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Glutathione is important for a variety of biological functions, including protection of cells from oxidative damage by free radicals, detoxification of xenobiotics, and membrane transport. 再加上，我们可以发GSX2 蛋白 (3)和数多这个蛋白质的别的产品。
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Gsx2 is required in septal progenitors for the correct expansion of subventricular zone progenitors specified toward the calretinin(+) subtype
we generated a transgenic mouse line driving Cre and EGFP expression with two putative cis-regulatory modules upstream of the homeobox gene Gsx2 , a critical gene for establishing lateral ganglionic eminence (LGE) identity.
homeodomain transcription factor Gsx2 plays a crucial role in the region-specific control of adult neural stem cells in both persistent and injury-induced neurogenesis
despite their redundant properties, Gsx1 and -2 have distinct interactions with Dlx1 and -2
high levels of Gsx2 suppress dendrocyte precursor cells specification in lateral ganglionic eminence progenitors; its downregulation is required for the transition from neurogenesis to oligodendrogenesis
Results show that Gsx1 and Gsx2 differentially regulate the maturation of LGE progenitors.
Required for retinoid production in the embryonic mouse telencephalon.
repression of Ngn1 by Gsh2 promotes the differentiation of dI3 interneurons
By analysis of Gsh2 in mutant mice, it can be demonstrated that Gsh2 along with Pax6 are directly required for major aspects of pallial-subpallial boundary progenitor cell specification.
Data show that in the Gsx2 mutants Gsx1 is expressed in only a subset of the ventral telencephalic progenitors that normally express Gsx2.
This study identified and validated a three-gene (EMP3, GSX2 and EMILIN3) prognostic signature in Lower-Grade Gliomas.
Data show that GS Homeobox 2 (Gsx2) and Early B-cell factor 1 (Ebf1) combined overexpression in human embryonic stem (hES) cells achieves high yields of medium spiny neurons (MSNs).
4q12 translocations with GSX2 expression identify a CD7(+) acute myeloid leukaemia subset.
Gsx2 negatively regulates neurogenesis from postnatal progenitor cells.
Transgenic mice lacking microRNAs miR-9-2 and miR-9-3 exhibit multiple defects of telencephalic structures which may be brought about by dysregulation of Foxg1, Nr2e1, Gsh2, and Meis2 expression.
ectopically expressed in myeloid leukemic cells with t(4;12)(q11-q12;p13), suggesting that expression of GSH2 was deregulated by the translocation, indicating a variant leukemogenic mechanism for translocations involving the 5' end of ETV6
Gsx2 specifies striatal projection neuron and olfactory bulb interneuron identity at distinct time points during neurogenesis in transgenic mice.
Glutathione is important for a variety of biological functions, including protection of cells from oxidative damage by free radicals, detoxification of xenobiotics, and membrane transport. The protein encoded by this gene functions as a homodimer to catalyze the second step of glutathione biosynthesis, which is the ATP-dependent conversion of gamma-L-glutamyl-L-cysteine to glutathione. Defects in this gene are a cause of glutathione synthetase deficiency.
genomic screened homeo box 2
, homeobox protein GSH-2
, genomic screened homeo box 2 protein
, GS homeobox 2
, GS homeobox 2-like
, genomic-screened homeobox 2