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抗Human G3BP2 抗体:
抗Mouse (Murine) G3BP2 抗体:
抗Rat (Rattus) G3BP2 抗体:
Human Polyclonal G3BP2 Primary Antibody for ICC, IF - ABIN4313043
Wei, Fattet, Tsai, Guo, Pai, Majeski, Chen, Sah, Taylor, Engler, Yang: Matrix stiffness drives epithelial-mesenchymal transition and tumour metastasis through a TWIST1-G3BP2 mechanotransduction pathway. in Nature cell biology 2015
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Human Polyclonal G3BP2 Primary Antibody for ICC, IF - ABIN4313044
Katz, Sims, Sproul, Caldwell, Dixon, Meehan, Harrison: Targeting of Rac GTPases blocks the spread of intact human breast cancer. in Oncotarget 2012
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High G3BP2 levels worsened prognosis in breast cancer. Cell lines with lower G3BP2 levels required substantially higher cell numbers to form tumors in NOD/SCID (显示 PRKDC 抗体) mice. G3BP2 regulates SART3 (显示 SART3 抗体), Nanog (显示 NANOG 抗体), and Oct-4 (显示 POU5F1 抗体) expression levels and may be responsible for the maintenance of subpopulations of breast cancer cells with long-term proliferative properties.G3BP2 is important in breast tumor initiation.
Translocation of p53 (显示 TP53 抗体) is regulated by androgen-dependent sumoylation mediated by the G3BP2-interacting SUMO-E3 ligase (显示 PIAS1 抗体), RanBP2 (显示 RANBP2 抗体). G3BP2 knockdown results in reduced tumor growth and increased nuclear p53 (显示 TP53 抗体) accumulation in mouse xenograft models of prostate cancer with or without long-term androgen deprivation.
G3BP (显示 G3BP1 抗体) mediates the condensation of stress granules by shifting between two different states that are controlled by the phosphorylation of S149 and by binding to Caprin1 or USP10 (显示 USP10 抗体).
FGDF peptide binds and changes conformation of the protruding N-terminal residues by providing hydrophobic interactions to a symmetry related molecule that facilitated crystallization of the G3BP2 isoform.
G3BP1, G3BP2 and CAPRIN1 are required for translation of interferon stimulated mRNAs and are targeted by a dengue virus non-coding RNA.
Reveal a TWIST1 (显示 TWIST1 抗体)-G3BP2 mechanotransduction pathway that responds to biomechanical signals from the tumour microenvironment to drive EMT (显示 ITK 抗体), invasion and metastasis.
Stress granule components G3BP1 (显示 G3BP1 抗体) and G3BP2 play a proviral role early in Chikungunya virus replication.
both G3BP1 (显示 G3BP1 抗体) and G3BP2 play a role in the formation of SGs (显示 FBN1 抗体) in various human cells and thereby recovery from these cellular stresses.
Data show that the nsP3 (显示 SH2D3C 抗体)/G3BP (显示 G3BP1 抗体) interaction also blocks stress granules (SGs (显示 FBN1 抗体)) induced by other stresses than virus infection.
first evidence of direct interactions between PKCalpha (显示 PKCa 抗体) and G3BP2 and that PKCalpha (显示 PKCa 抗体) can phosphorylate G3BP2.
Wnt3a (显示 WNT3A 抗体)-stimulated LRP6 (显示 LRP6 抗体) phosphorylation is dependent upon arginine methylation of G3BP2.
Probable scaffold protein that may be involved in mRNA transport (Potential).
ras GTPase-activating protein-binding protein 2
, Ras-GTPase activating protein SH3 domain-binding protein 2
, Ras GTPase-activating protein-binding protein 2
, GTPase activating protein (SH3 domain) binding protein 2
, GAP SH3 domain-binding protein 2
, Ras-GTPase-activating protein (GAP120) SH3-domain binding protein 2
, Ras-GTPase-activating protein (GAP<120>) SH3-domain binding protein 2