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Mouse (Murine) CSF2 ELISA Kit for Sandwich ELISA - ABIN415372
Cao, Xu, Jin, Zhang, Lu, Nie, Tong: Effects of 900-MHz microwave radiation on gamma-ray-induced damage to mouse hematopoietic system. in Journal of toxicology and environmental health. Part A 2010
Human CSF2 ELISA Kit for Sandwich ELISA - ABIN414381
Pollard, Rockstroh, Pantaleo, Asmuth, Peters, Lazzarin, Garcia, Ellefsen, Podzamczer, van Lunzen, Arastéh, Schürmann, Clotet, Hardy, Mitsuyasu, Moyle, Plettenberg, Fisher, Fätkenheuer, Fischl, Taiwo et al.: Safety and efficacy of the peptide-based therapeutic vaccine for HIV-1, Vacc-4x: a phase 2 randomised, double-blind, placebo-controlled trial. ... in The Lancet. Infectious diseases 2014
TRAF6 (显示 TRAF6 ELISA试剂盒) is also required for GM-CSF-induced ubiquitination and activation of Akt (显示 AKT1 ELISA试剂盒).
Upregulation of GM-CSF and M-CSF (显示 CSF1 ELISA试剂盒) production by endothelial cells, an effect that appears to be mediated by NF-kappaB (显示 NFKB1 ELISA试剂盒) and to be independent of IL-1 (显示 IL1A ELISA试剂盒), may be an additional mechanism through which IL-33 (显示 IL33 ELISA试剂盒) contributes to inflammatory activation of the vessel wall.
Oral and periodontal innate immunity is affected by HIV viremia and ART. GCF (显示 GUCY2F ELISA试剂盒) IL-8 (显示 IL8 ELISA试剂盒), G-CSF (显示 CSF3 ELISA试剂盒), as well as serum IL-8 (显示 IL8 ELISA试剂盒), MCP-1 (显示 CCL2 ELISA试剂盒) and GM-CSF may be useful biomarkers for the detection of disease presence and/or its severity due to HIV infection and ART use.
Data suggest that the intratumoral GM-CSF expression, as a potentially independent prognostic biomarker for recurrence, might improve conventional clinical and pathologic analysis to refine outcome prediction for clinically localized clear-cell renal cell carcinoma (ccRCC) patients after surgery.
High GM-CSF expression is associated with breast Cancer.
In gastric cancer (GC), tumour-derived GM-CSF activated neutrophils and induced neutrophil PD-L1 (显示 CD274 ELISA试剂盒) expression via Janus kinase (JAK (显示 JAK3 ELISA试剂盒))-signal transducer and activator of transcription 3 (STAT3 (显示 STAT3 ELISA试剂盒)) signalling pathway. The activated PD-L1 (显示 CD274 ELISA试剂盒)(+) neutrophils effectively suppressed normal T-cell immunity in vitro and contributed to the growth and progression of human GC in vivo.
The IL-3 (显示 IL-3 ELISA试剂盒)/ GM-CSF effected on the myofibroblastic differentiation of human adipose derive stromal cells (hASCs) as well as it did on human dermal fibroblasts (HDFs).
Obesity alters the lung neutrophil infiltration to enhance breast cancer metastasis through IL5 (显示 IL5 ELISA试剂盒) and GM-CSF.
Data suggest that the methylotrophic yeast Pichia pastoris is an effective recombinant host for heterologous granulocyte-macrophage colony-stimulating factor (rhGM-CSF) production.
Letter: Knockdown of either filaggrin (显示 FLG ELISA试剂盒) or loricrin (显示 LOR ELISA试剂盒) increases the productions of interleukin (IL)-1alpha, IL-8 (显示 IL8 ELISA试剂盒), IL-18 (显示 IL18 ELISA试剂盒) and granulocyte macrophage colony-stimulating factor in stratified human keratinocytes.
Impaired RASGRF1 (显示 RASGRF1 ELISA试剂盒)/ERK (显示 EPHB2 ELISA试剂盒)-mediated GM-CSF response characterizes CARD9 (显示 CARD9 ELISA试剂盒) deficiency in French-Canadians.
These results demonstrated that bovine colonic cells seem capable to respond to E. bovis merozoite I infection by the upregulation of CXCL10 (显示 CXCL10 ELISA试剂盒) and GM-CSF gene transcription.
Data suggest exposure to maternal CSF2 from D5-D7 of development is fundamentally different for female/male blastocysts with respect to embryo elongation, characteristics of transcriptome/methylome, and endometrial interferon tau secretion at D15 (显示 MRPL16 ELISA试剂盒).
The increase in calving rate caused by CSF2 treatment involves, in part, more extensive development of extraembryonic membranes and capacity of the conceptus to secrete IFNT2 at day 15 of pregnancy.
immunolocalization studies confirmed the presence of granulocyte-macrophage colony stimulating factor(GM-CSF) in the germ cell line in bovine and human testes and addition of GM-CSF enhances several parameters of sperm motility
the conceptus, through its secretion of IFN-tau, stimulates maternal epithelial expression of COX-2 (显示 PTGS2 ELISA试剂盒) and GM-CSF during the peri (显示 PLIN1 ELISA试剂盒)-attachment period in the cow.
CSF2 affect embryonic development and enhance embryo competence for posttransfer survival.
These impaired macrophage functions in leukemic mice were significantly corrected by IL-3 (显示 IL-3 ELISA试剂盒) and GM-CSF treatment indicating the therapeutic benefit of these two cytokines in leukemia.
Both IL-6 (显示 IL6 ELISA试剂盒) protein production and transcript levels were downregulated by RA in respiratory tract epithelial cells (LETs) , but upregulated in macrophages (MACs). RA also increased transcript levels of MCP-1 (显示 CPT1B ELISA试剂盒), GMCSF, and IL-10 (显示 IL10 ELISA试剂盒) in MACs, but not in LETs. Conversely, when LETs, but not MACs, were exposed to RA
T-GM-CSF and -IL-3 (显示 IL-3 ELISA试剂盒) significantly, and reciprocally, blunted receptor binding and myeloid progenitor cell proliferation activity of both FL-GM-CSF and -IL-3 (显示 IL-3 ELISA试剂盒) in vitro and in vivo
Results indicate GM-CSF as both a key contributor to the pathogenesis of MI and a potential therapeutic target.
GM-CSF is required for the normal balance of leukocyte subsets, including granulocytes, B cells, and naive vs. effector T cells. There was an approximately 3-fold increase in the percentages of granulocytes in Csf2-/- PBMCs. The presence of maximal experimental autoimmune encephalomyelitis in the complete absence of GM-CSF revealed that GM-CSF is not an obligate effector molecule in all forms of EAE.
chemerin (显示 RARRES2 ELISA试剂盒) inhibited nuclear factor-kappaB activation and the expression of granulocyte-macrophage colony-stimulating factor (GM-CSF) and interleukin-2 (显示 IL2 ELISA试剂盒) (IL-6 (显示 IL6 ELISA试剂盒)) by tumor cells and tumor-associated endothelial cell, respectively, via its receptors, and consequently, MDSC induction was impaired, leading to restoration of antitumor T-cell response and decreased tumor angiogenesis.
These findings describe a novel role for GM-CSF as an essential initiating cytokine in cardiac inflammation.
Data reviewed establish that any damage to brain tissue tends to cause an increase in G-CSF (显示 CSF3 ELISA试剂盒) and/or GM-CSF (G(M)-CSF (显示 CSF1R ELISA试剂盒)) synthesized by the brain. Glioblastoma cells themselves also synthesize G(M)-CSF (显示 CSF1R ELISA试剂盒). G(M)-CSF (显示 CSF1R ELISA试剂盒) synthesized by brain due to damage by a growing tumor and by the tumor itself stimulates bone marrow to shift hematopoiesis toward granulocytic lineages away from lymphocytic lineages.
Evi1 (显示 MECOM ELISA试剂盒)(+)DA-3 cells modified to express an intracellular form of GM-CSF, acquired growth factor independence and transplantability and caused an overt leukemia in syngeneic hosts, without increasing serum GM-CSF levels.
CSF2 stimulates proliferation of trophectoderm cells by activation of the PI3K-and ERK1/2 (显示 MAPK1/3 ELISA试剂盒) MAPK (显示 MAPK1 ELISA试剂盒)-dependent MTOR (显示 FRAP1 ELISA试剂盒) signal transduction cascades.
Data indicate that differentially expressed IL-17 (显示 IL17A ELISA试剂盒), IL-22 (显示 IL22 ELISA试剂盒), and IL-23 (显示 IL23A ELISA试剂盒) levels are associated with K-ras (显示 HRAS ELISA试剂盒) in a stage-specific fashion along colorectal cancer progression and an association was established between mutant K-ras (显示 HRAS ELISA试剂盒) and GM-CSF and IFN-gamma (显示 IFNG ELISA试剂盒).
The protein encoded by this gene is a cytokine that controls the production, differentiation, and function of granulocytes and macrophages. The active form of the protein is found extracellularly as a homodimer. This gene has been localized to a cluster of related genes at chromosome region 5q31, which is known to be associated with interstitial deletions in the 5q- syndrome and acute myelogenous leukemia. Other genes in the cluster include those encoding interleukins 4, 5, and 13.
, granulocyte-macrophage colony-stimulating factor
, colony stimulating factor 2 (granulocyte-macrophage)
, colony-stimulating factor
, granulocyte-macrophage colony stimulating factor 2
, put. GM-CSF
, granulate-macrophage stimulating factor
, granulocyte-macrophage stimulation factor