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抗Human NOX1 抗体:
抗Rat (Rattus) NOX1 抗体:
抗Mouse (Murine) NOX1 抗体:
Human Polyclonal NOX1 Primary Antibody for ICC, IF - ABIN441545
Balasubramaniyan, Wright, Sharma, Davies, Sharifi, Habtesion, Mookerjee, Jalan: Ammonia reduction with ornithine phenylacetate restores brain eNOS activity via the DDAH-ADMA pathway in bile duct-ligated cirrhotic rats. in American journal of physiology. Gastrointestinal and liver physiology 2011
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Human Polyclonal NOX1 Primary Antibody for WB - ABIN4893182
Quintela, Jiménez, Gómez-Guzmán, Zarzuelo, Galindo, Sánchez, Vargas, Cogolludo, Tamargo, Pérez-Vizcaíno, Duarte: Activation of peroxisome proliferator-activated receptor-β/-δ (PPARβ/δ) prevents endothelial dysfunction in type 1 diabetic rats. in Free radical biology & medicine 2012
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Human Polyclonal NOX1 Primary Antibody for IHC (p), WB - ABIN3044132
Wang, Wang, Liu, Wang, Zhao, Wang, Yin: Cordyceps sinensis polysaccharide inhibits PDGF-BB-induced inflammation and ROS production in human mesangial cells. in Carbohydrate polymers 2015
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Mouse (Murine) Polyclonal NOX1 Primary Antibody for IHC (p), WB - ABIN3044251
Gu, Gong, Zhang, Dong, Zhao, Burczynski, Wang, Sun, Zhu, Han, Wang, Li: Regulation of transforming growth factor beta 1 gene expression by dihydropteridine reductase in kidney 293T cells. in Biochemistry and cell biology = Biochimie et biologie cellulaire 2013
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Cow (Bovine) Polyclonal NOX1 Primary Antibody for WB - ABIN2782754
Kuwano, Kawahara, Yamamoto, Teshima-Kondo, Tominaga, Masuda, Kishi, Morita, Rokutan: Interferon-gamma activates transcription of NADPH oxidase 1 gene and upregulates production of superoxide anion by human large intestinal epithelial cells. in American journal of physiology. Cell physiology 2006
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Human Polyclonal NOX1 Primary Antibody for IF (p), IHC (p) - ABIN702580
Kashiwabara, Ambe, Nakagawa, Watanabe: Immunohistochemical localization of Nox in mouse circumvallate papillae. in Tissue & cell 2015
Human Polyclonal NOX1 Primary Antibody for ICC, IF - ABIN4335324
Fan, Hsiung, Cheng, Tzanakakis: Facile engineering of xeno-free microcarriers for the scalable cultivation of human pluripotent stem cells in stirred suspension. in Tissue engineering. Part A 2014
These results are consistent with the hypothesis that antioxidants or NOX1/4 inhibition may be useful in blocking profibrotic effects of TGFbeta (显示 TGFB1 抗体) on dermal and gingival fibroblasts and warrant consideration for further development as potential antifibrotic agents
We demonstrated that rapid deletion of p22phox (显示 CYBA 抗体) is possible and that the activity of Nox1 and Nox4 (显示 NOX4 抗体) but not Nox5 (显示 NOX5 抗体) exclusively depends on p22phox (显示 CYBA 抗体).
PAK4 (显示 PAK4 抗体) downregulation decreased PPARgamma (显示 PPARG 抗体)-mediated Nox1 expression and suppressed EMT (显示 ITK 抗体) in IR-treated glioma cells.
Nox1-SHH (显示 SHH 抗体)-Grem1 (显示 GREM1 抗体) signaling axis in pulmonary vascular endothelium that is likely to contribute to Pulmonary hypertension.
5-HT1B receptor-dependent cellular Src (显示 SRC 抗体)-related kinase-Nox1-pathways contribute to vascular remodeling in pulmonary arterial hypertension.
NOX1 has a role in maintaining the proliferative phenotype of some colon cancers and has potential as a therapeutic target in this disease
NOX1 mRNA was undetectable in the gastric mucosa.
P38 MAPK (显示 MAPK14 抗体), phosphorylated P38 MAPK (显示 MAPK14 抗体), and RAC2 (显示 RAC2 抗体) regulated in mutual feedback and negative feedback regulatory pathways, resulting in the radioresistance of G0 cells.
NS5A contributes to reactive oxygen species production by activating expression of NADPH (显示 NQO1 抗体) oxidases 1 and 4 as well as cytochrome P450 2E1 (显示 CYP2E1 抗体).
our results highlight that the Nox1/AKT (显示 AKT1 抗体) signaling pathway plays an important role in cell survival in oral squamous cell carcinoma (OSCC) cells.
Both Nox1 and Duox2 (显示 DUOX1 抗体) induce exfoliation of crypt epithelium, but only Nox1 induces apoptosis. NOX1 and DUOX2 (显示 DUOX1 抗体) may be potential therapeutic targets for treating ileocolitis in human patients suffering inflammatory bowel disease (IBD).
we used the Ang II (显示 AGT 抗体) infused hph-1 (显示 EGLN2 抗体) mice to examine the roles of NOX isoforms in the development of AAA (显示 AAAS 抗体). We generated double mutants of hph-1 (显示 EGLN2 抗体)-NOX1, hph-1 (显示 EGLN2 抗体)-NOX2 (显示 CYBB 抗体), hph-1 (显示 EGLN2 抗体)-p47phox, and hph-1 (显示 EGLN2 抗体)-NOX4 (显示 NOX4 抗体)
C-kit (显示 KIT 抗体)-positive hematopoietic stem/progenitor cells expressed significantly higher of Nox1 and catalase (显示 CAT 抗体), but less of lactoperoxidase (显示 LPO 抗体) than in matured mononuclear cells.
Nox1 deletion reduces oxidant load and restores microvascular health in obese mice.
Data suggests that ROS (显示 ROS1 抗体) produced during primitive endoderm differentiation is dependent in part on increased NOX1 and NOX4 (显示 NOX4 抗体) levels, which is under the control of GATA6 (显示 GATA6 抗体). Furthermore, these results suggest that the combined activity of multiple NOX proteins is necessary for the differentiation of F9 cells to primitive endoderm.
Gp91phox (显示 CYBB 抗体) NADPH oxidase modulates litter size by up-regulating mucin1 (显示 MUC1 抗体) expression in the uterus of mice.
NOX4 (显示 NOX4 抗体)- and NOX1-derived ROS (显示 ROS1 抗体) contribute to atherosclerosis in the aortic sinus of diabetic ApoE (显示 APOE 抗体) knockout mice.
Data show that pan (显示 SUPT6H 抗体)-NOX-inhibitor APX (显示 SHROOM1 抗体)-115 treatment decreased NADPH oxidase (Nox) Nox1, Nox2 (显示 CYBB 抗体), and Nox4 (显示 NOX4 抗体) protein expression in the kidney.
NOX1 is selectively important in GPCR (显示 GPBAR1 抗体)-dependent intracellular ROS (显示 ROS1 抗体) generation but dispensable for GPVI (显示 GP6 抗体)-ITAM-dependent ROS (显示 ROS1 抗体) generation in blood platelets..
NADPH oxidase plays an important role in proMMP-2 expression and activation and MMP-2 (显示 MMP2 抗体) mediated SMC (显示 DYM 抗体) proliferation occurs through the involvement of Spm (显示 NPC1 抗体)-Cer (显示 CBLN1 抗体)-S1P (显示 MBTPS1 抗体) signaling axis under ANG II (显示 AGT 抗体) stimulation of PASMCs
Differential Roles of Protein Complexes NOX1-NOXO1 and NOX2-p47phox in Mediating Endothelial Redox Responses to Oscillatory and Unidirectional Laminar Shear Stress.
The current study was designed to determine mechanisms underlying 20-hydroxyeicosatetraenoic acid -stimulated nitric oxide (NO) release, and particularly the role of NADPH oxidase, reactive oxygen species, and PI3-kinase (显示 PIK3CA 抗体) in stimulated NO release.
NEP (显示 MME 抗体) expression is down-regulated in vascular endothelial cells by physiological laminar shear, possibly via a mechanotransduction mechanism involving NADPH oxidase-induced reactive oxygen species production.
The nox1 expression in zebrafish during early nervous system development from 12 to 48 hours post fertilization.
Over inhibition of the NADPH oxidase by the NADPH (显示 NQO1 抗体) Inhibitor DPI (显示 DSP 抗体) may reduce the cell even the tissue in the progress of healing after the injury, in zebrafish liver cells.
This gene encodes a member of the NADPH oxidase family of enzymes responsible for the catalytic one-electron transfer of oxygen to generate superoxide or hydrogen peroxide. Alternatively spliced transcript variants encoding multiple isoforms have been observed for this gene.
NADH/NADPH mitogenic oxidase subunit P65-MOX
, NADPH oxidase homolog-1
, mitogenic oxidase (pyridine nucleotide-dependent superoxide-generating)
, mitogenic oxidase 1
, NADH/NADPH mitogenic oxidase subunit p65-mox
, GP91 phox homolog
, NADPH oxidase 1 alpha
, NADPH oxidase-1
, predicted NADPH oxidase-1
, NADPH oxidase 1