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抗Human SOX9 抗体:
抗Rat (Rattus) SOX9 抗体:
抗Mouse (Murine) SOX9 抗体:
Human Polyclonal SOX9 Primary Antibody for ICC, IF - ABIN4355424
Vestentoft, Jelnes, Hopkinson, Vainer, Møllgård, Quistorff, Bisgaard: Three-dimensional reconstructions of intrahepatic bile duct tubulogenesis in human liver. in BMC developmental biology 2011
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Human Polyclonal SOX9 Primary Antibody for IF (p), IHC (p) - ABIN754963
Papaioannou, Mirzamohammadi, Lisse, Nishimori, Wein, Kobayashi: MicroRNA-140 provides robustness to the regulation of hypertrophic chondrocyte differentiation by the PTHrP-HDAC4 pathway. in Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research 2014
Show all 6 Pubmed References
Human Monoclonal SOX9 Primary Antibody for IF, IHC (p) - ABIN520382
Hodgin, Borczuk, Nasr, Markowitz, Nair, Martini, Eichinger, Vining, Berthier, Kretzler, DAgati: A molecular profile of focal segmental glomerulosclerosis from formalin-fixed, paraffin-embedded tissue. in The American journal of pathology 2010
Show all 4 Pubmed References
Human Monoclonal SOX9 Primary Antibody for ICC, ELISA - ABIN969568
Dupasquier, Abdel-Samad, Glazer, Bastide, Jay, Joubert, Cavaillès, Blache, Quittau-Prévostel: A new mechanism of SOX9 action to regulate PKCalpha expression in the intestine epithelium. in Journal of cell science 2009
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Zebrafish Polyclonal SOX9 Primary Antibody for ELISA - ABIN408740
Yan, Willoughby, Liu, Crump, Wilson, Miller, Singer, Kimmel, Westerfield, Postlethwait: A pair of Sox: distinct and overlapping functions of zebrafish sox9 co-orthologs in craniofacial and pectoral fin development. in Development (Cambridge, England) 2005
Human Polyclonal SOX9 Primary Antibody for IF, IHC (p) - ABIN272082
Zhang, Ding, Nie, Li-Ling, Zhang, Zhang, Chen, Li, Ding: Association of hsa-miR‑145 overexpression in human testicular cells with male infertility. in Molecular medicine reports 2015
Cow (Bovine) Polyclonal SOX9 Primary Antibody for FACS, WB - ABIN2780338
Vidal, Ortonne, Schedl: SOX9 expression is a general marker of basal cell carcinoma and adnexal-related neoplasms. in Journal of cutaneous pathology 2008
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Human Monoclonal SOX9 Primary Antibody for ELISA, WB - ABIN562960
Wainwright, Svingen, Ng, Wicking, Koopman: Primary cilia function regulates the length of the embryonic trunk axis and urogenital field in mice. in Developmental biology 2014
Amphibian Polyclonal SOX9 Primary Antibody for DB, IHC - ABIN4355422
Sharma, Knowell, Chinaranagari, Komaragiri, Nagappan, Patel, Havrda, Chaudhary: Id4 deficiency attenuates prostate development and promotes PIN-like lesions by regulating androgen receptor activity and expression of NKX3.1 and PTEN. in Molecular cancer 2014
Cat (Feline) Polyclonal SOX9 Primary Antibody for IHC (p), SimWes - ABIN4355427
Antunes, Tsaryk, Gonçalves, Pereira, Landes, Brochhausen, Ghanaati, Barbosa, Kirkpatrick: Poly(γ-Glutamic Acid) as an Exogenous Promoter of Chondrogenic Differentiation of Human Mesenchymal Stem/Stromal Cells. in Tissue engineering. Part A 2015
Data show that the gene encoding the transcription factor SOX9 was identified by a global transcriptomic approach as an HDAC9 (显示 HDAC9 抗体) target gene.
SOX9 is a proliferation and stem cell factor (显示 KITLG 抗体) in hepatocellular carcinoma and possess widespread prognostic significance in different cancer types
Sox9 and Ngn3 (显示 NEUROG3 抗体), key transcription factors associated with pancreatic development.
Expression of bone morphogenetic protein (BMP) 4, an upstream stimulator of SOX9, was upregulated by CG.
Xenogeneic implantation of Sox9-overexpressing hUCMSCs embedded in the BMG/fibrin scaffolds promotes the formation of cartilage-like tissue without inducing evident host immune response. Therefore, Sox9-overexpressing hUCMSCs represent a promising cell candidate for cartilage tissue engineering.
KLF15 activates SOX9 expression directly. SOX9 is involved in KLF15 function during chondrogenic differentiation.
Tomo-Seq Identifies SOX9 as a Key Regulator of Cardiac Fibrosis During Ischemic Injury
High SOX9 expression is associated with glioblastoma.
These findings suggest that SOX9 may play an important role in tumor progression of Renal Cell Carcinoma (显示 MOK 抗体) and Bladder Cancer and it may be used as a biomarker of this malignancy.
Loss of DDRGK1 decreases SOX9 expression and causes a human skeletal dysplasia.
data presented here demonstrate that the 18 bp indel in the porcine SOX9 5'-UTR (显示 UTS2R 抗体) is of functional importance and may therefore indeed be a causative variation in SOX9 associated traits
HIF-1alpha (显示 HIF1A 抗体) activates Sox9 expression and enhances Sox9-mediated transcriptional activity.
Hypomethylation in the Sox9 promoter is correlated to increased Sox9 expression in db/db (显示 LEPR 抗体) IESCs. Although there is increased expression of Sox9 in db/db (显示 LEPR 抗体) IESCs, the loss of Sox9 transcriptional activation in specific repressors of the Wnt (显示 WNT2 抗体) signaling pathway might result in abnormalities in this pathway.
Concluding, this study shows that, in addition to its crucial role in testis development, Sox9, together with Sox8 (显示 SOX8 抗体) and coordinately with Dmrt1 (显示 DMRT1 抗体), also controls adult testis maintenance.
SOX9 regulation of ETV5 (显示 ETV5 抗体) contributes to the control of male fertility
Nuclear factors that bind to genomic regions with "Sertoli Cell Signature" could functionally interact with SOX9; TRIM28 (显示 TRIM28 抗体) is a new SOX9 partner in fetal testes.
Down-regulated expression of Sox9 and Dazl (显示 DAZL 抗体) may play important roles in MBP (显示 MBP 抗体)-induced testis injury.
Sox9 is positively regulated by mesenchymal Fgf10 (显示 FGF10 抗体), a process that requires active Erk (显示 EPHB2 抗体) signaling during salivary gland development
TSC1 (显示 TSC1 抗体)/TSC2 (显示 TSC2 抗体) complex upregulation of OPN (显示 SPP1 抗体) expression is mediated by transcription factor SOX9 in an mTOR (显示 FRAP1 抗体)-independent manner. Moreover, ablation of OPN (显示 SPP1 抗体) by deficient TSC1 (显示 TSC1 抗体)/TSC2 (显示 TSC2 抗体) complex contributed to inactivation of AKT (显示 AKT1 抗体) in TSC (显示 SLC12A3 抗体) cells
SOX9 is almost exclusively expressed by astrocytes in the adult brain except for ependymal cells and in the neurogenic regions, where SOX9 is also expressed by neural progenitor cells
hese findings indicate that chronic overexpression of Sox9 in the uterine epithelium can induce the development of endometrial hyperplastic lesions. Thus, SOX9 expression may be a factor in the formation of endometrial cancer.
The conditional ablation of Sox9 in the genital tubercle using Shh (显示 SHH 抗体)-Cre/+;Sox9(flox/flox) mice revealed no genital tubercle abnormalities, possibly due to compensation by similar Sox (显示 QSOX1 抗体) factors.
The protein encoded by this gene recognizes the sequence CCTTGAG along with other members of the HMG-box class DNA-binding proteins. It acts during chondrocyte differentiation and, with steroidogenic factor 1, regulates transcription of the anti-Muellerian hormone (AMH) gene. Deficiencies lead to the skeletal malformation syndrome campomelic dysplasia, frequently with sex reversal.
SRY (sex determining region Y)-box 9
, SRY (sex determining region Y)-box 9 (campomelic dysplasia, autosomal sex-reversal)
, transcription factor SOX-10
, SRY (sex-determining region Y)-box 9 protein
, SRY-related HMG-box, gene 9
, transcription factor SOX-9
, SRY-box containing protein 9
, SRY-box containing gene 9
, transcription factor SOX9