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Human TLR9 ELISA Kit for Sandwich ELISA - ABIN414673
Yang, Xie, Deng, Qin: Expression of soluble Toll-like receptors in pleural effusions. in Chinese medical journal 2010
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Rat (Rattus) TLR9 ELISA Kit - ABIN368321
Tunc, Aydemir, Karaoglu, Cekmez, Kul, Aydinoz, Babacan, Yaman, Sarici: Toll-like receptor levels and caffeine responsiveness in rat pups during perinatal period. in Regulatory peptides 2013
analysis of TLR2 (显示 TLR2 ELISA试剂盒) rs5743708, TLR2 (显示 TLR2 ELISA试剂盒) rs4696480, TLR4 (显示 TLR4 ELISA试剂盒) rs4986790, TLR9 rs5743836 and TLR9 rs352140 SNPs in children with pneumococcal and meningococcal meningitis and their family members; study did not show an association between the analyzed SNPs in TLR2 (显示 TLR2 ELISA试剂盒), TLR4 (显示 TLR4 ELISA试剂盒), and TLR9 and susceptibility to develop bacterial meningitis
The present study investigated the effects of vitamin D3 on the expression of TLR3 (显示 TLR3 ELISA试剂盒), TLR7 (显示 TLR7 ELISA试剂盒), and TLR9 in Systemic lupus erythematosus patients.
There appears to be a defect of TLR 9 signaling in patients with tuberculosis, the nature of which needs to be further explored.
sickle red blood cell induced TLR9, NLRP3 (显示 NLRP3 ELISA试剂盒), Caspase-1 (显示 CASP1 ELISA试剂盒), IL-1beta (显示 IL1B ELISA试剂盒) and IL-18 (显示 IL18 ELISA试剂盒) expression and induced IL-1beta (显示 IL1B ELISA试剂盒), LTB4 (显示 PTGR1 ELISA试剂盒) and nitrite production in PBMC cultures.
Findings indicate that genotype TT in the -1486 locus of the TLR9 gene could comprise a risk genotype for HPV infection, squamous intraepithelial cervical lesion, and uterine cervical neoplasm in Mexican female population.
data show that TLR-5 (显示 TLR5 ELISA试剂盒) and TLR-9 are susceptible genes to lupus nephritis (LN) and that their expression is dysregulated in LN patients' kidneys, supporting a role of these mediators in the pathogenesis of LN.
By CRISPR/Cas9-induced inactivation of TLR9, MyD88 (显示 MYD88 ELISA试剂盒), IRAK4 (显示 IRAK4 ELISA试剂盒) and IRAK1 (显示 IRAK1 ELISA试剂盒) we confirm that BZLF1 repression is dependent on functional TLR9 and MyD88 (显示 MYD88 ELISA试剂盒) signaling, and identify IRAK4 (显示 IRAK4 ELISA试剂盒) as an essential element for TLR9-induced repression of BZLF1 expression upon BCR (显示 BCR ELISA试剂盒) cross-linking
results imply that TLR9-mediated activation of B-cells not only promotes cell survival, but may via p53 (显示 TP53 ELISA试剂盒) provide cells with a barrier against harmful consequences of enhanced activation and proliferation
mutually exclusive transcriptional regulation by AP-1 (cjun (显示 JUN ELISA试剂盒)/cfos) and non-canonical NF-kappaB (显示 NFKB1 ELISA试剂盒) (RelB (显示 RELB ELISA试剂盒)/p52 (显示 FKBP4 ELISA试剂盒)) downstream of MEK (显示 MAP2K1 ELISA试剂盒)-ERK (显示 EPHB2 ELISA试剂盒) and NIK (显示 MAP3K14 ELISA试剂盒)-IKK-alpha (显示 CHUK ELISA试剂盒)-NF-kappaB2 (p100 (显示 CUX1 ELISA试剂盒)) phosphorylation, respectively was responsible for persistent Ccl20 (显示 CCL20 ELISA试剂盒) expression in the colonic cells.
This study evaluation the roles of SOCS1 (显示 SOCS1 ELISA试剂盒), the regulator of TLR9, RIG-I (显示 DDX58 ELISA试剂盒), and CD152 (显示 CTLA4 ELISA试剂盒) in patients with liver fibrosis/cirrhosis; the use of polymorphisms as markers for genetic risk is reported.
TLR9-driven inflammation induced a Ccr2-independent expansion of functionally enhanced extramedullary myeloid progenitors that correlated with the peripheral accumulation of monocytes in both wild-type and Ccr2(-/-) mice.
TLR9 expression on B1a cells is not critical for their IgM-dependent atheroprotection.
This study establishes a correlation between TLR-3 (显示 TLR3 ELISA试剂盒) and TLR-9 expression with the development of EAE. In addition, evidence of a role for the myelin peptide in targeting the innate inflammatory response to the CNS is presented.
these data demonstrate that TLR7 (显示 TLR7 ELISA试剂盒)/TLR9 ligands push the macrophage into a phagocytic long-lived cell, with a decreased capacity of antigen presentation and reminiscent of the M2 polarized state.
TLR9/MyD88 (显示 MYD88 ELISA试剂盒) signaling selectively in CD11c (显示 ITGAX ELISA试剂盒)(+) dendritic cells (DCs) strongly enhances murine cytomegalovirus clearance.
High-glucose induces tau hyperphosphorylation through activation of TLR9-P38 MAPK (显示 MAPK14 ELISA试剂盒) pathway.
proposed that TLR9 regulates the NF-kappaB (显示 NFKB1 ELISA试剂盒)-NLRP3 (显示 NLRP3 ELISA试剂盒)-IL-1beta (显示 IL1B ELISA试剂盒) pathway negatively in Salmonella-induced NKG2D (显示 KLRK1 ELISA试剂盒)-mediated intestinal inflammation and plays a critical role in defense against S. typhimurium infection and in the protection of intestinal integrity.
mechanism integrating BCR, TLR9, and cytokine signals provides a peripheral checkpoint for DNA-containing antigens that, if circumvented by survival and differentiative cues, yields B cells with the autoimmune-associated T-bet+ phenotype.
we demonstrated for the first time that the cross-talk of TLR2 (显示 TLR2 ELISA试剂盒) and TLR9 triggered Th1 (显示 HAND1 ELISA试剂盒) activation collaboratively
the cooperative role of TLR9 with TLR2 (显示 TLR2 ELISA试剂盒) or TLR6 (显示 TLR6 ELISA试剂盒) receptors in sensing Brucella, was determined.
TLR9 immunoreactivity is mainly detected in epithelial cells and antigen presenting cells, namely dendritic and macrophage-like cells, within the range of tissues examined. The pattern of TLR9 expression was similar in pigs of 3 weeks and 3 months of age.
These data demonstrated that TLR2 (显示 TLR2 ELISA试剂盒), TLR3 (显示 TLR3 ELISA试剂盒) and TLR9 contribute to NF-kappaB (显示 NFKB1 ELISA试剂盒) activation in response to porcine epidemic diarrhea virus infection, but not RIG-I (显示 DDX58 ELISA试剂盒).
expression of TLR3 (显示 TLR3 ELISA试剂盒), TLR7 (显示 TLR7 ELISA试剂盒) and TLR9 in alveolar macrophages infected with different genotype 1 PRRSV strains
we studied TLR9 expression in lung extracts from pigs, dogs, and cattle.
Moreover, the TLR9 transfectant demonstrated its usefulness for evaluation of immunostimulation by bacterial DNA through the detection of T(H)-1, T(H)-2 type cytokine induction via TLR9 signaling.
variants in the TLR1 (显示 TLR1 ELISA试剂盒) gene are associated with susceptibility to bovine tuberculosis, whereas no significant association can be inferred from the polymorphisms in the TLR9 gene
mRNA abundances of TLR9, TLR2 (显示 TLR2 ELISA试剂盒), and TLR4 (显示 TLR4 ELISA试剂盒) together with those of beta-defensin 5 (BNBD5 (显示 DEFB105A ELISA试剂盒)), an early bactericidal effector molecule of the innate system, in healthy and infected mammary glands
substantial conservation of TLR9 structure and TLR9 function in blood leukocytes
The protein encoded by this gene is a member of the Toll-like receptor (TLR) family which plays a fundamental role in pathogen recognition and activation of innate immunity. TLRs are highly conserved from Drosophila to humans and share structural and functional similarities. They recognize pathogen-associated molecular patterns (PAMPs) that are expressed on infectious agents, and mediate the production of cytokines necessary for the development of effective immunity. The various TLRs exhibit different patterns of expression. This gene is preferentially expressed in immune cell rich tissues, such as spleen, lymph node, bone marrow and peripheral blood leukocytes. Studies in mice and human indicate that this receptor mediates cellular response to unmethylated CpG dinucleotides in bacterial DNA to mount an innate immune response.
Toll-like receptor 9 protein