISA - ABIN4360432
Damiano, Caputo, Bianco, DArmiento, Leonardi, De Placido, Bianco, Agrawal, Ciardiello, Tortora: Novel toll-like receptor 9 agonist induces epidermal growth factor receptor (EGFR) inhibition and synergistic antitumor activity with EGFR inhibitors. in Clinical cancer research : an official journal of the American Association for Cancer Research 2006
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Dog (Canine) Monoclonal TLR9 Primary Antibody for ELISA - ABIN4360433
Abel, Wang, Fritts, Sanchez, Chung, Fitzgerald-Bocarsly, Krieg, Miller et al.: Deoxycytidyl-deoxyguanosine oligonucleotide classes A, B, and C induce distinct cytokine gene expression patterns in rhesus monkey peripheral blood mononuclear cells and distinct alpha interferon ... in Clinical and diagnostic laboratory immunology 2005
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Human Monoclonal TLR9 Primary Antibody for FACS, IF - ABIN2191964
Ahmad-Nejad, Häcker, Rutz, Bauer, Vabulas, Wagner: Bacterial CpG-DNA and lipopolysaccharides activate Toll-like receptors at distinct cellular compartments. in European journal of immunology 2002
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Human Monoclonal TLR9 Primary Antibody for FACS, IF - ABIN2191962
Rumio, Besusso, Palazzo, Selleri, Sfondrini, Dubini, Ménard, Balsari: Degranulation of paneth cells via toll-like receptor 9. in The American journal of pathology 2004
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Human Monoclonal TLR9 Primary Antibody for FACS, IF - ABIN2191963
Rutz, Metzger, Gellert, Luppa, Lipford, Wagner, Bauer: Toll-like receptor 9 binds single-stranded CpG-DNA in a sequence- and pH-dependent manner. in European journal of immunology 2004
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Mouse (Murine) Polyclonal TLR9 Primary Antibody for IHC - ABIN967189
Hemmi, Takeuchi, Kawai, Kaisho, Sato, Sanjo, Matsumoto, Hoshino, Wagner, Takeda, Akira: A Toll-like receptor recognizes bacterial DNA. in Nature 2000
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Mouse (Murine) Polyclonal TLR9 Primary Antibody for IHC - ABIN967190
Bauer, Kirschning, Häcker, Redecke, Hausmann, Akira, Wagner, Lipford: Human TLR9 confers responsiveness to bacterial DNA via species-specific CpG motif recognition. in Proceedings of the National Academy of Sciences of the United States of America 2001
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Dog (Canine) Monoclonal TLR9 Primary Antibody for ELISA, FACS - ABIN4360452
Tengroth, Arebro, Kumlien Geor��n, Winqvist, Cardell: Deprived TLR9 expression in apparently healthy nasal mucosa might trigger polyp-growth in chronic rhinosinusitis patients. in PLoS ONE 2014
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TLR4 (显示 TLR4 抗体) and TLR9 mRNA were elevated in blood samples from celiac disease patients compared to the healthy controls
Finding suggest that genetic variations in TLR9 (rs352139 and rs352140) are associated with severity and prognosis of bacterial meningitis in Chinese children.
The findings show that the expression of TLR2, TLR4 (显示 TLR4 抗体) and TLR9, and hypoxia markers HIF-1alpha (显示 HIF1A 抗体) and CAIX (显示 CA9 抗体) is abnormal in pancreatic intraepithelial neoplasia suggesting that both the innate immunity activation and hypoxia response are involved in early pancreatic carcinogenesis.
Genotyping was performed to detect -T1237C (rs5743836) and G2848A (rs352140) SNP in TLR9 gene in patients with pulmonary tuberculosis and healthy controls. The A allele of G2848A SNP in TLR9 gene was found with a marginally higher frequency among tuberculosis patients.
The TLR3 (显示 TLR3 抗体).rs3775290 "CC" genotype was associated with hepatitis C virus chronicity, while the TLR9 gene may not play a major role in hepatitis C virus infection.
Identify both expansion of T-bet(+) B cells and production of anti-erythrocyte antibodies in ex vivo cultures of naive peripheral blood mononuclear cells exposed to P. falciprum infected erythrocyte lysates. We propose that synergistic TLR9/IFN-gammaR activation of T-bet(+) B cells is a mechanism underlying infection-induced autoimmune-like responses.
TLR9, especially TLR9/CD3 (显示 CD3 抗体), is highly expressed in systemic sclerosis patients particularly those with diffuse cutaneous subtype and could form a potential marker for skin fibrosis and disease severity.
TLR9 Gene Polymorphism is not associated with Chronic Lymphocytic Leukemia.
TLR-9 gene rs187084 polymorphism is positively correlated with susceptibility to Osteoarthritis of Knee, especially among old people in a Chinese population.
Data showed strong interactions of toll (显示 TLR4 抗体)-like receptors TLR2 and 4 associated with Leishmania(V.) braziliensis, while toll (显示 TLR4 抗体)-like receptor TLR9, in contrast, a strong interaction with Leishmania (L.) amazonensis.
Study shows that placental and/or fetal DNA stimulates toll-like receptor 9 (TLR9) leading to secretion of pro-inflammatory cytokines by macrophage cells.
Plasmodium DNA induces autoreactive responses against erythrocytes by activating a population of B cells expressing CD11c and the transcription factor T-bet, which become major producers of autoantibodies that promote malarial anaemia.
Our findings suggested the collaboration of TLR2-TLR9 at least in the regulation of SARM (显示 SARM1 抗体) expression.
High cholesterol intake exacerbated acetaminophen-induced acute liver injury via the accumulation of free cholesterol in the endolysosomes of liver sinusoidal endothelial cells. This accumulation enhanced Toll-like receptor 9 signaling via impairment of its membrane trafficking mechanism.
these data provide a previously uncharacterized in vivo mechanism contingent on oligodeoxy-nucleotide -administered dose, where TLR9 governs the primary response and RAGE (显示 AGER 抗体) plays a distinct and cooperative function in providing a pivotal role in balancing the immune response.
this study shows that CD205 (显示 LY75 抗体)-TLR9-IL-12 (显示 IL12A 抗体) axis contributes to CpG-induced oversensitive liver injury in Hepatitis B viral antigens transgenic mice by promoting the interaction of NKT (显示 CTSL1 抗体) cells with Kupffer cells
The findings support a salutary role of TLR9 in some subsets of HF conditions and underline the importance for future studies on the mechanisms of TLR9 in diastolic HF.
TLR9-driven inflammation induced a Ccr2-independent expansion of functionally enhanced extramedullary myeloid progenitors that correlated with the peripheral accumulation of monocytes in both wild-type and Ccr2(-/-) mice.
TLR9 expression on B1a cells is not critical for their IgM-dependent atheroprotection.
This study establishes a correlation between TLR-3 (显示 TLR3 抗体) and TLR-9 expression with the development of EAE. In addition, evidence of a role for the myelin peptide in targeting the innate inflammatory response to the CNS is presented.
these results indicate clear responses of porcine neonatal antigen presenting cells after TLR2 and TLR9 stimulation
Porcine parvovirus virus infection significantly induced IL-6 (显示 IL6 抗体) expression and this induction depended on NF-kappaB (显示 NFKB1 抗体) activation and TLR9 signaling pathways in PK-15 cell.
TLR9 immunoreactivity is mainly detected in epithelial cells and antigen presenting cells, namely dendritic and macrophage-like cells, within the range of tissues examined. The pattern of TLR9 expression was similar in pigs of 3 weeks and 3 months of age.
These data demonstrated that TLR2, TLR3 (显示 TLR3 抗体) and TLR9 contribute to NF-kappaB (显示 NFKB1 抗体) activation in response to porcine epidemic diarrhea virus infection, but not RIG-I (显示 DDX58 抗体).
expression of TLR3 (显示 TLR3 抗体), TLR7 (显示 TLR7 抗体) and TLR9 in alveolar macrophages infected with different genotype 1 PRRSV strains
we studied TLR9 expression in lung extracts from pigs, dogs, and cattle.
Moreover, the TLR9 transfectant demonstrated its usefulness for evaluation of immunostimulation by bacterial DNA through the detection of T(H)-1, T(H)-2 type cytokine induction via TLR9 signaling.
variants in the TLR1 (显示 TLR1 抗体) gene are associated with susceptibility to bovine tuberculosis, whereas no significant association can be inferred from the polymorphisms in the TLR9 gene
mRNA abundances of TLR9, TLR2, and TLR4 together with those of beta-defensin 5 (BNBD5), an early bactericidal effector molecule of the innate system, in healthy and infected mammary glands
substantial conservation of TLR9 structure and TLR9 function in blood leukocytes
The protein encoded by this gene is a member of the Toll-like receptor (TLR) family which plays a fundamental role in pathogen recognition and activation of innate immunity. TLRs are highly conserved from Drosophila to humans and share structural and functional similarities. They recognize pathogen-associated molecular patterns (PAMPs) that are expressed on infectious agents, and mediate the production of cytokines necessary for the development of effective immunity. The various TLRs exhibit different patterns of expression. This gene is preferentially expressed in immune cell rich tissues, such as spleen, lymph node, bone marrow and peripheral blood leukocytes. Studies in mice and human indicate that this receptor mediates cellular response to unmethylated CpG dinucleotides in bacterial DNA to mount an innate immune response.
Toll-like receptor 9 protein