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抗Human p38 抗体:
抗Rat (Rattus) p38 抗体:
抗Mouse (Murine) p38 抗体:
Human Polyclonal p38 Primary Antibody for IF (cc), IF (p) - ABIN671241
Li, Dong, Song, Xu, Liu, Song: Nrf2/ARE pathway activation, HO-1 and NQO1 induction by polychlorinated biphenyl quinone is associated with reactive oxygen species and PI3K/AKT signaling. in Chemico-biological interactions 2014
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Human Polyclonal p38 Primary Antibody for IF (p), IHC (p) - ABIN710141
Zhao, Liu, Liu, Han, Zhao: Betulin attenuates lung and liver injuries in sepsis. in International immunopharmacology 2015
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Human Monoclonal p38 Primary Antibody for ICS - ABIN1177122
Brunet, Pouysségur: Identification of MAP kinase domains by redirecting stress signals into growth factor responses. in Science (New York, N.Y.) 1996
Show all 5 Pubmed References
Human Polyclonal p38 Primary Antibody for IHC, IHC (p) - ABIN152964
Ito, Miyado, Nakagawa, Muraki, Imai, Yamakawa, Qin, Hosoi, Saito, Takahashi: Age-associated changes in the subcellular localization of phosphorylated p38 MAPK in human granulosa cells. in Molecular human reproduction 2010
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Human Polyclonal p38 Primary Antibody for IHC, WB - ABIN362314
Zheng, Reynolds, Jo, Wersto, Han, Xiao: Intracellular acidosis-activated p38 MAPK signaling and its essential role in cardiomyocyte hypoxic injury. in FASEB journal : official publication of the Federation of American Societies for Experimental Biology 2005
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Human Monoclonal p38 Primary Antibody for ICS - ABIN1177118
Han, Lee, Bibbs, Ulevitch: A MAP kinase targeted by endotoxin and hyperosmolarity in mammalian cells. in Science (New York, N.Y.) 1994
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Human Monoclonal p38 Primary Antibody for ICS - ABIN1177121
Winston, Chan, Johnson, Riches: Activation of p38mapk, MKK3, and MKK4 by TNF-alpha in mouse bone marrow-derived macrophages. in Journal of immunology (Baltimore, Md. : 1950) 1997
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Hsp27 (显示 HSPB1 抗体) and P38MAPK (显示 MAPK14 抗体) could be used as prognostic factors in Esophageal squamous cell carcinoma.
High p38MAPK (显示 MAPK14 抗体) expression is associated with non-small cell lung cancer metastasis.
when the cells were treated with SB203580, an inhibitor of the p38 MAPK (显示 MAPK14 抗体) pathway, the osteogenic effects of Epo (显示 EPO 抗体) on hPDLSCs and pPDLSCs were attenuated. In conclusion, Epo (显示 EPO 抗体) may upregulate the bone formation ability of hPDLSCs and pPDLSCs via the p38 MAPK (显示 MAPK14 抗体) pathways
p38alpha (显示 MAPK14 抗体) and ATF2 (显示 ATF2 抗体) expression play a crucial role in the malignant phenotypes of ovarian tumor cells and are a markers of poor prognosis in patients with ovarian serous adenocarcinomas.
KLF4 (显示 KLF4 抗体) overcomes tamoxifen resistance by suppressing MAPK (显示 MAPK1 抗体) signaling pathway and predicts good prognosis in breast cancer.
These results suggest that PYP treatment had a preventive effect on nephrotoxicity, specifically by downregulating the MAPK and NFkappaB signaling pathways and the mRNA levels of inflammatory genes
hepatic p38alpha (显示 MAPK14 抗体) MAPK (显示 MAPK1 抗体) functions as a negative regulator of liver steatosis in maintaining hepatic bile acid synthesis and fatty acid beta-oxidation by antagonizing the c-Jun N-terminal kinase (JNK).
The results reveal a new connection between p38MAPK (显示 MAPK14 抗体), MYC (显示 MYC 抗体) and NOTCH (显示 NOTCH1 抗体) signaling, demonstrate two mechanisms of NOTCH3 (显示 NOTCH3 抗体) regulation and provide evidence for NOTCH3 (显示 NOTCH3 抗体) involvement in prostate luminal cell differentiation.
Overall, these results suggest that p53 (显示 TP53 抗体) is involved in improving insulin (显示 INS 抗体) sensitivity of hepatic cells via inhibition of mitogen-activated protein kinases (MAPKs) and NF-kappaB (显示 NFKB1 抗体) pathways.
Data show that the combination of targeting RAD51 (显示 RAD51 抗体) and p38 (显示 CRK 抗体) inhibits cell proliferation both in vitro and in vivo, which was further enhanced by targeting of PARP1 (显示 PARP1 抗体).
results suggest that ET-1 (显示 EDN1 抗体)-induced activation of proMMP-2 is mediated via cross-talk between NADPH oxidase (显示 NOX1 抗体)-PKCalpha (显示 PKCa 抗体)-p(38)MAPK (显示 MAPK1 抗体) and NFkappaB-MT1MMP (显示 MMP14 抗体) signaling pathways along with a marked decrease in TIMP-2 (显示 TIMP2 抗体) expression in the cells
cross-talk between p(38)MAPK (显示 MAPK1 抗体) and Gialpha play a pivotal role for full activation of cPLA2 (显示 PLA2G4A 抗体) during ET-1 (显示 EDN1 抗体) stimulation of pulmonary artery smooth muscle cells.
MAPK14 (显示 MAPK14 抗体) signalling pathway is largely involved in heat-induced sperm damage.
p38 MAPK (显示 MAPK14 抗体) is an early redox sensor in the laminar shear stress with hydrogen peroxide being a signaling mediator.
Blockade of p38 (显示 MAPK14 抗体) enhances chondrocyte phenotype in monolayer culture and may promote more efficient cartilage tissue regeneration for cell-based therapies.
p38 (显示 MAPK14 抗体) phosphorylation and MMP13 (显示 MMP13 抗体) expression are regulated by Rho/ROCK activation, and support the potential novel pathway that Rho/ROCK is in the upper part of the mechanical stress-induced matrix degeneration cascade in cartilage.
These data suggest that the p38 (显示 MAPK14 抗体) and JNK (显示 MAPK8 抗体) signaling pathways play pivotal roles in PRRSV replication and may regulate immune responses during virus infection.
findings support the hypothesis that ischemic factor stimulation of the blood-brain barrier Na-K-Cl cotransporter (显示 SLC12A1 抗体) involves activation of p38 (显示 MAPK14 抗体) and JNK (显示 MAPK8 抗体) MAPKs
These data suggest a differential requirement of JNK1 (显示 MAPK8 抗体) and p38 MAPK (显示 MAPK14 抗体) in TNF (显示 TNF 抗体) regulation of E2F1 (显示 E2F1 抗体). Targeted inactivation of JNK1 (显示 MAPK8 抗体) at arterial injury sites may represent a potential therapeutic intervention for ameliorating TNF (显示 TNF 抗体)-mediated EC dysfunction.
p38 MAPK (MAPK14 (显示 MAPK14 抗体)) is redox-regulated in reactive oxygen species-dependent endothelial barrier dysfunction.
P38 (显示 CRK 抗体) kinase role in the inflammatory pain.CXCL13, upregulated by peripheral inflammation, acts on CXCR5 (显示 CXCR5 抗体) on dorsal root ganglia neurons and activates p38 (显示 CRK 抗体), which increases Nav1.8 (显示 SCN10A 抗体) current density and further contributes to the maintenance of inflammatory pain.
these findings indicate that BMS309403 reduces fatty acid-induced ER stress-associated inflammation in skeletal muscle by reducing p38 MAPK (显示 MAPK14 抗体) activation.
report insulin-like growth factor-II binding protein 1 (IGF2BP1 (显示 B4GALNT2 抗体)) as a novel interacting partner of p38 MAPK (显示 MAPK14 抗体).
These results were supported by the opposite outcomes observed for cells treated with A779 or DX600. Therefore, it was concluded that the ACE2 (显示 ACE2 抗体)-Ang (显示 ANG 抗体)(17)-Mas (显示 MAS1 抗体) axis significantly inhibits pancreatitis by inhibition of the p38 MAPK (显示 MAPK14 抗体)/NF-kappaB (显示 NFKB1 抗体) signaling pathway
results suggest that c-Jun (显示 JUN 抗体), p38 MAPK (显示 MAPK14 抗体), PIK3CA (显示 PIK3CA 抗体)/Akt (显示 AKT1 抗体), and GSK3 signaling involved in the effect of miR (显示 MLXIP 抗体)-203 on the proliferation of hepatocellular carcinoma cells.
The Macrophage Activation Induced by Bacillus thuringiensis Cry1Ac Protoxin Involves ERK1/2 and p38 (显示 CRK 抗体) Pathways and the Interaction with Cell-Surface-HSP70 (显示 HSP70 抗体)
MAPK (显示 MAPK1 抗体) in, and found that p38alpha (显示 MAPK14 抗体) deficiency causes Th1 (显示 HAND1 抗体) cells to hyperproliferate via the Mnk1 (显示 MKNK1 抗体)/eIF4E (显示 EIF4E 抗体) pathway
B7-H1 (显示 CD274 抗体) suppresses p38 MAPK (显示 MAPK14 抗体) activation by sequestering DNA-PKcs (显示 PRKDC 抗体) in order to preserve T cell survival
increased in lentivirus vector thioredoxin interacting protein (显示 TXNIP 抗体) (LV-GFP-TXNIP (显示 TXNIP 抗体)) cells.
Our data demonstrated that p38 MAPK (显示 MAPK14 抗体) may be a potential therapeutic target for hypertension-related cognitive dysfunction.
P38 (显示 MAPK14 抗体) and JNK (显示 MAPK8 抗体) have opposing effects on persistence of in vivo leukocyte migration in zebrafish.
Adult zebrafish cardiomyocytes express active p38alpha (显示 MAPK14 抗体) MAPK (显示 MAPK1 抗体), which is switched off upon entry into mitosis.
Dkk3r regulates p38a (显示 MAPK14 抗体) phosphorylation to maintain Smad4 (显示 SMAD4 抗体) stability, in turn enabling the Smad2 (显示 SMAD2 抗体).Smad3a.Smad4 complex to form and activate the myf5 (显示 MYF5 抗体) promoter.
The protein encoded by this gene is a member of the MAP kinase family. MAP kinases act as an integration point for multiple biochemical signals, and are involved in a wide variety of cellular processes such as proliferation, differentiation, transcription regulation and development. This kinase is activated by various environmental stresses and proinflammatory cytokines. The activation requires its phosphorylation by MAP kinase kinases (MKKs), or its autophosphorylation triggered by the interaction of MAP3K7IP1/TAB1 protein with this kinase. The substrates of this kinase include transcription regulator ATF2, MEF2C, and MAX, cell cycle regulator CDC25B, and tumor suppressor p53, which suggest the roles of this kinase in stress related transcription and cell cycle regulation, as well as in genotoxic stress response. Four alternatively spliced transcript variants of this gene encoding distinct isoforms have been reported.
Csaids binding protein
, MAP kinase 14
, MAP kinase 2
, MAP kinase Mxi2
, MAP kinase p38 alpha
, MAPK 14
, MAX-interacting protein 2
, cytokine suppressive anti-inflammatory drug binding protein
, cytokine-supressive anti-inflammatory drug binding protein
, mitogen-activated protein kinase 14
, mitogen-activated protein kinase 14A
, mitogen-activated protein kinase p38 alpha
, p38 MAP kinase
, p38 mitogen activated protein kinase
, p38alpha Exip
, reactive kinase
, stress-activated protein kinase 2A
, cytokine suppressive anti-inflammatory drug binding protein 1
, mitogen activated protein kinase 14
, p38 MAP kinase alpha
, p38 MAPK
, p38 alpha
, tRNA synthetase cofactor p38
, MAP kinase 14A
, MAP kinase p38a
, MAPK 14A
, Mitogen-activated protein kinase p38a
, mitogen-activated protein kinase p38a
, CRK proto-oncogene, adaptor protein
, Proto-oncogene c-Crk
, adapter molecule crk
, proto-oncogene C-crk
, v-crk avian sarcoma virus CT10 oncogene homolog
, v-crk sarcoma virus CT10 oncogene homolog