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Protects actin filaments from depolymerization (By similarity). 再加上，我们可以发XIRP1 试剂盒 (2)和数多这个蛋白质的别的产品。
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the porcine CMYA1 gene has effects on porcine back fat deposition
Xin deficiency leads to a myopathic condition characterized by increased muscle fatigability
Aciculin (显示 PGM5 抗体) interacts with filamin C (显示 FLNC 抗体) and Xin and is essential for myofibril assembly.
The results suggest that mXinalpha play a role in myotendinous (显示 TNC 抗体) junction conductance of contractile and stretching forces.
mXinalpha controls the dynamics of cortical actin cytoskeleton.
Data show that mXinbeta (CMYA3/XIRP2 (显示 XIRP2 抗体)) but not mXinalpha ((CMYA1/XIRP1) was uniquely up-regulated during the redistribution of intercellular junction from the lateral membrane of cardiomyocytes to their termini.
Total Xin deficiency leads to topographical intercalated disc alterations, premature fibrosis and subtle changes in contractile behaviour; this is a milder cardiac phenotype than that observed in XinAB(-/-) mice, which still can express XinC
Xin has a role in the formation of cell-cell contacts and possibly in myofibrillogenesis
Xin is expressed within muscle satellite cells during skeletal muscle regeneration and is involved in the regulation of myoblast function.
The binding of mXin alpha to beta-catenin at the adherens junction would then facilitate actin binding.
The strong correlation between the degree of muscle damage and Xin immunoreactivity suggests that Xin may be a suitable outcome measure to evaluate disease progression and treatment effects in clinical trials.
We identify the SH3 domains of nebulin (显示 NEB 抗体) and nebulette (显示 NEBL 抗体) as novel ligands of proline-rich regions of Xin and XIRP2 (显示 XIRP2 抗体).
Data provide further support that ALS2CL (显示 ALS2CL 抗体), EPHA3 (显示 EPHA3 抗体), and CMYA1 are bona-fide tumor-suppressor genes and contribute to the tumorigenesis of HNSCC.
Xin and XIRP2 define a novel actin-binding motif
Here, we identify Xin, the protein encoded by the human gene 'cardiomyopathy associated 1'(CMYA1) as filamin c (显示 FLNC 抗体) binding partner at these specialized structures where the ends of myofibrils are attached to the sarcolemma
Data show that in myofibrillar myopathies Xin protein exhibites significant alterations in their localization.
The results thus suggest that the CMYA1 promoter could be an effective muscle-specific (显示 EIF3K 抗体) promoter, which may be useful in further studies of cardiomyopathy treatment and transgenic animal research
Protects actin filaments from depolymerization (By similarity).
, cardiomyopathy associated 3
, xin actin-binding repeat containing 2
, xin actin-binding repeat-containing protein 1
, xin actin-binding repeat containing 1
, cardiomyopathy associated 1
, cardiomyopathy-associated 1
, xin actin-binding repeat-containing protein 1-like
, Xin alpha
, cardiomyopathy-associated protein 1
, mXin alpha
, Xin alpha 2
, Xin repeat-containing protein
, cardiomyopathy associated protein 1