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抗Human p38 抗体:
抗Rat (Rattus) p38 抗体:
抗Mouse (Murine) p38 抗体:
Human Polyclonal p38 Primary Antibody for IF (cc), IF (p) - ABIN671241
Li, Dong, Song, Xu, Liu, Song: Nrf2/ARE pathway activation, HO-1 and NQO1 induction by polychlorinated biphenyl quinone is associated with reactive oxygen species and PI3K/AKT signaling. in Chemico-biological interactions 2014
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Human Polyclonal p38 Primary Antibody for ELISA, ICC - ABIN6267706
Liu, Zheng, Zhang, Wang, Yang, Bai, Dai: Fucoxanthin Activates Apoptosis via Inhibition of PI3K/Akt/mTOR Pathway and Suppresses Invasion and Migration by Restriction of p38-MMP-2/9 Pathway in Human Glioblastoma Cells. in Neurochemical research 2017
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Human Polyclonal p38 Primary Antibody for IF (p), IHC (p) - ABIN710141
Zhao, Liu, Liu, Han, Zhao: Betulin attenuates lung and liver injuries in sepsis. in International immunopharmacology 2015
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Human Polyclonal p38 Primary Antibody for ELISA, ICC - ABIN6263958
Zou, Xiang, Wang, Peng, Wei: Oregano Essential Oil Improves Intestinal Morphology and Expression of Tight Junction Proteins Associated with Modulation of Selected Intestinal Bacteria and Immune Status in a Pig Model. in BioMed research international 2017
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Human Monoclonal p38 Primary Antibody for ICS - ABIN1177122
Brunet, Pouysségur: Identification of MAP kinase domains by redirecting stress signals into growth factor responses. in Science (New York, N.Y.) 1996
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Human Polyclonal p38 Primary Antibody for ELISA, ICC - ABIN6255833
Jin, Han, Yang, Hu, Liu, Zhao: 11-O-acetylcyathatriol inhibits MAPK/p38-mediated inflammation in LPS-activated RAW 264.7 macrophages and has a protective effect on ethanol-induced gastric injury. in Molecular medicine reports 2017
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Human Polyclonal p38 Primary Antibody for IHC, IHC (p) - ABIN152964
Ito, Miyado, Nakagawa, Muraki, Imai, Yamakawa, Qin, Hosoi, Saito, Takahashi: Age-associated changes in the subcellular localization of phosphorylated p38 MAPK in human granulosa cells. in Molecular human reproduction 2010
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Human Monoclonal p38 Primary Antibody for ICS - ABIN1177120
Han, Lee, Bibbs, Ulevitch: A MAP kinase targeted by endotoxin and hyperosmolarity in mammalian cells. in Science (New York, N.Y.) 1994
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Human Monoclonal p38 Primary Antibody for ICS - ABIN1177124
Winston, Chan, Johnson, Riches: Activation of p38mapk, MKK3, and MKK4 by TNF-alpha in mouse bone marrow-derived macrophages. in Journal of immunology (Baltimore, Md. : 1950) 1997
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Immune profiling of human prostate epithelial cells in health and pathology determined by expression of p38 (显示 CRK 抗体)/TRAF-6 (显示 TRAF6 抗体)/ERK (显示 EPHB2 抗体) MAP kinases pathways has been reported.
The cytotoxicity induced by EB1 (显示 MAPRE2 抗体) gene knockdown was due to the activation and generation of reactive oxygen species by p38 mitogen-activated protein kinase (显示 MAPK14 抗体)..this signaling cascade, however not nuclear factor-kappaB-mediated signaling, induced the expression of cyclooxygenase-2 (显示 PTGS2 抗体), a key effector of apoptotic death.
Data, including data using network analysis, suggest that angiotensinogen (AGT (显示 AGT 抗体)), mitogen-activated protein kinase-14 (MAPK14 (显示 MAPK14 抗体)), and prothrombin (显示 F2 抗体) (F2) in placental villous tissues are core factors in early embryonic development; these studies involved proteomics and bioinformatics analysis of altered protein expression in placental villous tissue from early recurrent miscarriage patients in comparison to control tissues.
The role of p38 MAP kinase (显示 MAPK14 抗体) signaling in metastatic clear cell renal cell carcinoma (显示 MOK 抗体)
Rhythmic luciferase activity from clock gene luciferase reporter cells lines was used to test the effect of p38 MAPK (显示 MAPK14 抗体) inhibition on clock properties as determined using the damped sine fit and Levenberg-Marquardt algorithm.Glioma treatment with p38 MAPK (显示 MAPK14 抗体) inhibitors may be more effective and less toxic if administered at the appropriate time of the day.
Hsp27 (显示 HSPB1 抗体) and P38MAPK (显示 MAPK14 抗体) could be used as prognostic factors in Esophageal squamous cell carcinoma.
High p38MAPK (显示 MAPK14 抗体) expression is associated with non-small cell lung cancer metastasis.
when the cells were treated with SB203580, an inhibitor of the p38 MAPK (显示 MAPK14 抗体) pathway, the osteogenic effects of Epo (显示 EPO 抗体) on hPDLSCs and pPDLSCs were attenuated. In conclusion, Epo (显示 EPO 抗体) may upregulate the bone formation ability of hPDLSCs and pPDLSCs via the p38 MAPK (显示 MAPK14 抗体) pathways
p38alpha (显示 MAPK14 抗体) and ATF2 (显示 ATF2 抗体) expression play a crucial role in the malignant phenotypes of ovarian tumor cells and are a markers of poor prognosis in patients with ovarian serous adenocarcinomas.
KLF4 (显示 KLF4 抗体) overcomes tamoxifen resistance by suppressing MAPK (显示 MAPK1 抗体) signaling pathway and predicts good prognosis in breast cancer.
results suggest that ET-1 (显示 EDN1 抗体)-induced activation of proMMP-2 is mediated via cross-talk between NADPH oxidase (显示 NOX1 抗体)-PKCalpha (显示 PKCa 抗体)-p(38)MAPK (显示 MAPK1 抗体) and NFkappaB-MT1MMP (显示 MMP14 抗体) signaling pathways along with a marked decrease in TIMP-2 (显示 TIMP2 抗体) expression in the cells
cross-talk between p(38)MAPK (显示 MAPK1 抗体) and Gialpha play a pivotal role for full activation of cPLA2 (显示 PLA2G4A 抗体) during ET-1 (显示 EDN1 抗体) stimulation of pulmonary artery smooth muscle cells.
MAPK14 (显示 MAPK14 抗体) signalling pathway is largely involved in heat-induced sperm damage.
p38 MAPK (显示 MAPK14 抗体) is an early redox sensor in the laminar shear stress with hydrogen peroxide being a signaling mediator.
Blockade of p38 (显示 MAPK14 抗体) enhances chondrocyte phenotype in monolayer culture and may promote more efficient cartilage tissue regeneration for cell-based therapies.
p38 (显示 MAPK14 抗体) phosphorylation and MMP13 (显示 MMP13 抗体) expression are regulated by Rho/ROCK activation, and support the potential novel pathway that Rho/ROCK is in the upper part of the mechanical stress-induced matrix degeneration cascade in cartilage.
These data suggest that the p38 (显示 MAPK14 抗体) and JNK (显示 MAPK8 抗体) signaling pathways play pivotal roles in PRRSV replication and may regulate immune responses during virus infection.
findings support the hypothesis that ischemic factor stimulation of the blood-brain barrier Na-K-Cl cotransporter (显示 SLC12A1 抗体) involves activation of p38 (显示 MAPK14 抗体) and JNK (显示 MAPK8 抗体) MAPKs
These data suggest a differential requirement of JNK1 (显示 MAPK8 抗体) and p38 MAPK (显示 MAPK14 抗体) in TNF (显示 TNF 抗体) regulation of E2F1 (显示 E2F1 抗体). Targeted inactivation of JNK1 (显示 MAPK8 抗体) at arterial injury sites may represent a potential therapeutic intervention for ameliorating TNF (显示 TNF 抗体)-mediated EC dysfunction.
p38 MAPK (MAPK14 (显示 MAPK14 抗体)) is redox-regulated in reactive oxygen species-dependent endothelial barrier dysfunction.
the present findings suggested that artesunate may exert protective effects against cerebral ischemia/reperfusion injury through the suppression of oxidative and inflammatory processes, via activating Nrf2 (显示 NFE2L2 抗体) and downregulating ROSdependent p38 MAPK (显示 MAPK14 抗体) in mice.
Results show that the p38 MAPK (显示 MAPK14 抗体) signaling pathway could regulate mitochondria Abeta (显示 APP 抗体) internalization by manipulating the expression of alpha7nAChR. Pretreatment of alpha7nAChR agonist could attenuate these biochemical changes which are tightly associated with Abeta1-42 induced apoptosis. Suggesting there is an endogenous, previously unrecognized cholinergic mechanism to control mitochondria functions and their apoptotic ...
Prdx1 (显示 PRDX1 抗体) knockout can aggravate the oxidative stress and lung injury by increasing the level of Reactive Oxygen Species (ROS (显示 ROS1 抗体)), and also activate P38 (显示 CRK 抗体)/JNK (显示 MAPK8 抗体) signaling pathway.
P38 (显示 CRK 抗体) kinase role in the inflammatory pain.CXCL13, upregulated by peripheral inflammation, acts on CXCR5 (显示 CXCR5 抗体) on dorsal root ganglia neurons and activates p38 (显示 CRK 抗体), which increases Nav1.8 (显示 SCN10A 抗体) current density and further contributes to the maintenance of inflammatory pain.
these findings indicate that BMS309403 reduces fatty acid-induced ER stress-associated inflammation in skeletal muscle by reducing p38 MAPK (显示 MAPK14 抗体) activation.
report insulin-like growth factor-II binding protein 1 (IGF2BP1 (显示 B4GALNT2 抗体)) as a novel interacting partner of p38 MAPK (显示 MAPK14 抗体).
These results were supported by the opposite outcomes observed for cells treated with A779 or DX600. Therefore, it was concluded that the ACE2 (显示 ACE2 抗体)-Ang (显示 ANG 抗体)(17)-Mas (显示 MAS1 抗体) axis significantly inhibits pancreatitis by inhibition of the p38 MAPK (显示 MAPK14 抗体)/NF-kappaB (显示 NFKB1 抗体) signaling pathway
results suggest that c-Jun (显示 JUN 抗体), p38 MAPK (显示 MAPK14 抗体), PIK3CA (显示 PIK3CA 抗体)/Akt (显示 AKT1 抗体), and GSK3 signaling involved in the effect of miR (显示 MLXIP 抗体)-203 on the proliferation of hepatocellular carcinoma cells.
The Macrophage Activation Induced by Bacillus thuringiensis Cry1Ac Protoxin Involves ERK1/2 and p38 (显示 CRK 抗体) Pathways and the Interaction with Cell-Surface-HSP70 (显示 HSP70 抗体)
MAPK (显示 MAPK1 抗体) in, and found that p38alpha (显示 MAPK14 抗体) deficiency causes Th1 (显示 HAND1 抗体) cells to hyperproliferate via the Mnk1 (显示 MKNK1 抗体)/eIF4E (显示 EIF4E 抗体) pathway
P38 (显示 MAPK14 抗体) and JNK (显示 MAPK8 抗体) have opposing effects on persistence of in vivo leukocyte migration in zebrafish.
Adult zebrafish cardiomyocytes express active p38alpha (显示 MAPK14 抗体) MAPK (显示 MAPK1 抗体), which is switched off upon entry into mitosis.
Dkk3r regulates p38a (显示 MAPK14 抗体) phosphorylation to maintain Smad4 (显示 SMAD4 抗体) stability, in turn enabling the Smad2 (显示 SMAD2 抗体).Smad3a.Smad4 complex to form and activate the myf5 (显示 MYF5 抗体) promoter.
The protein encoded by this gene is a member of the MAP kinase family. MAP kinases act as an integration point for multiple biochemical signals, and are involved in a wide variety of cellular processes such as proliferation, differentiation, transcription regulation and development. This kinase is activated by various environmental stresses and proinflammatory cytokines. The activation requires its phosphorylation by MAP kinase kinases (MKKs), or its autophosphorylation triggered by the interaction of MAP3K7IP1/TAB1 protein with this kinase. The substrates of this kinase include transcription regulator ATF2, MEF2C, and MAX, cell cycle regulator CDC25B, and tumor suppressor p53, which suggest the roles of this kinase in stress related transcription and cell cycle regulation, as well as in genotoxic stress response. Four alternatively spliced transcript variants of this gene encoding distinct isoforms have been reported.
Csaids binding protein
, MAP kinase 14
, MAP kinase 2
, MAP kinase Mxi2
, MAP kinase p38 alpha
, MAPK 14
, MAX-interacting protein 2
, cytokine suppressive anti-inflammatory drug binding protein
, cytokine-supressive anti-inflammatory drug binding protein
, mitogen-activated protein kinase 14
, mitogen-activated protein kinase 14A
, mitogen-activated protein kinase p38 alpha
, p38 MAP kinase
, p38 mitogen activated protein kinase
, p38alpha Exip
, reactive kinase
, stress-activated protein kinase 2A
, cytokine suppressive anti-inflammatory drug binding protein 1
, mitogen activated protein kinase 14
, p38 MAP kinase alpha
, p38 MAPK
, p38 alpha
, tRNA synthetase cofactor p38
, MAP kinase 14A
, MAP kinase p38a
, MAPK 14A
, Mitogen-activated protein kinase p38a
, mitogen-activated protein kinase p38a