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SELPLG encodes a glycoprotein that functions as a high affinity counter-receptor for the cell adhesion molecules P-, E- and L- selectin expressed on myeloid cells and stimulated T lymphocytes. 再加上，我们可以发SELPLG 试剂盒 (35) 和 SELPLG 蛋白 (18)和数多这个蛋白质的别的产品。
Showing 10 out of 260 products:
Mouse (Murine) Monoclonal SELPLG Primary Antibody for BR, FACS - ABIN1177261
Borges, Eytner, Moll, Steegmaier, Campbell, Ley, Mossmann, Vestweber: The P-selectin glycoprotein ligand-1 is important for recruitment of neutrophils into inflamed mouse peritoneum. in Blood 1997
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Human Monoclonal SELPLG Primary Antibody for CyTOF, ELISA - ABIN262947
Walcheck, Leppanen, Cummings, Knibbs, Stoolman, Alexander, Mattila, McEver: The monoclonal antibody CHO-131 binds to a core 2 O-glycan terminated with sialyl-Lewis x, which is a functional glycan ligand for P-selectin. in Blood 2002
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Human Monoclonal SELPLG Primary Antibody for Func, FACS - ABIN610433
Connolly, Chait, Duncan, Taylor: CT-guided percutaneous needle biopsy of small lung nodules in children. in Pediatric radiology 1999
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Human Monoclonal SELPLG Primary Antibody for BR, CyTOF - ABIN5012982
Santen, Schramm, Menger, Wang, Jeppsson, Thorlacius: P-selectin glycoprotein ligand-1 regulates chemokine-dependent leukocyte recruitment in colonic ischemia-reperfusion. in Inflammation research : official journal of the European Histamine Research Society ... [et al.] 2008
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Mouse (Murine) Polyclonal SELPLG Primary Antibody for ICC, IHC - ABIN1870024
Baba, Batanova, Kitoh, Takashima: Adhesion of Toxoplasma gondii tachyzoite-infected vehicle leukocytes to capillary endothelial cells triggers timely parasite egression. in Scientific reports 2017
Human Monoclonal SELPLG Primary Antibody for CyTOF, FACS - ABIN4348165
Wake, Mori, Liu, Morioka, Teshigawara, Sakaguchi, Kuroda, Gao, Takahashi, Ohtsuka, Yoshino, Morimatsu, Nishibori: Histidine-Rich Glycoprotein Prevents Septic Lethality through Regulation of Immunothrombosis and Inflammation. in EBioMedicine 2016
These data indicate that cardiac surgery influences the expression of CD162, CD166 (显示 ALCAM 抗体) and CD195 and that the intensity of the immune system response, displayed as the change in the CD162, CD166 (显示 ALCAM 抗体), CD195 expression, varies, depending on the surgical technique used.
PSGL1 mediated leukocyte rolling and the inflammatory response in general.PSGL1 is a ligand for Siglec-5 (显示 SIGLEC5 抗体) and interacts with Siglec-5 (显示 SIGLEC5 抗体) ectodomain.
The Interaction of Selectins and PSGL-1 as a Key Component in Thrombus Formation and Cancer Progression.
Studies indicate that P-selectin glycoprotein ligand-1 (PSGL-1) PSGL-1 can negatively regulate T cell function.
Results suggest that PSGL-1 may play an oncogenic role in the development of intestinal tumors, and this is likely mediated through activation of NFkB signaling by MIP (显示 TNPO1 抗体)-1g.
c-Myc (显示 MYC 抗体) regulates P-selectin glycoprotein ligand-1 expression in monocytes during HIV-1 infection.
The percentage of CXCR3 (显示 CXCR3 抗体)(+) CD4 (显示 CD4 抗体)(+) TEM (显示 CYLD 抗体) cells negatively correlated with the severity of the cutaneous disease in psoriasis patients. Importantly CLA(+) CD4 (显示 CD4 抗体)(+) TCM cells expressing CCR6 (显示 CCR6 抗体)(+) or CCR4 (显示 CCR4 抗体)(+)CXCR3 (显示 CXCR3 抗体)(+) negatively correlated with psoriasis severity suggesting recruitment to the skin compartment.
Platelet-leukocyte aggregations increased in acute ischemic stroke patients rapidly within 3h. The I allele of PSGL-1 M62I was associated with risk of developing acute ischemic stroke, especially large artery atherosclerosis stroke and small artery occlusion stroke. Small artery occlusion stroke patients with the II genotype of PSGL-1 M62I have the higher level of platelet-neutrophil aggregates.
The significant presence of CLA+ T cells and E-selectin (显示 SELE 抗体) expressions in the OLPG suggests their involvement in the etiopathogenesis of OLP; however, only a weak correlation between CLA+ T cells and E-selectin (显示 SELE 抗体) was observed.
PSGL-1 is a novel receptor for S. pneumoniae that contributes to protection against invasive pneumococcal disease.
results indicate that P-selectin (显示 SELP 抗体) deletion significantly decreases tumor stiffness in Rip1 (显示 RALBP1 抗体)-Tag2 mice by inhibiting LOX (显示 LOX 抗体) expression.
this study shows that PSGL-1 plays a fundamental role in balancing viral control and immunopathology and also functions to regulate T cell responses in the tumor microenvironment
Psgl-1 deficiency accelerates bleomycin-induced lung fibrosis and inflammation in mice through activating the PI3K/AKT (显示 AKT1 抗体) axis.
Circulating soluble P-selectin (显示 SELP 抗体) must dimerize to promote inflammation and thrombosis in mice.
The results from the present study suggest that activated platelets secrete Pselectin to promote cardiac inflammation and fibrosis in Ang (显示 ANG 抗体) IIinduced hypertension.
These findings indicate a novel pathway in tumor metastasis, i.e., tumor cell mediated activation of P-selectin (显示 SELP 抗体) in platelets, followed by activation and secretion of Asm (显示 SMPD1 抗体) and in turn release of ceramide and tumor metastasis. The data suggest that p38 MAPK (显示 MAPK14 抗体) acts downstream from P-selectin (显示 SELP 抗体) and is necessary for the secretion of Asm (显示 SMPD1 抗体).
Psgl-1 deficiency is protective against the prothrombotic effects of IL-1beta (显示 IL1B 抗体) .
PSGL-1, CD43, and CD44 moved normally to the uropods of chemokine-stimulated C1galt1(-/-) neutrophils
these results demonstrate that P-selectin (显示 SELP 抗体) expression on ECs is regulated in part by glycosylation mechanisms and that glycosylation inhibitors efficiently reduce the adhesion of sRBCs and leukocytes to ECs.
endothelial colony-forming cells interact with activated neutrophils via PSGL-1 and L-selectin (显示 SELL 抗体)
This gene encodes a glycoprotein that functions as a high affinity counter-receptor for the cell adhesion molecules P-, E- and L- selectin expressed on myeloid cells and stimulated T lymphocytes. As such, this protein plays a critical role in leukocyte trafficking during inflammation by tethering of leukocytes to activated platelets or endothelia expressing selectins. This protein requires two post-translational modifications, tyrosine sulfation and the addition of the sialyl Lewis x tetrasaccharide (sLex) to its O-linked glycans, for its high-affinity binding activity. Aberrant expression of this gene and polymorphisms in this gene are associated with defects in the innate and adaptive immune response. Alternate splicing results in multiple transcript variants.
P-selectin glycoprotein ligand 1
, cutaneous lymphocyte-associated associated antigen
, selectin P ligand
, P-selectin glycoprotein ligand 1 propeptide
, P-selectin glycoprotein ligand-1
, leukocyte cell surface adhesion molecule
, selectin, platelet (p-selectin) ligand