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Involved in transport from the endoplasmic reticulum to the Golgi apparatus. 再加上，我们可以发SAR1A 蛋白 (14) 和 和数多这个蛋白质的别的产品。
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Study shows that the bona fide nonglycoprotein Nox5 (显示 NOX5 抗体), a transmembrane superoxide-producing NADPH oxidase (显示 NOX1 抗体), is transported to the cell surface in a manner resistant to co-expression of Sar1 (显示 IQGAP1 抗体) (H79G), a GTP (显示 AK3 抗体)-fixed mutant of the small GTPase (显示 RACGAP1 抗体) Sar1 (显示 IQGAP1 抗体), which blocks COPII vesicle fission from the endoplasmic reticulum.
No associations were observed between any of the secretion associated Ras related GTPase (显示 RACGAP1 抗体) 1A (SAR1a) promoter variants and baseline fetal hemoglobin (显示 HBB 抗体) HbF among sickle cell disease (SCD (显示 SCD 抗体)) patients.
Sar1 (显示 IQGAP1 抗体) is a novel regulator of endoplasmic reticulum-mitochondrial contact sites in the metazoans.
SAR1A gene silencing in hepatocytes mimics the effect of ethanol: dedimerization of giantin (显示 GOLGB1 抗体), arresting PDIA3 (显示 PDIA3 抗体) in the endoplasmic reticulum (ER) and large-scale alterations in Golgi architecture.
Data indicate that hydroxyurea (HU) induces SAR1 (显示 IQGAP1 抗体) protein expression, which in turn activates gamma-globin (显示 HBG1 抗体) expression, predominantly through the Gialpha/JNK (显示 MAPK8 抗体)/Jun (显示 JUN 抗体) pathway.
although Sar1A antagonizes the lipoprotein secretion-promoting activity of Sar1B (显示 SAR1B 抗体), both isoforms modulate the expression of genes encoding cholesterol biosynthetic enzymes and the synthesis of cholesterol de novo.
Sar1 (显示 IQGAP1 抗体) mutant proteins added to metaphase-arrested Xenopus laevis egg extracts cause dramatic effects on membrane organization.
the behavior of the human of Sar1A and Sar1B (显示 SAR1B 抗体), a key component of the COPII family of vesicle coat proteins, was examined.
Sedlin (显示 TRAPPC2 抗体) bound and promoted efficient cycling of Sar1 (显示 IQGAP1 抗体), a guanosine triphosphatase that can constrict membranes, and thus allowed nascent carriers to grow and incorporate procollagen prefibrils.
lipid-directed and tether-assisted Sar1 (显示 IQGAP1 抗体) organization controls membrane constriction to regulate ER export.
Involved in transport from the endoplasmic reticulum to the Golgi apparatus. Required to maintain SEC16A localization at discrete locations on the ER membrane perhaps by preventing its dissociation. SAR1A-GTP-dependent assembly of SEC16A on the ER membrane forms an organized scaffold defining endoplasmic reticulum exit sites (ERES) (By similarity).
GTP-binding protein Sar1A
, GTP-binding protein SAR1a
, GTP-binding protein SAR1A
, SAR1a gene homolog 1
, COPII-associated small GTPase
, SAR1 gene homolog A