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抗Human Angiotensin II 抗体:
抗Rat (Rattus) Angiotensin II 抗体:
抗Mouse (Murine) Angiotensin II 抗体:
Human Polyclonal Angiotensin II Primary Antibody for IF (p), IHC (p) - ABIN670521
Anand, Yiangou, Sinisi, Fox, MacQuillan, Quick, Korchev, Bountra, McCarthy, Anand: Mechanisms underlying clinical efficacy of Angiotensin II type 2 receptor (AT2R) antagonist EMA401 in neuropathic pain: clinical tissue and in vitro studies. in Molecular pain 2015
Show all 2 Pubmed References
The ACE (显示 ACE 抗体) and AGT (显示 AGXT 抗体) gene polymorphisms are not associated with the progress of diabetes developing into retinopathy in Chinese patients with type 2 diabetes.
AGT (显示 AGXT 抗体) M235T and T174M variants contribute to an increased risk of developing preeclampsia (PE), and for M235T to PE severity.
Data, including data using network analysis, suggest that angiotensinogen (AGT (显示 AGT 抗体)), mitogen-activated protein kinase-14 (MAPK14 (显示 MAPK14 抗体)), and prothrombin (显示 F2 抗体) (F2) in placental villous tissues are core factors in early embryonic development; these studies involved proteomics and bioinformatics analysis of altered protein expression in placental villous tissue from early recurrent miscarriage patients in comparison to control tissues.
The AGT (显示 AGXT 抗体) (M235T) gene polymorphism do not seem to have a significant effect on the development of clinical properties or cardiovascular comordities of acromegalic patients.
Angiotensin II has a role in increasing glomerular permeability by beta-arrestin mediated nephrin (显示 NPHS1 抗体) endocytosis
After donor nephrectomy, increasing uAGT (显示 DPAGT1 抗体) levels can be the result of activation of the intrarenal renin (显示 REN 抗体)-angiotensin system affecting the compensatory changes in the remaining kidney.
M235T polymorphism of the AGT (显示 AGXT 抗体) gene seems unrelated to the development or the clinical course of endometriosis.
AGT (显示 AGXT 抗体) missense polymorphisms are not associated with diabetic nephropathy in our subset of Slovenian type 2 diabetes mellitus patients
Association of AGT (显示 AGXT 抗体) single nucleotide polymorphism rs3789678 and gestational hypertension in Chinese population.
The T allele of AGT (显示 AGXT 抗体) may play a role in the pathogenesis of preeclampsia in South African Black women.
ANG II is up-regulated in serum and heart tissues of mice with EAM and that ANG II significantly drives monocyte/macrophage infiltration through the C-C chemokine receptor 2/5 (CCR2/5) axis.
results established that A20 (显示 TNFAIP3 抗体) is involved in the renoprotective effect by calcitriol via negatively modulating the NF-kappaB (显示 NFKB1 抗体) pathway and necroptotic pathway in AngII-induced renal injury.
NLRP3 (显示 NLRP3 抗体) gene deletion attenuates Ang II (显示 AGT 抗体)-induced NLRP3 (显示 NLRP3 抗体) inflammasome activation, phenotypic transformation from a contractile phenotype to a synthetic phenotype and proliferation in primary mice Vascular Smooth Muscle Cells.
adipocyte-derived Agt (显示 AGXT 抗体) has essentially no contribution to the plasma concentration and no impact on blood pressure compared to liver-derived Agt (显示 AGXT 抗体).
Lung ischemia-reperfusion injury causes a dysregulation of circulating Ang 2 (显示 ANGPT2 抗体) levels and plasma PREP (显示 PREP 抗体) activity, although no direct link between both phenomena could be shown.
Inhibition of TLR4 (显示 TLR4 抗体) ameliorates AngII-impaired cavernosal relaxation, decreases TNF-alpha (显示 TNF 抗体) levels, and restores Nitric Oxide bioavailability, demonstrating that TLR4 (显示 TLR4 抗体) partly mediates AngII-induced cavernosal dysfunction.
Our study is the first to show the important role of IL-6 (显示 IL6 抗体) in regulating cardiac pathogenesis via inflammation and apoptosis during AngII-induced hypertension. We also provide a novel link between IL-6 (显示 IL6 抗体)/STAT3 (显示 STAT3 抗体) and EndoG (显示 ENDOG 抗体)/MEF2A (显示 MEF2A 抗体) pathway that affects cardiac hypertrophy during AngII stimulation.
this study demonstrated that Ang II (显示 AGT 抗体) could increase TRPC6 (显示 TRPC6 抗体) induced Ca(2 (显示 CA2 抗体)+) influx and enhance autophagy through increasing reactive oxygen species levels in podocytes, and autophagy could protect Ang II (显示 AGT 抗体)-treated podocytes.
These results implied that AngII could effectively induce EpiCs to differentiate into vascular smooth muscle-like cells through the AT1 receptor (显示 AGTRAP 抗体).
Results suggest the involvement of angiotensin II (Ang II), through its angiotensin type-1 receptor (AT1R (显示 AGTRAP 抗体)) in the inflammation induced by Aah (显示 ASPH 抗体) venom, in the heart and the aorta.
The protein encoded by this gene, pre-angiotensinogen or angiotensinogen precursor, is expressed in the liver and is cleaved by the enzyme renin in response to lowered blood pressure. The resulting product, angiotensin I, is then cleaved by angiotensin converting enzyme (ACE) to generate the physiologically active enzyme angiotensin II. The protein is involved in maintaining blood pressure and in the pathogenesis of essential hypertension and preeclampsia. Mutations in this gene are associated with susceptibility to essential hypertension, and can cause renal tubular dysgenesis, a severe disorder of renal tubular development. Defects in this gene have also been associated with non-familial structural atrial fibrillation, and inflammatory bowel disease.
alpha-1 antiproteinase, antitrypsin
, angiotensin I
, angiotensin II
, serine (or cysteine) proteinase inhibitor
, serpin A8
, angiotensinogen (PAT)
, angiotensin ll