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抗Human VEGFB 抗体:
抗Mouse (Murine) VEGFB 抗体:
抗Rat (Rattus) VEGFB 抗体:
Data show that metformin treatment reduces serum vascular endothelial growth factor B (VEGF-B) levels and ameliorates insulin (显示 INS 抗体) resistance.
VEGFA (显示 VEGFA 抗体) and VEGFB associated sub-network, kinase insert domain receptor (KDR (显示 KDR 抗体)), fibronectin 1 (FN1 (显示 FN1 抗体)), transforming growth factor beta induced (TGFBI (显示 TGFBI 抗体)) and proliferating cell nuclear antigen (PCNA (显示 PCNA 抗体)) found to interact with at least two of the three hub genes.
Frameshift mutations of VEGFB gene is associated with stomach and colorectal cancers.
plasma VEGF levels before treatment were lower in patients with schizophrenia and that their VEGF levels increased after treatment.
fluid shear stress induces the synthesis of Insulin growth factor-2 and vascular endothelial growth factor (VEGF) B and D, which in turn transactivate MMP-12.
MMP9 (显示 MMP9 抗体) may activate VEGF-B via PI3K (显示 PIK3CA 抗体)/Akt (显示 AKT1 抗体) signaling pathway.
Roles of vascular endothelial growth factor in amyotrophic lateral sclerosis.
Low VEGFB and VEGFD (显示 Figf 抗体) gene expression is associated with early-stage non-small cell lung cancer.
Our study suggested that VEGF-B was an angiogenesis factor in vitro and that ERK1/2 (显示 MAPK1/3 抗体) and p38 (显示 CRK 抗体)-related signaling pathways were involved in these VEGF-B activities.
VEGF-B has possible roles in cardiac protection, energy metabolism support, and neuroprotectin [review]
These data therefore support a tightly controlled, paracrine signaling mechanism of VEGF-B to VEGFR1 (显示 FLT1 抗体).
VEGFB-Induced Vascular Remodeling in Adipose Tissue Requires VEGF (显示 VEGFA 抗体)/VEGFR2 (显示 KDR 抗体). VEGFB-Induced Vascular Remodeling Improves Insulin (显示 INS 抗体) Supply, Signaling, and Function in Obese Mice.
VEGF-B is dispensable for normal cardiac function under unstressed conditions and for high fat diet-induced metabolic changes.
VEGF-B is a vascular remodeling factor promoting cancer metastasis.
Data indicate that vascular endothelial growth factor B knockout (Vegf-b-/-) mice showed impaired nerve repair with concomitant impaired trophic function.
Data indicate that VEGF-B is a high-affinity VEGFR-1 (显示 FLT1 抗体) ligand that, unlike PlGF (显示 PGF 抗体), cannot efficiently induce signaling downstream of VEGFR-1 (显示 FLT1 抗体).
results demonstrate that the vascular endothelium can function as an efficient barrier to excess muscle lipid uptake even under conditions of severe obesity and type 2 diabetes, and that this barrier can be maintained by inhibition of VEGF-B signalling
the RTEF-1 (显示 TEAD4 抗体)-driven increase of VEGF-B plays an important role in communication between the endothelium and myocardium
Study indicates that VEGF-B, instead of acting as an angiogenic factor (显示 VEGFA 抗体), exerts direct neuroprotective effects through FLT1 (显示 FLT1 抗体).
Data show that no differences in vascular density, perfusion or immune cell infiltration upon altered Vegfb gene dosage were noted.
PCR-SSCP and DNA sequencing methods were applied to reveal 3 SNPs and a duplication in the bovine VEGF-B gene among 675 samples belonging to three native Chinese cattle breeds.
This gene encodes a member of the PDGF (platelet-derived growth factor)/VEGF (vascular endothelial growth factor) family. The VEGF family members regulate the formation of blood vessels and are involved in endothelial cell physiology. This member is a ligand for VEGFR-1 (vascular endothelial growth factor receptor 1) and NRP-1 (neuropilin-1). Studies in mice showed that this gene was co-expressed with nuclear-encoded mitochondrial genes and the encoded protein specifically controlled endothelial uptake of fatty acids. Alternatively spliced transcript variants encoding distinct isoforms have been identified.