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抗Human CCL21 抗体:
抗Mouse (Murine) CCL21 抗体:
抗Rat (Rattus) CCL21 抗体:
Human Polyclonal CCL21 Primary Antibody for IHC, IHC (p) - ABIN4288804
Andersson, Nilsson, Fagerberg, Hallström, Sundström, Danielsson, Edlund, Uhlen, Asplund: The transcriptomic and proteomic landscapes of bone marrow and secondary lymphoid tissues. in PLoS ONE 2014
Human Polyclonal CCL21 Primary Antibody for IF (p), IHC (p) - ABIN732698
Borrelli, Abrão, Taube, Darb-Esfahani, Köhler, Kaufmann, Chiantera, Mechsner: Immunohistochemical Investigation of Metastasis-Related Chemokines in Deep-Infiltrating Endometriosis and Compromised Pelvic Sentinel Lymph Nodes. in Reproductive sciences (Thousand Oaks, Calif.) 2015
CCL21/IL21 (显示 IL17C 抗体)-armed oncolytic adenovirus enhances antitumor activity against TERT (显示 TERT 抗体)-positive tumor cells.
plasmin (显示 PLG 抗体) cleaves surface-bound CCL21 to release the C-terminal peptide responsible for CCL21 binding to glycosaminoglycans on the extracellular matrix and cell surfaces, thereby generating the soluble form.
the white pulp regions of ME7-infected spleens were smaller, and contained markedly diminished T zones, as compared to control spleens. Although lymphoid tissue inducer cells were not affected, the expression of both CCL19 (显示 CCL19 抗体) and CCL21 was decreased.
Taken together, these results suggest that SERCA2 (显示 ATP2A2 抗体) contributes to the migration of CCL21-activated Dendritic Cells as an important feature of the adaptive immune response and provide novel insights regarding the role of SERCA2 (显示 ATP2A2 抗体) in Dendritic Cells functions.
An expanded lymphatic network is capable of enhanced chemoattractant CCL21 production, and lymphangiogenesis will facilitate initial lymph formation favoring increased clearance of fluid in situations of augmented fluid filtration.
Results provide evidence for an association between an increase level of CCL21 and IP-10 (显示 CXCL10 抗体) in the blood and pulmonary involvement in systemic lupus erythematosus patients.
Gata1 (显示 GATA1 抗体)-KO(DC) DCs have reduced polysialic acid levels on their surface, which is a known determinant for the proper migration of DCs toward CCL21.
CCL21 and CXCL13 (显示 CXCL13 抗体) levels are increased in the minor salivary glands of patients with Sjogren's syndrome.
Deletion of this extended C-terminus reduces CCL21's affinity for heparin and transferring the CCL21 C-terminus to CCL19 (显示 CCL19 抗体) enhances heparin binding mainly through non-specific, electrostatic interactions
CCL21/CCR7 (显示 CCR7 抗体) interaction contributes to the time-dependent proliferation of PTC (显示 F9 抗体) cells by upregulating cyclin A (显示 CCNA2 抗体), cyclin B1 (显示 CCNB1 抗体) and cyclin-dependent kinase 1 (CDK1 (显示 CDK1 抗体)) expression via the extracellular signal-regulated kinase (ERK (显示 EPHB2 抗体)) pathway associated with iodine.
the CCR7 (显示 CCR7 抗体)/CCL21 signaling pathway leading to T lymphocyte migration on fibronectin (显示 FN1 抗体) is a beta1 integrin-dependent pathway involving transient ERK1/2 (显示 MAPK1/3 抗体) phosphorylation, which is modulated by PLCgamma1 (显示 PLCG1 抗体)
Data suggest that lymphotoxin (显示 LTB 抗体)-dependent and -independent regulation of CC chemokine ligand 21-b and -c plays a role in balancing the central and peripheral immune responses between lymphoid and nonlymphoid tissues.
CCL21 and, to a lesser extent, CCL19 (显示 CCL19 抗体) play significant roles in the distinctive localization of marginal zone macrophages within the splenic marginal zone.
CCL21 and 5'-Nase may be involved in the interaction between infiltrating cells and lymphatic vessels to induce the functional changes of lymphatic endothelial cells during insulitic and diabetic development
CXCL9 (显示 CXCL9 抗体) has a role in graft rejection in the absence of CCL19 (显示 CCL19 抗体) and CCL21
T-cell defects in the nasal-associated lymphoid tissue of lymphotoxin (显示 LTB 抗体)(-/-) mice can be attributed to the impaired expression of CCL21.
alpha-GalCer-loaded dendritic cells expressing tumor-associated antigen along with SLC (显示 CCL21A 抗体) can stimulate multiple subsets of effector cells to induce a potent therapeutic effect against peritoneally disseminated tumor cells.
following Leishmania donovani infection of mice, the migration of splenic DC is regulated by the CCR7 (显示 CCR7 抗体) ligands CCL19 (显示 CCL19 抗体)/CCL21
Results suggest that CCR7 (显示 CCR7 抗体)-positive fibrocytes infiltrate the kidney via CCL21-positive vessels, thereby contributing to the pathogenesis of renal fibrosis.
The presence of CCL21 during the in vitro stimulation of CD4 (显示 CD4 抗体)+ T cells with anti-CD3 (显示 CD3E 抗体) plus anti-CD28 (显示 CD28 抗体) significantly enhanced in vitro AICD induction of the restimulated T cells, partially through enhancing expression of Fas ligand (显示 FASL 抗体).
This gene is one of several CC cytokine genes clustered on the p-arm of chromosome 9. Cytokines are a family of secreted proteins involved in immunoregulatory and inflammatory processes. The CC cytokines are proteins characterized by two adjacent cysteines. Similar to other chemokines the protein encoded by this gene inhibits hemopoiesis and stimulates chemotaxis. This protein is chemotactic in vitro for thymocytes and activated T cells, but not for B cells, macrophages, or neutrophils. The cytokine encoded by this gene may also play a role in mediating homing of lymphocytes to secondary lymphoid organs. It is a high affinity functional ligand for chemokine receptor 7 (CCR7) that is expressed on T and B lymphocytes and a known receptor for another member of the cytokine family (small inducible cytokine A19).
C-C motif chemokine 21
, CC chemokine ligand 21
, putative CCL21 chemokine
, small inducible cytokine A21
, small inducible cytokine A24
, Efficient Chemoattractant for Lymphocytes
, beta chemokine exodus-2
, secondary lymphoid tissue chemokine
, small inducible cytokine subfamily A (Cys-Cys), member 21
, chemokine CCL21/6CKINE
, small-inducible cytokine A21
, C-C motif chemokine 21b
, C-C motif chemokine 21c
, Small-inducible cytokine A21b
, beta-chemokine exodus-2
, small inducible cytokine A21c
, small-inducible cytokine A21c
, thymus-derived chemotactic agent 4