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Mouse (Murine) IL5 Protein expressed in Escherichia coli (E. coli) - ABIN1305123
Kinashi, Harada, Severinson, Tanabe, Sideras, Konishi, Azuma, Tominaga, Bergstedt-Lindqvist, Takahashi: Cloning of complementary DNA encoding T-cell replacing factor and identity with B-cell growth factor II. in Nature 1986
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Human IL5 Protein expressed in Escherichia coli (E. coli) - ABIN1305122
Kitamura, Takaku, Miyajima: IL-1 up-regulates the expression of cytokine receptors on a factor-dependent human hemopoietic cell line, TF-1. in International immunology 1991
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Human IL5 Protein expressed in Escherichia coli (E. coli) - ABIN1112346
Xin, Mizukami, Urabe, Toda, Shinoda, Yoshida, Oomura, Kojima, Ichino, Klinman, Ozawa, Okuda: Induction of robust immune responses against human immunodeficiency virus is supported by the inherent tropism of adeno-associated virus type 5 for dendritic cells. in Journal of virology 2006
Obesity alters the lung neutrophil infiltration to enhance breast cancer metastasis through IL5 and GM-CSF (显示 CSF2 蛋白).
these studies establish a basal defect in eosinophilopoiesis in IL-33 (显示 IL33 蛋白)- and ST2 (显示 SULT2A1 蛋白)-deficient mice and a mechanism whereby IL-33 (显示 IL33 蛋白) supports eosinophils by driving both systemic IL-5 production and the expansion of IL-5Ralpha-expressing precursor cells
Increased production of IL-5 from Peyer's patch cells and the restored Th1-type immune response might cause the production of abnormal IgA and might induce the glomerular deposition of IgA in IGA nephropathy.
E. granulosus infection remarkably reduces the severity of OVA-induced airway inflammation likely through enhancing IL-10 (显示 IL10 蛋白) and down-regulation of IL-5 and IL-17A (显示 IL17A 蛋白).
selective proliferation of IgM (显示 CD40LG 蛋白) rheumatoid factor-secreting B-1a cells is induced by co-stimulation by the specific pathogen antigen and IL-5 in the development of MC in Capillaria hepatica-infected mice
IL5, a cytokine involved in allergic and infectious diseases, facilitates metastatic colonization through recruitment of sentinel eosinophils and regulation of other inflammatory/immune cells in the microenvironment of the distal lung.
In mice, low-dose IL-2 (显示 IL2 蛋白)-anti-IL-2 (显示 IL2 蛋白) antibody complexes drove group 2 innate lymphoid cells (ILC2) to produce IL-5 and proliferate.
Data (including data from knockout mice) suggest that up-regulation of IL5 production in lungs during influenza virus infection is due to infiltration of natural killer cells and alveolar macrophages into infected lung tissue.
A decrease in the levels of IL-5, IL-9 (显示 IL9 蛋白), and IL-6R in the BALF.
eosinophils express CAR4 (显示 CA4 蛋白) following IL-5 or allergen exposure, and that CAR4 (显示 CA4 蛋白) is involved in regulating the lung transcriptome associated with allergic airway inflammation
simvastatin was demonstrated to inhibit IL-5-induced CCR3 (显示 CCR3 蛋白) expression and chemotaxis of eosinophils mediated via the mevalonate pathway.
Interleukin-5/-targeted treatments offer promises to patients with eosinophilic respiratory disorders; review
Data indicate that interleukin 5 (IL-5)-associated single nucleotide polymorphisms (SNPs) were not associated with carotid intima-media thickness.
Post-liver transplant patients with food allergy showed a unique cytokine profile in response to various stimuli, with extremely elevated IL-5, low IL-10 (显示 IL10 蛋白) secretion.
high levels of IL-33 (显示 IL33 蛋白) and a high IL-33 (显示 IL33 蛋白)/soluble ST2 (显示 SULT2A1 蛋白) ratio correlates with elevated levels of IFN-gamma (显示 IFNG 蛋白), TNF-alpha (显示 TNF 蛋白) and IL-17alpha as well as IL-5, demonstrating that IL-33 (显示 IL33 蛋白) has pleiotropic effects.
Production of IL-5 in response to both extract and the Bet v1-derived peptide mix strongly suggested that T cells were a major source of IL-5.
TRPV1 (显示 TRPV1 蛋白) expression level, IL-4 (显示 IL4 蛋白) level, and rs4790522 site mutation are the main risk factors inducing bronchial asthma in children
It has been shown in this study that the level of IL-5, one of the markers of eosinophilic inflammation, is higher in EBC of children with atopic asthma than in those with nonatopic asthma.
IL-5 may be part of protective mechanisms operating in early atherosclerosis, at least in women
This study showed that pronounced lung eosinophilia in horses can be transient, abate without specific treatment, and in this instance, lack correlation to upregulation of expression of either IL-4 (显示 IL4 蛋白) or IL-5.
This hormone is responsible for the regulation and release of thyroid-stimulating hormone, as well as prolactin. Deficiency of this hormone has been associated with hypothalamic hypothyroidism.
, B-cell growth factor II
, T-cell replacing factor
, cytotoxic T-lymphocyte inducer
, eosinophil differentiation factor
, Interleukin 5 (colony-stimulating factor eosinophil)
, Interleukin 5 (colony-stimulating factor, eosinophil)
, B-cell differentiation factor I
, colony-stimulating factor, eosinophil
, TSH-releasing factor
, pro-thyrotropin-releasing hormone
, thyrotropin releasing hormone
, thyrotropin-releasing factor