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Plxna2 encodes a member of the plexin-A family of semaphorin co-receptors. 再加上，我们可以发Plexin A2 试剂盒 (17) 和 Plexin A2 蛋白 (5)和数多这个蛋白质的别的产品。
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Human Polyclonal Plxna2 Primary Antibody for CyTOF, FACS - ABIN4900258
Plein, Calmont, Fantin, Denti, Anderson, Scambler, Ruhrberg: Neural crest-derived SEMA3C activates endothelial NRP1 for cardiac outflow tract septation. in The Journal of clinical investigation 2015
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Dog (Canine) Polyclonal Plxna2 Primary Antibody for WB - ABIN2783972
Budel, Shim, Feng, Zhao, Hisama, Strittmatter: No association between schizophrenia and polymorphisms of the PlexinA2 gene in Chinese Han Trios. in Schizophrenia research 2008
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used Sema6A (显示 SEMA6A 抗体)-deficient and PlxnA2-deficient zebrafish embryos in a microarray analysis to identify genes that were differentially expressed when this signaling pathway was disrupted.
Sema6a (显示 SEMA6A 抗体) and Plxna2 have roles in promoting eye vesicle cohesion through mediating spatially regulated repulsion within the developing zebrafish eye
Data suggest that Fyn (显示 FYN 抗体) tyrosine kinase (显示 TXK 抗体) (Fyn (显示 FYN 抗体))-dependent phosphorylation at two critical tyrosines is a key feature of vertebrate plexin A1 (PlxnA1 (显示 PLXNA1 抗体)) and plexin A2 (PlxnA2) signal transduction.
Data indicate that plexin A1 (显示 PLXNA1 抗体)-4 (PLXNA1 (显示 PLXNA1 抗体)-4) mediation of neuroanatomical traits can be detected using in vivo neuroimaging techniques.
although plexin-A4 (显示 PLXNA4 抗体) overexpression restored Sema3A (显示 SEMA3A 抗体) signaling in plexin-A1 (显示 PLXNA1 抗体)-silenced cells, it failed to restore Sema3B (显示 SEMA3B 抗体) signaling in plexin-A2-silenced cells.
PLXNA2 upregulation contributes to TMPRSS2 (显示 TMPRSS2 抗体):ERG (显示 ERG 抗体)-mediated enhancements of PC3c cell migration and invasion.
PLXNA2 has been identified as a new rare copy number variations gene for tetralogy of Fallot.
results of our study reveal that PlxnA2 has a pro-osteogenic function by modulating BMP2 (显示 BMP2 抗体) signaling
PLXNA2 polymorphisms show association with ankylosing spondylitis.
in vitro analysis of PLXNA2 revealed that the gene has higher expression in more aggressive breast cancer cell types.
An association is identified between variants in the PLXNA2 gene and schizophrenia in two collections of schizophrenia cases and controls. High-density SNP mapping showed that the region of association spans approximately 60 kb of the PLXNA2 gene.
results suggest that PLXNA2 may not play a major role in the development of schizophrenia in our Japanese sample
These findings implicate Semaphorin-6A (显示 SEMA6A 抗体) - Plexin-A2/Plexin-A4 (显示 PLXNA4 抗体) interactions in dorsal lateral geniculate nucleus axon guidance and in the spatiotemporal organization of guidepost cell populations in the mammalian subpallium.
describe procedures for protein production and purification that we utilized in the crystallographic study of the mouse Plexin A2 (mPlxnA2) extracellular fragment, including the sema (显示 SEMA3B 抗体) domain
Using time-lapse videomicroscopy we provide evidence that Sema6A (显示 SEMA6A 抗体)/PlexinA2 signaling participates to interkinetic nuclear migration of retinal progenitor cell
Plexin A2 is expressed in both On and Off starburst amacrine cell.
crystal structures of cognate complexes of the semaphorin-binding regions of plexins B1 and A2 with semaphorin ectodomains (human PLXNB1 (显示 PLXNB1 抗体)(1-2)-SEMA4D (显示 SEMA4D 抗体)(ecto (显示 TRIM33 抗体)) and murine PlxnA2(1-4)-Sema6A (显示 SEMA6A 抗体)(ecto (显示 TRIM33 抗体))), plus unliganded structures of PlxnA2(1-4) and Sema6A (显示 SEMA6A 抗体)(ecto (显示 TRIM33 抗体))
crystal structures of the semaphorin 6A (Sema6A (显示 SEMA6A 抗体)) receptor-binding fragment and the plexin A2 (PlxnA2) ligand-binding fragment in both their pre-signalling (that is, before binding) and signalling (after complex formation) states
This study demonstrated that Sema6B (显示 SEMA6B 抗体), another class 6 transmembrane semaphorin, is expressed in CA3 (显示 CA3 抗体) and repels mossy fibers in a PlxnA4 (显示 PLXNA4 抗体)-dependent manner in vitro.
In plexin-A2-deficient mice many granule cells remained in the molecular layer; mouse chimera studies further suggested that plexin-A2 acts in a cell-autonomous manner.
mutations in GATA6 (显示 GATA6 抗体) are genetic causes of congenital heart diseases involving outflow tract defects, as a result of the disruption of the direct regulation of semaphorin 3C (显示 SEMA3C 抗体)-plexin A2 signaling
This gene encodes a member of the plexin-A family of semaphorin co-receptors. Semaphorins are a large family of secreted or membrane-bound proteins that mediate repulsive effects on axon pathfinding during nervous system development. A subset of semaphorins are recognized by plexin-A/neuropilin transmembrane receptor complexes, triggering a cellular signal transduction cascade that leads to axon repulsion. This plexin-A family member is thought to transduce signals from semaphorin-3A and -3C.
, plexin A2
, plexin 2
, semaphorin receptor OCT
, transmembrane protein OCT
, plex 2