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Rat (Rattus) Angiotensin II ELISA Kit for Competition ELISA - ABIN416068
Liu, Hu, Wang, Zhang, Ma, Feng, Wang, Wang, Dong, Gao, Zhang, Zhang: Angiotensin-converting enzyme (ACE) 2 overexpression ameliorates glomerular injury in a rat model of diabetic nephropathy: a comparison with ACE inhibition. in Molecular medicine (Cambridge, Mass.) 2011
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Human Angiotensin II ELISA Kit for Competition ELISA - ABIN414350
Zhang, Long, Sun, Wang, Li, Wu, Guo, Li, Niu, Li, Liu, Mei et al.: Evidence for the complementary and synergistic effects of the three-alkaloid combination regimen containing berberine, hypaconitine and skimmianine on the ulcerative colitis rats induced by ... in European journal of pharmacology 2010
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Mouse (Murine) Angiotensin II ELISA Kit for Competition ELISA - ABIN415355
Wang, Liu, Ye, Zhang, Tang, Chen, Zhang, Lou: Mesangial medium with IgA1 from IgA nephropathy inhibits nephrin expression in mouse podocytes. in European journal of clinical investigation 2009
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Rat (Rattus) Angiotensin II ELISA Kit for Sandwich ELISA - ABIN368044
Simic, Hermann, Shaw, Bigler, Stalder, Dörries, Besler, Lüscher, Ruschitzka: Torcetrapib impairs endothelial function in hypertension. in European heart journal 2011
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Mouse (Murine) Angiotensin II ELISA Kit for Sandwich ELISA - ABIN366511
Than, Leow, Chen: Control of adipogenesis by the autocrine interplays between angiotensin 1-7/Mas receptor and angiotensin II/AT1 receptor signaling pathways. in The Journal of biological chemistry 2013
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Human Angiotensin II ELISA Kit for Sandwich ELISA - ABIN366510
Mao, Cao, Lan, He, Chen, Wang, Hu, Lv, Wang, Yan: Therapeutic effect of forest bathing on human hypertension in the elderly. in Journal of cardiology 2012
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Autosomal dominant polycystic kidney disease (ADPKD), uniquely increases urinary angiotensinogen (显示 AGT ELISA试剂盒) and renin (显示 REN ELISA试剂盒) excretion despite their circulating levels being comparable with those in non-ADPKD chronic kidney disease.
Quaternary interactions and supercoiling modulate the cooperative DNA binding of AGT (显示 AGXT ELISA试剂盒).
results show that SNPs in the Hap (显示 SAFB ELISA试剂盒)-I of the hAGT gene promote high-fat diet-induced binding of transcription factors GR, CEBP-beta (显示 CEBPB ELISA试剂盒) and STAT3 (显示 STAT3 ELISA试剂盒), which lead to elevated expression of the hAGT gene in hepatic and adipose tissues
Angiotensinogen (显示 AGT ELISA试剂盒) import and subsequent trafficking to the mitochondria occurs in proximal kidney tubules.
Transgenic mice expressing human AGT (显示 AGXT ELISA试剂盒) in the subfornical organ AGT (显示 AGXT ELISA试剂盒) and possibly ANG I/ANG II (显示 AGT ELISA试剂盒) into the cerebral ventricles.
AngII could induce pulmonary injury by triggering endothelial barrier injury, and such process may be related to the dephosphorylation of Y685-VE-cadherin (显示 CDH5 ELISA试剂盒) and the endothelial skeletal rearrangement
Renin-angiotensin system transgenic mouse model suggests that renal injury in preeclampsia may be mediated through local VEGF.
endoplasmic reticulum stress induces apoptosis in human alveolar epithelial cells through mediation of unfolded protein response pathways, which in turn regulate the autocrine ANGII/ANG1 (显示 ANGPT1 ELISA试剂盒)-7 system.
Angiotensin II stimulates PYY secretion, in turn inhibiting epithelial anion fluxes, thereby reducing net fluid secretion into the colonic lumen.
NOXs had two time-dependent reactions in response to Ang II (显示 AGT ELISA试剂盒) stimulation via MAPK (显示 MAPK1 ELISA试剂盒) pathwa
this study demonstrated that Ang II (显示 AGT ELISA试剂盒) could increase TRPC6 (显示 TRPC6 ELISA试剂盒) induced Ca(2 (显示 CA2 ELISA试剂盒)+) influx and enhance autophagy through increasing reactive oxygen species levels in podocytes, and autophagy could protect Ang II (显示 AGT ELISA试剂盒)-treated podocytes.
These results implied that AngII could effectively induce EpiCs to differentiate into vascular smooth muscle-like cells through the AT1 receptor (显示 AGTRAP ELISA试剂盒).
Results suggest the involvement of angiotensin II (Ang II), through its angiotensin type-1 receptor (AT1R (显示 AGTRAP ELISA试剂盒)) in the inflammation induced by Aah (显示 ASPH ELISA试剂盒) venom, in the heart and the aorta.
expression of spinal ACE (显示 ACE ELISA试剂盒) increased in streptozotocin-induced diabetic mice, which in turn led to an increase in Ang II (显示 AGT ELISA试剂盒) levels and tactile allodynia.
the beneficial actions of insulin (显示 INS ELISA试剂盒) in diabetic nephropathy appear to be mediated, in part, by suppressing renal Nrf2 (显示 NFE2L2 ELISA试剂盒) and Agt (显示 AGXT ELISA试剂盒) gene transcription and preventing Nrf2 (显示 NFE2L2 ELISA试剂盒) stimulation of Agt (显示 AGXT ELISA试剂盒) expression via hnRNP F (显示 HNRNPF ELISA试剂盒)/K.
Angiotensinogen (显示 AGT ELISA试剂盒)-mediated downregulation of aquaporin 1 (显示 AQP1 ELISA试剂盒) and Nrf2 (显示 NFE2L2 ELISA试剂盒) signalling may play an important role in intrarenal renin (显示 REN ELISA试剂盒)-angiotensin system-induced hypertension and kidney injury.
This study suggests that deletion of AT2R decreases the expression of the beneficial ACE2/Ang-(1-7)/MasR.
an inverse correlation was found between Ang-(1 (显示 ANGPT1 ELISA试剂盒)-7) level and tau hyperphosphorylation, a pathological hallmark of Alzheimer's disease, in cerebral cortex and hippocampus of SAMP8 mice.
The inhibition of pathological autophagy in the heart in response to chronic Ang II (显示 AGT ELISA试剂盒) by Interleukin-10 (显示 IL10 ELISA试剂盒), and its implications, has been described.
The protein encoded by this gene, pre-angiotensinogen or angiotensinogen precursor, is expressed in the liver and is cleaved by the enzyme renin in response to lowered blood pressure. The resulting product, angiotensin I, is then cleaved by angiotensin converting enzyme (ACE) to generate the physiologically active enzyme angiotensin II. The protein is involved in maintaining blood pressure and in the pathogenesis of essential hypertension and preeclampsia. Mutations in this gene are associated with susceptibility to essential hypertension, and can cause renal tubular dysgenesis, a severe disorder of renal tubular development. Defects in this gene have also been associated with non-familial structural atrial fibrillation, and inflammatory bowel disease.
alpha-1 antiproteinase, antitrypsin
, angiotensin I
, angiotensin II
, serine (or cysteine) proteinase inhibitor
, serpin A8
, angiotensinogen (PAT)
, angiotensin ll