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Bone morphogenetic proteins (e.g., BMP9\; MIM 605120) are members of the transforming growth factor-beta (see TGFB1\; MIM 190180) superfamily and regulate tissue differentiation and maintenance. 再加上，我们可以发GDF15 抗体 (310) 和 GDF15 蛋白 (37)和数多这个蛋白质的别的产品。
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Human GDF15 ELISA Kit for Sandwich ELISA - ABIN624984
Barderas, Mendes, Torres, Bartolomé, López-Lucendo, Villar-Vázquez, Peláez-García, Fuente, Bonilla, Casal: In-depth characterization of the secretome of colorectal cancer metastatic cells identifies key proteins in cell adhesion, migration, and invasion. in Molecular & cellular proteomics : MCP 2013
Show all 9 Pubmed References
Human GDF15 ELISA Kit for Sandwich ELISA - ABIN365728
Jiang, Yu, Wang, Tang, Guo, Jiao: Regulation of hepcidin through GDF-15 in cancer-related anemia. in Clinica chimica acta; international journal of clinical chemistry 2014
Human GDF15 ELISA Kit for Sandwich ELISA - ABIN415137
Sun, Huang, Phillips, Luo, Zhu, Shi, Li: Growth differentiation factor 15 and coronary collateral formation. in Clinical cardiology 2010
the association between GDF-15 and contrast-induced nephropathy in acute myocardial infarction patients after percutaneous coronary intervention
Age-dependent GDF-15 centile values developed from a population of apparently healthy older adults are independently predictive of all-cause mortality
To the best of our knowledge, this is the first documentation of cytokine GDF-15 presence in follicular fluid. Its concentration was not associated with donor/patient fertility status. Our data also show that GDF-15 is expressed and inducible in both normal healthy and cancerous granulosa cells, as well as in oocytes
Findings indicate that global platelet function was positively correlated with serum GDF-15 concentration.
GDF-15 in blood is more accurate in differentiating pancreatic mass due to chronic pancreatitis from pancreatic adenocarcinoma than Ca19-9. the combination of GDF-15 with Ca125 (显示 MUC16 ELISA试剂盒) is remarkably useful in differentiating pancreatic mass.
Our results show GDF-15 as an effective biomarker in patients with metastatic colorectal cancer
GDF-15 and TRAIL-R2 (显示 TNFRSF10B ELISA试剂盒) were the most powerful Proximity Extension Assay chip biomarkers in predicting long-term all-cause mortality in patients with acute myocardial infarction.
This study found that the mean serum GDF15 levels in the Parkinson Disease patients were significantly higher than those of the healthy control group. Secondly, in the Parkinson Disease patients, the GDF15 levels were significantly higher in the men than in the women.
Elevated GDF-15 correlates with lack of reverse remodeling and increased mortality after TAVR and improves risk prediction of mortality.
Initial serum GDF15 level showed an inverse correlation with serum glomerular filtration rate and was associated with worse renal outcome in immunoglobulin A nephropathy.
ur results demonstrate a novel and critical role of hepatic GDF15 activated by IRE1alpha (显示 ERN1 ELISA试剂盒)-XBP1s branch in regulating adaptive responses of liver upon starvation stress.
GDF15 is a myokine secreted by aging-related stress and may control aging phenotype
Membrane-protein extraction and use of an intracellular protein-transport inhibitor showed that GDF-15 promoted CaV3.1 and CaV3.3 alpha-subunit (显示 POLG ELISA试剂盒) expression by trafficking to the membrane.
CHOP (显示 DDIT3 ELISA试剂盒) activates Gdf15 transcription via binding to its promoter upon endoplasmic reticulum stress.
The ANT1 (显示 SLC25A4 ELISA试剂盒)-deficient muscle mitochondria produce excess reactive oxygen species (ROS (显示 ROS1 ELISA试剂盒)) and are partially uncoupled. Hence, the muscle respiration under nonphosphorylating conditions is increased. Muscle transcriptome analysis revealed the induction of mitochondrial biogenesis, down-regulation of diabetes-related genes, and increased expression of the genes encoding the myokines FGF21 (显示 FGF21 ELISA试剂盒) and GDF15.
isolating GFRAL as the receptor for GDF15-induced anorexia and weight loss, the identification of a mechanistic basis for the non-homeostatic regulation of neural circuitry by a peripheral signal associated with tissue damage and stress; findings provide opportunities to develop therapeutic agents for the treatment of disorders with altered energy demand
These data suggest that GDF15 is required for IL-13 (显示 IL13 ELISA试剂盒)-induced improvement of glucose intolerance in mice fed an HFD.
HIF-1alpha (显示 HIF1A ELISA试剂盒) and GDF15 expression are inversely related under normoxia and hyperoxia.
This is the first study defining the role of Mic-1/Gdf15 in a number of behavioural domains.
epithelial restitution was promoted by enhanced NAG-1 expression and subsequent enterocyte locomotion during the early wound-healing process
Bone morphogenetic proteins (e.g., BMP9\; MIM 605120) are members of the transforming growth factor-beta (see TGFB1\; MIM 190180) superfamily and regulate tissue differentiation and maintenance. They are synthesized as precursor molecules that are processed at a dibasic cleavage site to release C-terminal domains containing a characteristic motif of 7 conserved cysteines in the mature protein.
growth differentiation factor 15
, NSAID (nonsteroidal anti-inflammatory drug)-activated protein 1
, NSAID-activated gene 1 protein
, NSAID-regulated gene 1 protein
, growth/differentiation factor 15
, macrophage inhibitory cytokine 1
, placental TGF-beta
, placental bone morphogenetic protein
, prostate differentiation factor
, macrophage inhibiting cytokine-1