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Human Polyclonal Lp-PLA2 Primary Antibody for IF (p), IHC (p) - ABIN686722
Rosing, Fobker, Kannenberg, Gunia, DellAquila, Kwiecien, Stypmann, Nofer: Everolimus therapy is associated with reduced lipoprotein-associated phospholipase A2 (Lp-Pla2) activity and oxidative stress in heart transplant recipients. in Atherosclerosis 2013
Show all 2 Pubmed References
Human Monoclonal Lp-PLA2 Primary Antibody for ELISA - ABIN563501
Klee, Finlay, McDonald, Attewell, Hebrink, Dyer, Love, Vasmatzis, Li, Beechem, Klee: Bioinformatics methods for prioritizing serum biomarker candidates. in Clinical chemistry 2007
Human Polyclonal Lp-PLA2 Primary Antibody for ELISA, WB - ABIN4238414
Moldoveanu, Serban, Marta, Serban, Huica: Lipoprotein-associated phospholipase A2 activity in patients with preserved left ventricular ejection fraction. in Biomarkers : biochemical indicators of exposure, response, and susceptibility to chemicals 2011
Human Polyclonal Lp-PLA2 Primary Antibody for FACS, IHC (p) - ABIN653776
Bouchareb, Mahmut, Nsaibia, Boulanger, Dahou, Lépine, Laflamme, Hadji, Couture, Trahan, Pagé, Bossé, Pibarot, Scipione, Romagnuolo, Koschinsky, Arseneault, Marette, Mathieu: Autotaxin Derived From Lipoprotein(a) and Valve Interstitial Cells Promotes Inflammation and Mineralization of the Aortic Valve. in Circulation 2015
Lifelong lower Lp-PLA2 activity was not associated with major risks of vascular or non-vascular diseases in Chinese adults.
phospholipase A2 (显示 YWHAZ 抗体) Group 7 (PLA2G7), to be important in regulating tumor cell migration and a novel tumor-promoting factor in nasopharyngeal carcinoma.
Suggest that the plasma biomarkers Lp-PLA2 activity and mass are markers of abdominal aortic aneurysm risk after adjusting for relevant confounders.
The substitution of whole grains and legumes for refined rice resulted in a reduction in Lp-PLA2 activities in plasma and PBMCs partly through improved glycemic control, increased consumption of protein relative to carbohydrate, and reduced lipid peroxides.
High levels of high-sensitive C-reactive protein (显示 CRP 抗体) and lipoprotein-associated phospholipase-A2 did not relate to endothelial dysfunction in ST-elevation myocardial infarction patients treated with percutaneous coronary intervention.
In persistent prehypertension, increased ox-LDL hydrolysis by Lp-PLA2 enhances arterial stiffness without an age-related increase in blood pressure.
Data show that all the six inflammation-related CpG-SNPs genotypes including IL1B (显示 IL1B 抗体) rs16944, IL1R2 (显示 IL1R2 抗体) rs2071008, PLA2G7 rs9395208, FAM5C rs12732361, CD40 (显示 CD40 抗体) rs1800686, and CD36 (显示 CD36 抗体) rs2065666 were associated with coronary heart disease (CHD (显示 CHDH 抗体)), suggesting an important role of inflammation in the risk of CHD (显示 CHDH 抗体).
LpPLA2 is found in both LDL and HDL (显示 HSD11B1 抗体) and is distributed differently in men with T1D without any relationship to CAC (显示 CA2 抗体) score progression
In a multi-ethnic cohort without baseline cardiovascular disease, higher Lp-PLA2 mass and activity were not significantly associated with an increased risk for incident peripheral arterial disease.
The simultaneous presence of the PLA2G7 279VV genotype and persistence of overweight synergistically increases the risk for hypertension.
Lp-PLA2 augments the inflammatory response after MI and antagonizes healing by disrupting the balance between inflammation and repair.
Plg (显示 PLG 抗体) from mouse plasma contains oxPtdPC adducts that are not affected by the action of Lp-PLA(2), suggesting that linkage to Plg (显示 PLG 抗体) protects oxPtdPCs from metabolism during their transport in the plasma.
SAA (显示 SAA1 抗体) up-regulates Lp-PLA2 production significantly via a FPRL1 (显示 FPR2 抗体)/MAPKs./PPAR-gamma (显示 PPARG 抗体) signaling pathway.
Deletion of Pla2g7 decreases small intestinal polyp and colon tumor incidence in ApcMin/+ mice.
Macrophage VLDL receptor (显示 VLDLR 抗体) promotes PAFAH (显示 PAFAH1B1 抗体) secretion in mother's milk and suppresses systemic inflammation in nursing neonates.
Pla2g7 and Tnfrsf21 (显示 TNFRSF21 抗体) have been identified as genetic susceptibility to influenza genes in mice.
Pla2g7 plays a crucial physiological role in smooth muscle cell differentiation from stem cells, and interactions between Nrf3 (显示 NFE2L3 抗体) and Pla2g7 are essential.
The effects of a specific lp-PLA2 inhibitor on atherosclerosis in ApoE (显示 APOE 抗体)-deficient mice and its associated mechanisms, are reported.
Studies designed to evaluate the ability of precursor forms of PAF-AH to mature to fully active proteins indicated that the N-terminal end of human and mouse PAF-AH played important and opposite roles in this process.
PAF acetylhydrolase activity in the uterus in early pregnancy was not produced locally but probably resulted from the influx of the plasma form of the enzyme
The results demonstrated that Lp-PLA2 is associated with triglycerides which may be helpful for understanding the relationship of this protein with cardiovascular disease.
Effect of splenectomy and autologous spleen transplantation on the serum PAF-AH activity and acute-phase response in a porcine model are reported.
observations support a proatherogenic role for Lp-PLA2
Variable gene expression (eg, matrix metalloproteinase-9 (显示 MMP9 抗体), CCL2 (显示 CCL2 抗体) and Lp-PLA(2) mRNAs), both in regard to the arterial bed and duration of disease, was associated with variable plaque development and progression.
The significant correlation between PLA2G7 RNA expression in plaque macrophages and plasma PAFAH activity suggests that the latter is a consequence, rather than a cause of macrophage accumulation.
The protein encoded by this gene is a secreted enzyme that catalyzes the degradation of platelet-activating factor to biologically inactive products. Defects in this gene are a cause of platelet-activating factor acetylhydrolase deficiency. Two transcript variants encoding the same protein have been found for this gene.
, 2-acetyl-1-alkylglycerophosphocholine esterase
, LDL-associated phospholipase A2
, PAF 2-acylhydrolase
, PAF acetylhydrolase
, group-VIIA phospholipase A2
, lipoprotein-associated phospholipase A2
, platelet-activating factor acetylhydrolase
, group VII phospholipase A2
, plasma PAF acetylhydrolase