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VAMP8 encodes an integral membrane protein that belongs to the synaptobrevin/vesicle-associated membrane protein subfamily of soluble N-ethylmaleimide-sensitive factor attachment protein receptors (SNAREs). 再加上，我们可以发VAMP8 抗体 (42) 和 VAMP8 蛋白 (8)和数多这个蛋白质的别的产品。
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our study suggested that VAMP8 gene variants might not contribute to glioma susceptibility and associated with glioma in the Chinese Han population.
Vesicle-associated membrane protein 8 as a novel oncogene (显示 RAB1A ELISA试剂盒) by promoting cell proliferation and therapeutic resistance in glioma. Targeting VAMP8 may serve as a potential therapeutic regimen for the treatment of glioma.
Starvation-induced MTMR13 (显示 SBF2 ELISA试剂盒) and RAB21 (显示 RAB21 ELISA试剂盒) activity regulates VAMP8 to promote autophagosome-lysosome fusion.
Cytotoxic granule exocytosis is a sequential, multivesicle fusion process requiring VAMP8-mediated recycling endosome fusion before cytotoxic granule fusion.
Inhibition of VAMP8, but not VAMP7 (显示 VAMP7 ELISA试剂盒), significantly reduces viral entry.
VAMP8 regulates mucin (显示 SLC13A2 ELISA试剂盒) granule exocytosis in airway goblet cells, and reduction of its expression may provide a novel therapeutic target to ameliorate airway mucus obstruction in lung diseases
Assessment of KIF6 genotype is not useful in predicting low density lipoprotein cholesterol lowering response to pravastatin, and heart disease risk reduction in the elderly.
Dynamics of FIP3 (显示 IKBKG ELISA试剂盒)- and VAMP8-containing endosomes reflect the progressive stages of abscission.
The VAMP8 rs1010 polymorphism was associated with CHD risk in Chinese Han population, the A allele might serve as a genetic risk factor of coronary heart disease.
Results suggest that the combinational SNARE proteins VAMP8 and Vti1b mediate the fusion of antimicrobial and canonical autophagosomes with lysosomes, an essential event for autophagic degradation.
acute inhibition of VAMP8-mediated secretion during pancreatitis triggers intracellular trypsin accumulation and loss of the early endosomal compartment
These findings reveal a role for VAMP8 in LC3 (显示 MAP1LC3A ELISA试剂盒)-associated phagocytosis and highlight a novel mechanism exploited by L. major promastigotes to interfere with the host antimicrobial machinery
VAMP8-mediated secretion is dependent on anterograde endosomal trafficking.
Leishmania evades host immunity by GP63 (显示 LMLN ELISA试剂盒)-mediated VAMP8 cleavage.
VAMP8 mediates insulin (显示 INS ELISA试剂盒) granule recruitment to the plasma membrane, which partly accounts for GLP-1 (显示 GCG ELISA试剂盒) potentiation of glucose-stimulated insulin (显示 INS ELISA试剂盒) secretion.
VAMP8 knockout mice were used to show that the SNARE (显示 VTI1B ELISA试剂盒) machinery plays a critical role in the process of granule homotypic fusion.
that granule-to-granule fusion is regulated by VAMP8 containing SNARE (显示 VTI1B ELISA试剂盒) complexes distinct from those that regulate primary granule fusion.
data demonstrate that, in the absence of VAMP8, the relative subcellular distribution of GLUT4 (显示 SLC2A4 ELISA试剂盒) is altered, resulting in increased sarcolemma levels that can account for increased glucose clearance and insulin (显示 INS ELISA试剂盒) sensitivity
VAMP8 mediates the regulated fusion of AQP2 (显示 AQP2 ELISA试剂盒)-positive vesicles with the plasma membrane.
This gene encodes an integral membrane protein that belongs to the synaptobrevin/vesicle-associated membrane protein subfamily of soluble N-ethylmaleimide-sensitive factor attachment protein receptors (SNAREs). The encoded protein is involved in the fusion of synaptic vesicles with the presynaptic membrane.
vesicle-associated membrane protein 8
, vesicle-associated membrane protein 8 (endobrevin)
, Vesicle-associated membrane protein 8