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investigated neural progenitor cells in respect to their glycine receptor (显示 GLRB ELISA试剂盒) function and subunit expression using electrophysiology, calcium imaging, immunocytochemistry, and quantitative real-time PCR
Enhancement of azurophil granule-phagosome fusion via glycine receptor alpha 2 subunit (GlyRalpha2)/transient receptor potential melastatin (显示 TRPM1 ELISA试剂盒) (TRPM)2 (显示 CLU ELISA试剂盒)/p38 MAP kinase (显示 MAPK14 ELISA试剂盒) signaling is a novel target for enhancement of neutrophil bactericidal activity.
Effects of 12 times normal atmospheric pressure of helium-oxygen gas (pressure) on ethanol-induced potentiation of GlyR function in Xenopus oocytes expressing human alpha1, alpha2 or the mutant alpha1(A52S) GlyRs were measured by voltage clamp technics
The molecular basis for the differential sensitivity of GlyR alpha(1) and GlyR alpha(2) to Zn(2+) potentiation is reported.
GcgR (显示 GCGR ELISA试剂盒) knockout (Gcgr (显示 GCGR ELISA试剂盒)(-/-)) mice displayed lower blood glucose levels accompanied by elevated plasma ghrelin (显示 GHRL ELISA试剂盒) levels. Hyperglycemia was averted in streptozocin treated Gcgr (显示 GCGR ELISA试剂盒)(-/-) mice and the plasma ghrelin (显示 GHRL ELISA试剂盒) level was further increased.
Findings show that the loss of embryonic GlyRalpha2 ultimately impairs the formation of cortical circuits in the mature brain; results demonstrate a physiological role for alpha2 GlyRs (显示 GARS ELISA试剂盒) during cortical network formation and homeostasis
glucagon receptor (显示 GCGR ELISA试剂盒) antagonist improves glycemia in diet-induced obese angptl4 (显示 ANGPTL4 ELISA试剂盒) knockout mice without increasing glucagon (显示 GCG ELISA试剂盒) levels or alpha-cell proliferation, underscoring the importance of this protein.
genetic inactivation of Glra2, the gene coding the alpha2 subunit of GlyRs (显示 GARS ELISA试剂盒), disrupts dorsal cortical progenitor homeostasis with an impaired capability of apical progenitors to generate basal progenitors.
Data indicate that the exocrine pancreas in the glucagon receptor Gcgr (显示 GCGR ELISA试剂盒)-/- mice exhibited larger nuclear size than in WT or heterozygous controls, most obviously at old ages.
Simultaneous and sufficient activation of GLP1R (显示 GLP1R ELISA试剂盒) is required to reduce GCCR (显示 NR3C1 ELISA试剂盒) mediated blood glucose elevation following administration of a GLP1R (显示 GLP1R ELISA试剂盒)/GCGR (显示 GCGR ELISA试剂盒) co-agonist.
Knockdown of liver glucagon receptor (显示 GCGR ELISA试剂盒) in mice reduces blood glucose and increases blood LDL levels.
Endogenous activation of Glra2 promotes neuronal migration by regulating nucleokinesis.
Gcgr (显示 GCGR ELISA试剂盒)(-/-) mice became lethargic (显示 CACNB4 ELISA试剂盒) & cachexic & died early. Autopsy revealed numerous PNETs up to 15 mm in diameter in most well-preserved Gcgr (显示 GCGR ELISA试剂盒)(-/-) pancreata.
Data suggest that GcgR (显示 GCGR ELISA试剂盒) activation raises hepatic expression of fibroblast growth factor 21 (FGF21 (显示 FGF21 ELISA试剂盒)) and increases circulating levels of FGF21 (显示 FGF21 ELISA试剂盒); GcgR (显示 GCGR ELISA试剂盒) activation induces body weight loss and stimulates lipid metabolism.
The glycine receptor consists of two subunits, alpha and beta, and acts as a pentamer. The protein encoded by this gene is an alpha subunit and can bind strychnine. Several transcript variants encoding different isoforms have been found for this gene.
glycine receptor alpha 2 subunit
, glycine receptor subunit alpha-2
, glycine receptor, alpha-2 polypeptide
, Glycine receptor alpha 2 subunit (glycine receptor, neonatal)
, glycine receptor strychnine-binding subunit
, glycine receptor, alpha 2 subunit
, glycine receptor, alpha 2
, glycin receptor,alpha 2
, glycine receptor subunit alpha-2-like
, glucagon receptor perhaps same as Niddm3