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抗Mouse (Murine) AGTR1a 抗体:
抗Rat (Rattus) AGTR1a 抗体:
抗Human AGTR1a 抗体:
Human Polyclonal AGTR1a Primary Antibody for IF (p), IHC (p) - ABIN677873
Liu, Liu, Tanabe, Maeda, Zou, Komano: Differential effects of angiotensin II receptor blockers on A? generation. in Neuroscience letters 2014
Nrf2 (显示 NFE2L2 抗体)-mediated stimulation of intrarenal Renin (显示 REN 抗体)-angiotensin system gene expression, by which chronic hyperglycemia induces hypertension and renal injury in diabetes.
This study aimed to define whether sex chromosome complement (SCC (显示 CYP11A1 抗体)) may differentially modulate sex differences in relative gene expression of basal Agtr1a (显示 AGTR1 抗体), Agtr2 (显示 AGTR2 抗体), and Mas1 (显示 MAS1 抗体) receptors at fore/hindbrain nuclei and at medulla/cortical kidney.
The formation of liver metastasis, in a mouse model of colorectal cancer, correlated with collagen deposition in the metastatic area, which was dependent on AT1a signaling.
Perivascular Adipose Tissue Angiotensin II Type 1a Receptor Promotes Vascular Inflammation and Aneurysm formation in apolipoprotein E (显示 APOE 抗体)-deficient (ApoE (显示 APOE 抗体)(-/-)) mice.
The findings suggested that ox-LDL could induce cardiac hypertrophy through the direct association of AT1-R (显示 AGTR1 抗体) and LOX-1 (显示 OLR1 抗体).
Mice lacking the AT1A receptor specifically in LEPR (显示 LEPR 抗体)-expressing cells failed to show an increase in resting metabolic rate in response to a high-fat diet and deoxycorticosterone acetate-salt (DOCA-salt) treatments, but blood pressure control remained intact.
In wild-type, total (tNCC (显示 TNNC1 抗体)) and phosphorylated (pNCC) NCC (显示 SLC12A3 抗体) protein expressions were 1.8- and 4.6-fold higher in females compared with males, consistent with the larger response to HCTZ. In AT1a receptor knockout mice, tNCC (显示 TNNC1 抗体) and pNCC increased significantly in males to levels not different from those in females.
This study showed that activation of the AT1a receptor may contribute to maintenance of the glomerular structure against hypertensive renal damage.
Altered expression of AT1 and AT2 receptors with aging may induce mitochondrial dysfunction, the main risk factor for neurodegeneration.
Results provide evidence that blockade of the AT1a receptor could have some effects on browning of WAT, with inhibitory effects on adipose tissue-derived stem cells differentiation into adipocytes.
These results identify bTREK-1 K(+) channels as a pivotal control point where ANG II (显示 AGT 抗体) receptor activation is transduced to depolarization-dependent Ca(2 (显示 CA2 抗体)+) entry and aldosterone secretion.
Angiotensin II (ANGII) inhibits adrenocortical cell KCNK2 in an ATP dependent, PLC/PKC independent manner.
Luminal ANG II is internalized as a complex with AT1R/AT2R heterodimers to target endoplasmic reticulum in LLC-PK1 cells, where it might trigger intracellular calcium responses.
AT1 (显示 AGTR1 抗体) and AT2 (显示 AGTR2 抗体) receptors heterodimerize and are involved in the angiotensin II effect on SERCA (显示 ATP2A3 抗体) in proximal kidney tubules.
A critical role for lipid raft microdomains in AGTR1 (显示 AGTR1 抗体)-mediated signal transduction in neonatal glomerular mesengial cells.
AT1 (显示 AGTR1 抗体) receptors are positively coupled to the proliferative response of vascular smooth muscle cells to angiotensin II.
receptor for and mediator of vascular remodeling effects of angiotensin II
angiotensin II type-1A receptor
, angiotensin receptor 1
, angiotensin receptor 1a
, type-1A angiotensin II receptor
, vascular type-1 angiotensin II receptor
, angiotensin II receptor 1
, angiotensin II type-1 receptor
, type-1 angiotensin II receptor
, angiotensin II type 1 receptor
, Angiotensin II type-1 receptor
, Type-1 angiotensin II receptor