anti-SAG (SAG) 抗体产品概述

Full name:
anti-S-Antigen, Retina and Pineal Gland (Arrestin) 抗体 (SAG)
在www.antibodies-online.cn可供76 S-Antigen, Retina and Pineal Gland (Arrestin) (SAG) 抗体的15不同的供货商。 再加上,我们可以发SAG 试剂盒 (25)SAG 蛋白 (13)和数多这个蛋白质的别的产品。 总共114 SAG产品已列进来了。
A930001K18Rik, ARR, Arr1, arrestin, Irbp, MGC75721, MGC84416, RP47, S-AG, sag, sag1, SAGMR, SANTI, zgc:114197

最受欢迎的抗anti-SAG (SAG) 抗体


抗Mouse (Murine) SAG 抗体:

抗Human SAG 抗体:

抗Rat (Rattus) SAG 抗体:

所有可销售的anti-SAG 抗体


引用最多的anti-SAG 抗体

  1. Human Monoclonal SAG Primary Antibody for ICC, IF - ABIN1580413 : Dorey, Faure: [Isolation and characterization of a retinal antigen inducing experimental autoimmune uveo-retinitis]. in Annales d'immunologie 1977 (PubMed)
    Show all 4 references for 1580413

  2. Cow (Bovine) Monoclonal SAG Primary Antibody for ICC, IHC (fro) - ABIN250780 : Banga, LeRoy, Suleyman, Kasp, Brown, Dumonde: Analysis of antigenic determinants of retinal S-antigen with monoclonal antibodies. in Investigative ophthalmology & visual science 1988 (PubMed)
    Show all 3 references for 250780

  3. Rat (Rattus) Monoclonal SAG Primary Antibody for ICC, IHC (fro) - ABIN108637 : Banga, Suleyman, Kasp, Brown, LeRoy, Sanders, Dumonde: Immunoaffinity purification of S-antigen using monoclonal antibodies to different antigenic sites. in Investigative ophthalmology & visual science 1987 (PubMed)
    Show all 2 references for 108637


Mouse (Murine) S-Antigen, Retina and Pineal Gland (Arrestin) (SAG) interaction partners

  1. The G-protein coupled receptor, DRD4, requires ARR1 and ARR4 for desensitization and internalization.

  2. ARR4 modulates essential functions in high acuity vision and downstream cellular signaling pathways that are not fulfilled or substituted by the coexpression of ARR1, despite its high expression levels in all mouse cones.

  3. crystal structure of a constitutively active form of human rhodopsin (显示 RHO 抗体) bound to a pre-activated form of the mouse visual arrestin, determined by serial femtosecond X-ray laser crystallography

  4. Sag (显示 RNF7 抗体) is essential for embryonic vasculogenesis and tumor angiogenesis.

  5. SAG (显示 RNF7 抗体) knockdown caused the accumulation of proapoptotic Bax (显示 BAX 抗体) and SARM (显示 SARM1 抗体), imbalance of Bcl-2 (显示 BCL2 抗体)/Bax (显示 BAX 抗体) in the mitochondria, induction of cytosolic cytochrome c (显示 CYCS 抗体) and activation of caspases, all of which led to disequilibrium between life and death of macrophages.

  6. tetrameric visual arrestin 1 is a biomarker for retinal function in diabetic mice, assessed by MRI (显示 C7ORF49 抗体)

  7. The data suggest that monomeric arrestin-1 is cytotoxic and WT arrestin-1 protects rods by forming mixed oligomers with the mutant and/or competing with it for the binding to non-receptor partners.

  8. Findings suggest a role for Bardet-Biedl syndrome 5 (BBS5) in regulating light-dependent translocation of arrestin1 (Arr1).

  9. Visual arrestin interaction with clathrin adaptor AP-2 (显示 TFAP2A 抗体) regulates photoreceptor survival in the vertebrate retina.

  10. the 139-loop stabilizes basal conformation of arrestin-1 and acts as a brake, preventing its binding to non-preferred forms of rhodopsin (显示 RHO 抗体).

Human S-Antigen, Retina and Pineal Gland (Arrestin) (SAG) interaction partners

  1. This is the first dominant-acting mutation identified in SAG (显示 DMBT1 抗体), a founder mutation possibly originating in Mexico several centuries ago. The phenotype is clearly adRP (显示 PLIN2 抗体) and is distinct from the previously reported phenotypes of recessive null mutations, that is, Oguchi disease and recessive RP.

  2. Macular dysfunction can occur in Oguchi disease with the 1147delA mutation in the SAG (显示 DMBT1 抗体) gene.

  3. NEDD4-1 (显示 NEDD4 抗体) overexpression sensitizes cancer cells to etoposide-induced apoptosis by reducing SAG (显示 DMBT1 抗体) levels through targeted degradation. SAG (显示 DMBT1 抗体) is added to a growing list of NEDD4-1 (显示 NEDD4 抗体) substrates and mediates its biological function.

  4. Sag (显示 DMBT1 抗体) is a Kras-cooperating oncogene (显示 RAB1A 抗体) that promotes lung tumorigenesis

  5. Based on their observed affinity for arrestin-1, P-opsin (显示 RHO 抗体) and inactive P-Rh very likely affect the physiological monomer-dimer-tetramer equilibrium of arrestin-1, and should therefore be taken into account when modeling photoreceptor function.

  6. Compound heterozygosity of a nonsense R193X mutation and a heterozygous deletion of 3,224 bp encompassing exon 2 in the SAG (显示 DMBT1 抗体) gene is the cause of Oguchi's disease in a Chinese family.

  7. Identification of autoantibodies specific for two retinal antigens (CRALBP (显示 RLBP1 抗体) and S-Ag) supports the concept of an autoimmunological origin of the disease.

  8. the arrestin 1147delA, which has been known as a frequent cause of Oguchi disease, also may be related to the pathogenesis of autosomal recessive RP.

  9. We describe a case of Oguchi disease with unusual findings caused by a putative heterozygous mutation in the SAG (显示 DMBT1 抗体) gene.

  10. maintenance of low levels of the active monomer is the biological role of arrestin-1 self-association

Cow (Bovine) S-Antigen, Retina and Pineal Gland (Arrestin) (SAG) interaction partners

  1. Conformational changes are involved in the arrestin-rhodopsin (显示 RHO 抗体) binding interface through multiple docking modes.

  2. K2A mutations in arrestin-1, -2, and -3 significantly reduced their binding to active phosphorhodopsin.

  3. maintenance of low levels of the active monomer is the biological role of arrestin-1 self-association

  4. We hypothesize that, although arrestin requires at least a single Rho*P to bind the membrane, a single arrestin can actually interact with a pair of receptors

  5. variant form of arrestin-1 binds rod outer segment membranes

  6. Models suggest that the phosphorylated carboxy-terminal region of rhodopsin (显示 RHO 抗体), Rh(330-348), undergoes significant conformational changes and becomes structured upon binding to arrestin.

  7. arrestin and p44 bind differently to different phosphorylated rhodopsin species and that this may be due to a structural difference between p44's and arrestin's basal states.

SAG 抗原简介

Antigen Summary

may act as a GTP/GDP-binding protein\; may play a role in signal transduction in the retina

Alternative names and synonyms associated with SAG

  • S-antigen; retina and pineal gland (arrestin) (sag) 抗体
  • S-antigen; retina and pineal gland (arrestin) b (sagb) 抗体
  • S-antigen (PF10_0343) 抗体
  • S-antigen; retina and pineal gland (arrestin) (SAG) 抗体
  • arrestin (sag) 抗体
  • S-antigen, retina and pineal gland (arrestin) (Sag) 抗体
  • S-antigen; retina and pineal gland (arrestin) (Sag) 抗体
  • A930001K18Rik 抗体
  • ARR 抗体
  • Arr1 抗体
  • arrestin 抗体
  • Irbp 抗体
  • MGC75721 抗体
  • MGC84416 抗体
  • RP47 抗体
  • S-AG 抗体
  • sag 抗体
  • sag1 抗体
  • SAGMR 抗体
  • SANTI 抗体
  • zgc:114197 抗体

Protein level used designations for SAG

S-antigen; retina and pineal gland (arrestin) , -antigen; retina and pineal gland (arrestin) , Arrestin Sb , S-antigen , s-arrestin-like , S-AG , S-arrestin , retinal S-antigen , rod photoreceptor arrestin , 48 kDa protein , arrestin 1 , rod arrestin , visual arrestin 1 , retinal S-antigen (48 KDa protein) , arrestin1 , arrestin, S-antigen

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