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ovarian BAD-interacting protein\; may inhibit apoptosis [RGD, Feb 2006].. 再加上，我们可以发MCL1 试剂盒 (1)和数多这个蛋白质的别的产品。
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The findings support a model in which survival is determined by quantitative participation of multiple anti-apoptotic proteins, BCL2 (显示 BCL2 抗体), Mcl1, and BCL2A1 (显示 BCL2A1 抗体), rather than by a single anti-apoptotic protein.
This dynamic change in B cell survival mechanisms is unique to Epstein-Barr virus-infected cells and relies on regulation of MCL-1 mitochondrial localization and BFL-1 (显示 BCL2A1 抗体) transcription by the viral EBNA3A (显示 EBNA-3A 抗体) protein.
Overexpression of Mcl-1 via the hemopoietic cell specific vavP-Mcl-1 transgene markedly exacerbates and accelerates the lpr (显示 FAS 抗体) phenotype. The progressive splenomegaly and lymphadenopathy displayed by lpr (显示 FAS 抗体) mice was far more severe in Mcl (显示 CLEC4D 抗体)-1tg/lpr (显示 FAS 抗体) littermates
Endothelial cell -specific deletion of Mcl1 resulted in a dose-dependent increase in endothelial cell apoptosis in the angiogenic vasculature and a corresponding decline in vessel density.
Our findings indicate that MCL-1 expression is an important biomarker of TEC (显示 NR4A3 抗体) survival
These results identify MCL-1 as a critical prosurvival protein for preventing beta-cell death and clarify the mechanisms behind its downregulation by proinflammatory cytokines.
These data show that Mcl-1 is dispensable for the regulation of apoptosis during infection with different large DNA viruses.Bcl-XL, on the other hand, can be important to maintain survival of virus-infected cells
BCL-XL (显示 BCL2L1 抗体) expression promotes survival of immature B cells, expression of BCL-2 (显示 BCL2 抗体) is important for survival of mature B cells and long-lived plasma cells (PC), and expression of MCL-1 is important for survival throughout B-cell development.
miR (显示 MLXIP 抗体)-32/MCL-1 pathway members were identified as key early genetic events driving melanoma progression.
GSK3B-MCL1 signaling to regulate axonal autophagy might be important for the successful completion of Wallerian degeneration.
ovarian BAD-interacting protein\; may inhibit apoptosis
bcl-2-related protein EAT/mcl1
, induced myeloid leukemia cell differentiation protein Mcl-1 homolog