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ANKH encodes a multipass transmembrane protein that is expressed in joints and other tissues and controls pyrophosphate levels in cultured cells. 再加上，我们可以发ANKH 蛋白 (5)和数多这个蛋白质的别的产品。
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Human Polyclonal ANKH Primary Antibody for IHC - ABIN965553
Williams: Familial calcium pyrophosphate dihydrate deposition disease and the ANKH gene. in Current opinion in rheumatology 2003
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Human Polyclonal ANKH Primary Antibody for IHC - ABIN965554
Ho, Johnson, Kingsley: Role of the mouse ank gene in control of tissue calcification and arthritis. in Science (New York, N.Y.) 2000
Show all 3 Pubmed References
Craniometaphyseal dysplasia mutations in ANKH diminish hiPSC differentiation into osteoclasts in vitro.
our findings demonstrate that the miR (显示 MLXIP 抗体)-20a/ANKH axis mediates the stiff matrix- promoted CEP calcification, suggesting that miR (显示 MLXIP 抗体)-20a and ANKH are potential targets in restraining the progression of IDD (显示 COL9A3 抗体).
Results show that ANK localizes to the trans-Golgi network (TGN), clathrin-coated vesicles and the plasma membrane, and is required for endosomal function and endocytosis
This study validates the association between a functional polymorphism in the 5' UTR (显示 UTS2R 抗体) of ANKH and Chondrocalcinosis
Polymorphisms in ALP (显示 ALP 抗体), ENPP1 (显示 ENPP1 抗体) and ANKH are important genetic risk factors contributing to Pseudoxanthoma elasticum
ANK (显示 ANK1 抗体) was concentrated around crystal deposits and correlated with markers of chondrocyte hypertrophy. These findings support a role for ANK (显示 ANK1 抗体) in CPPD crystal formation in cartilage.
expression levels of type II collagen (显示 COL2A1 抗体), aggrecan (显示 ACAN 抗体), and ANK (显示 ANK1 抗体) in endplate chondrocytes of experimental group were lower than that of control group and phosphorylation level of JNK (显示 MAPK8 抗体) in the experimental group which was higher than that in the control group
Analysis of the present CMD (显示 ACAN 抗体) family suggested the presence of a maternal mosaicism in an ANKH mutation, and the mother who was mosaic for the ANKH mutation had no apparent clinical or radiological features of CMD (显示 ACAN 抗体).
The aim of this study was to investigate two mineralization-related genes TNAP and ANKH polymorphisms associated with ankylosing spondylitis (AS) in the North Chinese Han population.
Neither ANKH nor ENPP1 (显示 ENPP1 抗体) mutations are the cause of chondrocalcinosis in these Slovakian families.
Data indicate that ankylosis gene product ANK siRNA suppressed basal and hypotonically-stressed ATP levels.
our study provides evidence that ANK plays a critical role in the adipogenic/osteogenic fate decision of adult mesenchymal precursor cells
regulates the transport of inorganic pyrophosphate, and Ank knock-out mice feature a rapidly forming and thick cementum and increased cementum regeneration
Loss of Ank alters cementoblast gene expression and cervical cementum extracellular matrix composition.
the relationship between the type of temporomandibular disorders (TMD (显示 TTN 抗体)) and ANKH polymorphisms
Phe377del mutation in ANK causes impaired osteoblastogenesis and osteoclastogenesis resulting in hypomineralization and a high bone mass phenotype.
ANK is a positive regulator of osteoblastic and osteoclastic differentiation events toward a mature osteoblastic and osteoclastic phenotype.
Analyses on mineralized tissues showed no decrease in mineralization between ank/ank cementum vs. WT controls. Nanoindentation on enamel, dentin or cementum of ank/ank vs. WT molars revealed no significant difference in hardness and elastic modulus.
endogenous expression of ANKH mRNA and protein in whole mouse kidney as well as mouse renal epithelial cell lines and a osteoblast cell line.
Linked regulatory effects on extracellular PP(i) and OPN (显示 SPP1 抗体) expression mediate the ability of PC-1 (显示 PCSK1 抗体) and ANK to regulate calcification.
intra-renal and subcellular localisation of ANK is consistent with pyrophosphate export from collecting duct cells and supports a role for ANK in limiting intra-renal calcium-crystal formation
This gene encodes a multipass transmembrane protein that is expressed in joints and other tissues and controls pyrophosphate levels in cultured cells. Progressive ankylosis-mediated control of pyrophosphate levels has been suggested as a possible mechanism regulating tissue calcification and susceptibility to arthritis in higher animals. Mutations in this gene have been associated with autosomal dominant craniometaphyseal dysplasia.
ankylosis, progressive homolog
, progressive ankylosis protein homolog
, progressive ankylosis homolog
, progressive ankylosis protein
, ankylosis, progressive homolog (mouse)
, progressive ankylosis protein homolog-like
, fn54 protein
, progressive ankylosis protein homolog B
, progressive ankylosis-like protein
, ankylosis, progressive homolog a