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抗Human Angiotensin II/III 抗体:
抗Rat (Rattus) Angiotensin II/III 抗体:
抗Mouse (Murine) Angiotensin II/III 抗体:
Human Monoclonal Angiotensin II/III Primary Antibody for IHC (p), IHC - ABIN253542
Ni, Ma, Wang, Liu, Zhang, Lv, Ni, Chen, Ruan, Liu: Activation of renin-angiotensin system is involved in dyslipidemia-mediated renal injuries in apolipoprotein E knockout mice and HK-2 cells. in Lipids in health and disease 2014
Show all 7 Pubmed References
Angiotensin II initiates hepatocyte epithelial-mesenchymal transition by activating the NOX-derived H2O2-mediated NLRP3 (显示 NLRP3 抗体) inflammasome/IL-1ss/Smad (显示 SMAD1 抗体) circuit.
present study has demonstrated, for the first time, that high glucose augments AGT (显示 AGXT 抗体) in human RPTCs through HNF-5, which provides a potential therapeutic target for diabetic nephropathy
AngII-dependent phosphorylation of LCP1 (显示 LCP1 抗体) in cultured podocytes was mediated by the kinases ERK (显示 EPHB2 抗体), p90 (显示 CANX 抗体) ribosomal S6 kinase (显示 RPS6KB1 抗体), PKA, or PKC (显示 PRRT2 抗体). LCP1 (显示 LCP1 抗体) phosphorylation increased filopodia formation.
Autosomal dominant polycystic kidney disease (ADPKD), uniquely increases urinary angiotensinogen (显示 AGT 抗体) and renin (显示 REN 抗体) excretion despite their circulating levels being comparable with those in non-ADPKD chronic kidney disease.
Quaternary interactions and supercoiling modulate the cooperative DNA binding of AGT (显示 AGXT 抗体).
results show that SNPs in the Hap (显示 SAFB 抗体)-I of the hAGT gene promote high-fat diet-induced binding of transcription factors GR, CEBP-beta (显示 CEBPB 抗体) and STAT3 (显示 STAT3 抗体), which lead to elevated expression of the hAGT gene in hepatic and adipose tissues
Angiotensinogen (显示 AGT 抗体) import and subsequent trafficking to the mitochondria occurs in proximal kidney tubules.
Transgenic mice expressing human AGT (显示 AGXT 抗体) in the subfornical organ AGT (显示 AGXT 抗体) and possibly ANG I/ANG II (显示 AGT 抗体) into the cerebral ventricles.
AngII could induce pulmonary injury by triggering endothelial barrier injury, and such process may be related to the dephosphorylation of Y685-VE-cadherin (显示 CDH5 抗体) and the endothelial skeletal rearrangement
Renin-angiotensin system transgenic mouse model suggests that renal injury in preeclampsia may be mediated through local VEGF.
The protein encoded by this gene, pre-angiotensinogen or angiotensinogen precursor, is expressed in the liver and is cleaved by the enzyme renin in response to lowered blood pressure. The resulting product, angiotensin I, is then cleaved by angiotensin converting enzyme (ACE) to generate the physiologically active enzyme angiotensin II. The protein is involved in maintaining blood pressure and in the pathogenesis of essential hypertension and preeclampsia. Mutations in this gene are associated with susceptibility to essential hypertension, and can cause renal tubular dysgenesis, a severe disorder of renal tubular development. Defects in this gene have also been associated with non-familial structural atrial fibrillation, and inflammatory bowel disease.
alpha-1 antiproteinase, antitrypsin
, angiotensin I
, angiotensin II
, serine (or cysteine) proteinase inhibitor
, serpin A8