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Rat (Rattus) RHOA ELISA Kit for Sandwich ELISA - ABIN434358
Du, Wang, Wang: Role of RhoA/MERK1/ERK1/2/iNOS signaling in ocular ischemic syndrome. in Graefe's archive for clinical and experimental ophthalmology 2016
Mgc's GAP activity down-regulates the active populations of RhoA and Rac1 at localized regions of epithelial cells and is necessary for successful cytokinesis and cell-cell junction structure
Data show that shortly after anaphase onset oocytes and embryonic cells exhibit cortical waves of Rho activity and F-actin polymerization.
CASZ1 (显示 CASZ1 ELISA试剂盒)/Egfl7 (显示 EGFL7 ELISA试剂盒)/RhoA pathway is necessary for promoting endothelial cell behaviors associated with proper vascular assembly.
RhoA can be considered a component of the intracellular pattern formation system.
Kazrin (显示 KAZ ELISA试剂盒) interacts with ARVCF (显示 ARVCF ELISA试剂盒)-catenin, spectrin and p190B (显示 ARHGAP5 ELISA试剂盒) RhoGAP (显示 ARHGAP1 ELISA试剂盒), and modulates RhoA activity.
Morphogenesis of the primitive gut tube is generated by Rho/ROCK/myosin II-mediated endoderm rearrangements.
TET2 (显示 TET2 ELISA试剂盒) and RhoA mutations cooperatively disrupt T cell homeostasis
Genetic variant in RhoA gene is associated with progression of prostate cancer.
Downregulation of Cul3 (显示 CUL3 ELISA试剂盒) led to a marked increase in RhoA protein expression after 6 days of adipocytes differentiation, suggesting that Cul3 (显示 CUL3 ELISA试剂盒) is involved in the regulation of RhoA stability.
High RHOA expression is associated with colon cancer cell migration.
P311 could accelerate skin wound reepithelialization by promoting the migration of Epidermal Stem Cell through RhoA and Rac1 activation.
Findings indicate a tumor suppressive role for G protein subunit alpha 13 (Galpha13) and rhoA GTP-binding protein (RhoA) in Burkitt's lymphoma and diffuse large B-cell lymphoma (DLBCL).
RhoA/ROCK and Raf-1 (显示 RAF1 ELISA试剂盒)/CK2 (显示 CSNK2A1 ELISA试剂盒) pathway are responsible for TNF-alpha (显示 TNF ELISA试剂盒)-mediated endothelial cytotoxicity via regulation of the vimentin (显示 VIM ELISA试剂盒) cytoskeleton.
Rac (显示 AKT1 ELISA试剂盒) is required to stimulate the remodeling of mast cells, triggering actin-mediated flattening of the cell periphery to create an active degranulation zone, whereas RhoA controls the streaming of highly motile granules into the active zone.
the use of N1-Guanyl (显示 GUCA2A ELISA试剂盒)-1,7-diaminoheptane, a DHPS (显示 DHPS ELISA试剂盒) inhibitor, resulted in a significant decrease in tumor formation in vivo. In patients with esophageal squamous cell carcinoma (ESCC), overexpression of DHPS (显示 DHPS ELISA试剂盒) in ESCC tumors was significantly associated with worse recurrence-free survival, and correlated with distant metastasis.
Besides controlling cyclin (显示 PCNA ELISA试剂盒)/CDK (显示 CDK4 ELISA试剂盒) kinase activity, p27 (显示 PAK2 ELISA试剂盒) also regulates cytoskeletal dynamics, cell motility and cell invasion. Following processing by caspases, p27 (显示 PAK2 ELISA试剂盒) fails to bind to RhoA and to inhibit its activation, and thereby abolishes the ability of p27 (显示 PAK2 ELISA试剂盒) to stimulate cell migration and invasion
Pseudorabies virus US3 expression led to RhoA phosphorylation at serine 188 to induce actin rearrangements.
Data indicate that TNF-alpha (显示 TNF ELISA试剂盒) stimulates Rac (显示 AKT1 ELISA试剂盒), ADAM17/TACE (显示 ADAM17 ELISA试剂盒), and RhoA through the guanine nucleotide exchange factor (显示 ARHGEF12 ELISA试剂盒) (GEF)-H1 (显示 ARHGEF2 ELISA试剂盒).
Contractile pulmonary arterial myocytes exhibit marked Rho-dependent actin polymerization in hypoxia, with increased active RhoA and LIMK (显示 LIMK1 ELISA试剂盒) phosphorylation.
Results suggest that Rac1 and RhoA are regulated by TGFbeta1 (显示 TGFB1 ELISA试剂盒) in the process of endothelial tube formation in collagen I gels.
The concentration of RhoA mRNA and activated RhoA enzyme were greater in urothelium than in detrusor. Rho kinase (显示 ROCK1 ELISA试剂盒) inhibitor Y-27632 showed a stronger inhibitory effect in detrusor with intact urothelium.
Thrombin (显示 F2 ELISA试剂盒) stimulates swine smooth muscle cell differentiation from peripheral blood mononuclear cells via protease-activated receptor-1 (显示 F2R ELISA试剂盒), RhoA, and myocardin (显示 MYOCD ELISA试剂盒).
Rho attenuates the interaction between Amot (显示 AMOT ELISA试剂盒) and Nf2 (显示 NF2 ELISA试剂盒) by binding to the coiled-coil domain of Amot (显示 AMOT ELISA试剂盒).
we uncovered cell state plasticity and adhesion dynamics regulated by Ror2 (显示 ROR2 ELISA试剂盒), which influenced Ras Homology Family Member A (显示 CXCL14 ELISA试剂盒) (RhoA) and Rho-Associated Coiled-Coil Kinase 1 (ROCK1 (显示 ROCK1 ELISA试剂盒)) activity downstream of Dishevelled-2 (Dvl2 (显示 DVL2 ELISA试剂盒)).
Active Rho-kinase (显示 ROCK2 ELISA试剂盒) diffuses to growing other immature neurites and inhibits their outgrowth to ensure single axon formation.
Data indicate that oxidative stress in diabetes causes a decrease in miR (显示 MLXIP ELISA试剂盒)-133a expression leading to an increase in RhoA/Rho kinase (显示 ROCK2 ELISA试剂盒) pathway and muscle contraction.
Impaired denitrosylation is associated with detrusor overactivity, which is linked with upregulated RhoA/Rho-kinase (显示 ROCK2 ELISA试剂盒) signalling
The in vivo function of RhoA in corpus luteum (CL) luteal cell cytoskeleton integrity, cholesterol transport, StAR expression, and progesterone synthesis, and a positive feedback on StAR expression in CL by progesterone signaling.
Reveal novel intracellular signaling mechanisms involving RhoA/STAT3 (显示 STAT3 ELISA试剂盒) underlying the contribution of reactive astrocyte dynamics to glial scar formation.
We show that RhoA mRNA levels were significantly higher compared with the RhoB mRNA levels in ESCs (显示 NR2E3 ELISA试剂盒) as well in various cancer cell lines and this difference could be accounted for by differences in the activities of the corresponding promoters.
RhoA and membrane fluidity mediates the spatially polarized Src (显示 SRC ELISA试剂盒)/FAK (显示 PTK2 ELISA试剂盒) activation in response to shear stress.
the Lbc (显示 AKAP13 ELISA试剂盒)/alpha-catulin (显示 CTNNAL1 ELISA试剂盒) axis participates in 5-HT (显示 DDC ELISA试剂盒)-induced PASMC mitogenesis and RhoA/ROCK signaling, and may be an interventional target in diseases involving vascular smooth muscle remodeling.
The RhoA/ROCK signaling pathway is an important negative regulator of vascular calcification.
Vascular endothelial-cadherin signals through RhoA and actin cytoskeletal and affects cell-matrix adhesion
Thrombospondin has a role in inducing RhoA inactivation through FAK (显示 PTK2 ELISA试剂盒)-dependent signaling to stimulate focal adhesion disassembly
KCl directly increased Rho and ROCK activities in a concentration-dependent fashion that paralleled closely the effect of KCl on lung smooth muscle tone and [Ca(2 (显示 CA2 ELISA试剂盒)+)](i), as well as the voltage-dependent Ca(2 (显示 CA2 ELISA试剂盒)+) currents
the Rho-ROCK signal pathway contributes to VEGF-induced hyperpermeability. Myosin light-chain phosphorylation and actin stress fiber formation occur concomitantly with the increase in permeability upon VEGF stimulation.
Formation of polygonal actin network in endothelial cells is a novel rhoA associated response to hypertonic stress.
Cadherins, RhoA and Rac1, have important roles in mechanotransduction and that endothelial and smooth muscle cells use different mechanisms to respond to stretch.
Results indicate that hypergravity induces ATP release and actin reorganization via RhoA activation and FAK (显示 PTK2 ELISA试剂盒) phosphorylation, thereby activating cell proliferation and migration in bovine aortic endothelial cells.
Activating Rho could be beneficial to suppress Kras mutant-induced liver malignancies.
Regulates a signal transduction pathway linking plasma membrane receptors to the assembly of focal adhesions and actin stress fibers. Involved in a microtubule-dependent signal that is required for the myosin contractile ring formation during cell cycle cytokinesis. Plays an essential role in cleavage furrow formation. Required for the apical junction formation of keratinocyte cell-cell adhesion. Serves as a target for the yopT cysteine peptidase from Yersinia pestis, vector of the plague, and Yersinia pseudotuberculosis, which causes gastrointestinal disorders. Stimulates PKN2 kinase activity. May be an activator of PLCE1. Activated by ARHGEF2, which promotes the exchange of GDP for GTP. Essential for the SPATA13-mediated regulation of cell migration and adhesion assembly and disassembly. The MEMO1-RHOA- DIAPH1 signaling pathway plays an important role in ERBB2- dependent stabilization of microtubules at the cell cortex. It controls the localization of APC and CLASP2 to the cell membrane, via the regulation of GSK3B activity. In turn, membrane-bound APC allows the localization of the MACF1 to the cell membrane, which is required for microtubule capture and stabilization.
Aplysia ras-related homolog 12
, oncogene RHO H12
, ras homolog gene family, member A
, rho cDNA clone 12
, small GTP binding protein RhoA
, transforming protein RhoA
, Ras family member A
, Rho family GTPase
, aplysia ras-related homolog A
, aplysia ras-related homolog A1
, aplysia ras-related homolog A2
, ras homolog A1
, ras homolog A2
, ras homolog gene family, member A1
, ras homolog gene family, member A2
, plysia ras-related homolog A2
, rho1 GTP-binding protein
, RhoA GTPase
, Rho A