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抗Human MMP 9 抗体:
抗Mouse (Murine) MMP 9 抗体:
抗Rat (Rattus) MMP 9 抗体:
Human Polyclonal MMP 9 Primary Antibody for WB - ABIN3044382
Zhang, Chen, Qi, Wang, Xiao, Zhu: Inhibition of calcium-calmodulin-dependent kinase II suppresses cardiac fibroblast proliferation and extracellular matrix secretion. in Journal of cardiovascular pharmacology 2010
Show all 34 Pubmed References
Human Polyclonal MMP 9 Primary Antibody for ELISA, WB - ABIN3043582
Zhou, Wan, Chu, Song, Xing, Wu, Yin: Urotensin II contributes to the formation of lung adenocarcinoma inflammatory microenvironment through the NF-?B pathway in tumor-bearing nude mice. in Oncology letters 2012
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Mouse (Murine) Polyclonal MMP 9 Primary Antibody for IHC (p), ELISA - ABIN3043884
Jin, Jiang, Yang, Zhang, Yang, Zhang, Li, Yang, Ma: Acipimox attenuates atherosclerosis and enhances plaque stability in ApoE-deficient mice fed a palmitate-rich diet. in Biochemical and biophysical research communications 2012
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Human Polyclonal MMP 9 Primary Antibody for IF (p), IHC (p) - ABIN873171
Deng, Zhong, Gu, Shen, Guo: MiR-21 involve in ERK-mediated upregulation of MMP9 in the rat hippocampus following cerebral ischemia. in Brain research bulletin 2013
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Mammalian Monoclonal MMP 9 Primary Antibody for ISt, IHC - ABIN1304829
DeNiro, Al-Halafi, Al-Mohanna, Alsmadi, Al-Mohanna: Pleiotropic effects of YC-1 selectively inhibit pathological retinal neovascularization and promote physiological revascularization in a mouse model of oxygen-induced retinopathy. in Molecular pharmacology 2010
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Human Polyclonal MMP 9 Primary Antibody for IF (p), IHC (p) - ABIN668095
Zhao, Xu, He, Hua, Luo, Zuo: Expression of serum response factor in gastric carcinoma and its molecular mechanisms involved in the regulation of the invasion and migration of SGC-7901 cells. in Cancer biotherapy & radiopharmaceuticals 2013
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Human Polyclonal MMP 9 Primary Antibody for IHC (p), WB - ABIN390154
Behrens, Mathiak, Mangold, Kirdorf, Wellmann, Fogt, Rothe, Florin, Wernert: Stromal expression of invasion-promoting, matrix-degrading proteases MMP-1 and -9 and the Ets 1 transcription factor in HNPCC carcinomas and sporadic colorectal cancers. in International journal of cancer. Journal international du cancer 2003
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Cow (Bovine) Polyclonal MMP 9 Primary Antibody for IHC, WB - ABIN2777743
Nozell, Ma, Wilson, Shah, Benveniste: Class II major histocompatibility complex transactivator (CIITA) inhibits matrix metalloproteinase-9 gene expression. in The Journal of biological chemistry 2004
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Human Monoclonal MMP 9 Primary Antibody for ELISA, IHC - ABIN4335100
Goktolga, Cavkaytar, Altinbas, Tapisiz, Tapisiz, Erdem: Effect of the non-specific matrix metalloproteinase inhibitor Doxycycline on endometriotic implants in an experimental rat model. in Experimental and therapeutic medicine 2015
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Guinea Pig Polyclonal MMP 9 Primary Antibody for IHC, ELISA - ABIN1582307
Piña, Boutrid, Murray, Jager, Cebulla, Schefler, Ly, Alegret, Celdran, Feuer, Jockovich: Impact of tumor-associated macrophages in LH(BETA)T(AG) mice on retinal tumor progression: relation to macrophage subtype. in Investigative ophthalmology & visual science 2010
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KLF9 (显示 KLF9 抗体) could down-regulate MMP9 expression to inhibit breast cancer metastasis.
MMP-9 is one of the useful cell surface markers for the separation of angiogenic cells.
Concentrations of MMP-2 (显示 MMP2 抗体) and MMP-9 in serum in humans measured after acute stroke are potentially influenced by extraneous covariates rather than being directly associated with characteristics of the underlying stroke
There were no differences in activities of MMP-2 (显示 MMP2 抗体), proMMP-9, and MMP-9/NGAL (neutrophil gelatinase associated lipocalin (显示 LCN2 抗体)) complex (gelatin substrate) in men with detected prostate cancer, although the latter two were somewhat diminished.
Data indicate that the matrix metallopeptidases MMP-2 (显示 MMP2 抗体)/MMP (显示 MMP2 抗体)-9 ratio provided better compromise between specificity and sensitivity in distinguishing PE from normal pregnancies, than either of the two MMPs alone.
Circulating NGAL (显示 LCN2 抗体) and MMP-9/NGAL (显示 LCN2 抗体) are associated with plaque vulnerability in patients with carotid artery stenosis. Statin treatment could contribute to plaque stabilization by reducing circulating NGAL (显示 LCN2 抗体) and MMP-9/NGAL (显示 LCN2 抗体) levels.
study revealed decreased levels of VEGF (显示 VEGFA 抗体), ANGPT1 (显示 ANGPT1 抗体), and MMP-9 in the early post-transplant period as compared to the baseline (BC). ANGPT2 (显示 ANGPT2 抗体) was decreased after conditioning treatment, but tended to increase from day +7. On day +7, positive correlations between ANGPT1 (显示 ANGPT1 抗体) level as well as MMP-9 and the time to engraftment were observed.
The presence of the T-1702A polymorphism for MMP-9 increases the risk of recurrent depressive disorder development
Our studies also provide mechanistic insight into the increased level and activity of secreted MMP-9 in cortical neurons derived from FTD (显示 FTL 抗体) patient-specific iPSCs with MAPT (显示 MAPT 抗体) mutations. The increase seems to be mediated by ERK (显示 EPHB2 抗体)-pathway activation.
High MMP9 expression is associated with Bronchiolitis Obliterans Syndrome.
Optical imaging demonstrated increased MMP activity in TB lesions and MMP-9 was significantly expressed in cavitary lesions.
MMP-2 (显示 MMP2 抗体) and -9 expression were suppressed significantly by treatment with SB-3CT. The data demonstrated, for the first time, that SB-3CT strongly reduced corneal lymphangiogenesis and macrophage infiltration during inflammation.
Grooved surfaces showed time-dependent increase in soluble mediators involved in cell fusion, CCL2 (显示 CCL2 抗体) and MMP-9
A MMP-9-cleaved OPN fragment, OPN-32kDa, was responsible for inducing expansion of myeloid-derived suppressor cells, which may contribute to 3LL tumor's evasion of the immune response.
animals were submitted to the evaluation of Blood-Brain Barrier permeability and MMP-2 (显示 MMP2 抗体) and MMP-9 in striatum, hippocampus and cerebral cortex
p63alpha protein up-regulates heat shock protein 70 (显示 HSP70 抗体) expression via E2F1 transcription factor (显示 E2F1 抗体) 1 (显示 HNF1A 抗体), promoting Wasf3/Wave3 (显示 WASF3 抗体)/MMP9 signaling and bladder cancer invasion
IL-33 (显示 IL33 抗体)-induced MMP-9 expression in the mouse monocyte/macrophage line RAW264.7.
Cleavage of beta-DG still occurred when both MMP-2 (显示 MMP2 抗体) and MMP-9 were knocked out in gamma - sarcoglycan (显示 SGCG 抗体)-deficient mice. The study found that up-regulation of MMP-14 (显示 MMP14 抗体) is capable of cleaving beta-DG, and it may be involved in the pathogenesis of sarcoglycanopathy.
MMP-9's contribution to development of atherosclerotic lesions may be a direct stimulation of endothelial cells, and that PAR-1 (显示 MARK2 抗体) may serve as a receptor for MMP-9.
Results show that ablation of systemic MMP-9 initiated fatal communication between maintenance of physiological functions of MMP-9 in the bone marrow and invasive growth of pancreatic ductal adenocarcinoma via the deregulation of IL6 (显示 IL6 抗体).
Increased MMP-9 expression is associated with carotid atherosclerotic plaque.
Increased expression of MMP-9 is associated with intraplaque hemorrhage in a swine model of vulnerable carotid atherosclerosis
we demonstrated the presence of high molecular weight (HMW) complexes (130, 170, and 220 kDa) containing MMP9, TIMP1 (显示 TIMP1 抗体), and NGAL (显示 LCN2 抗体) (also MMP2 (显示 MMP2 抗体) in 220 kDa complex) without proteolytic activity.
Data demonstrate for the first time that MMP2 (显示 MMP2 抗体) and MMP9 are expressed in swine ovarian follicle both in theca and granulosa layers.
FiO2 used for resuscitation affects matrix metalloproteinases MMP-9 and MMP-2 (显示 MMP2 抗体), caspase-3 (显示 CASP3 抗体) and BDNF (显示 BDNF 抗体)
Oxygen for newborn resuscitation increases MMP-2 (显示 MMP2 抗体)/-9 activity resulting in tissue damage and influencing remodeling processes.
contribution of MMPs to the inflammatory breakdown of the blood-CSF (显示 CSF2 抗体) barrier in vitro
The levels of matrix metalloproteinase-2 (显示 MMP2 抗体) and matrix metalloproteinase-9 (MMP-9)in the corpus luteum of swine during luteolysis are reported.
Our data define pericyte interactions as a main inducer of endothelial MMP secretion and propose a new role for pericyte-endothelial cell crosstalk at the BBB (显示 ALMS1 抗体) in vitro
Variable gene expression (eg, matrix metalloproteinase-9, CCL2 (显示 CCL2 抗体) and Lp-PLA(2 (显示 Lp-PLA2 抗体)) mRNAs), both in regard to the arterial bed and duration of disease, was associated with variable plaque development and progression.
In diabetic retinopathy transcription of MMP-9 is regulated by AP-1 binding at both, proximal and distal sites of its promoter, and acetylation of c-Jun and c-Fos subunits is important in its regulation.
These data demonstrate that serum neutrophil haptoglobin (显示 HP 抗体)-MMP 9 complexes appear sooner and decline more rapidly than other acute phase proteins.
Activation of cytosolic MMP-9 and MMP-2 (显示 MMP2 抗体) was investigated in the retinal endothelial cells incubated in high glucose for 6-96 h, and correlated with their mitochondrial accumulation and mitochondrial damage.
Role of TGF-beta1 (显示 TGFB1 抗体) and TNF-alpha (显示 TNF 抗体) in IL-1beta (显示 IL1B 抗体) mediated activation of proMMP-9 in pulmonary artery smooth muscle cells: involvement of an aprotinin sensitive protease.
Decreased MMP-9 and increased TIMP-1 (显示 TIMP1 抗体) expression were found in peripheral blood cells from Mycobacterium avium subsp. paratuberculosis (Map)-infected cattle after stimulation with Map lysate and Map purified protein derivative than in control cattle.
We used a trophoblast cell line (F3) derived from bovine placentomes to examine the influence of EGF (显示 EGF 抗体) on MMP-9 and TIMP-1 (显示 TIMP1 抗体) expression by semiquantitative RT-PCR and MMP activity by zymography.
results suggest a significant role of matrix metalloproteinase-2 (显示 MMP2 抗体) and-9 in growth and development of bovine follicle
Cells constitutively produced proMMP-9 and proMMP-2, and treatment with TNFalpha (显示 TNF 抗体), hepatocyte growth factor (显示 HGF 抗体), and 12-O-tetradecanoylphorbol 13-acetate resulted in significant increase in level of proMMP-9 but not in level of proMMP-2.
MMP-2 and MMP-9 production in blastocysts attached to the endometrial cells is regulated by TNF-alpha and TNF-beta
Results suggest that MMP-2 (显示 MMP2 抗体), MMP-9, and TIMP-2 (显示 TIMP2 抗体) mRNAs are expressed in bovine placentomes during the gestational and postpartum periods and that these enzymes, in conjunction with steroidogenic enzymes, mediate fetal membrane detachment after parturition.
The findings of this study suggest that Mmp-9 is a protective molecule against infection by Listeria monocytogenes by engaging in migration of zebrafish macrophages to the site of infection via a non-proteolytic role.
elevated beta-oxidation-fuelled mitochondria-derived reactive oxygen species within epidermal cells helps guide matrix metalloproteinase-driven leukocyte recruitment.
Mmp9 regulates both acute and chronic tissue damage and plays an essential role in collagen reorganization during wound repair.
MeHg impairs tail development at least partially by activation of the tissue remodeling proteases Mmp9 and Mmp13 (显示 MMP13 抗体).
study identified mechanism by which mycobacteria induce granulomas: ESAT-6 induced MMP9 in epithelial cells neighboring infected macrophages; MMP9 enhanced recruitment of macrophages, which contributed to nascent granuloma maturation & bacterial growth
From 24h post fertilization, mmp9 expression was detected in a population of circulating white blood cells.
expression and activity of MMP-2 (显示 MMP2 抗体) and MMP-9 in the embryonic zebrafish.
The MMP-9 gene was duplicated and differentiated into two genes, one was specialized in a common ancestor of X. laevis and X. tropicalis expressed in degenerating and remodeling organs in response to thyroid hormone (显示 PTH 抗体) during metamorphosis.
MMP-9TH is responsible in the larval epithelial apoptosis through degrading ECM (显示 MMRN1 抗体) components in the basal lamina, whereas MMP-9 is involved in the removal of dying epithelial cells during amphibian intestinal remodeling
metamorphic tail and intestine RNA levels of TIMP-2 (显示 TIMP2 抗体), MT1-MMP (显示 MMP14 抗体) and Gel-A, but not MT3-MMP (显示 MMP24 抗体) or TIMP-3 (显示 TIMP3 抗体), are elevated during periods of cell death and proliferation
Expression of MMP-9 increased after cerebral aneurysm induction, peaking at week 3, leading to reduced smooth muscle cell number, damaged endothelial cells, and damage to the aneurysm wall elastic layer.
Inflammatory factors such as TNF-alpha (显示 TNF 抗体) can stimulate MMP-2 (显示 MMP2 抗体)/9 activity in corneal epithelium cells. This may be a potential manipulating mechanism of MMP expression in the pathogenesis of corneal diseases
Therefore, it was reasonable to speculate that the increased expression of VEGF (显示 VEGFA 抗体) and MMP-9 in residual hepatic tumor cells and tumor angiogenesis post-embolization would be responsible for the increased metastatic potentiality and invasiveness.
Performing minimally invasive surgical procedures in the early stages of intracerebral hemorrhage significantly decreases MMP-9.
Increased expression of MMP-9 in spinal cord follows cervical spondylotic myelopathy.
Hemoperfusion could obviously reduce oxidative stress and the expression levels of MMP-2 (显示 MMP2 抗体), MMP-9 and TIMP-1 (显示 TIMP1 抗体) in rabbits with acute paraquat poisoning.
In experimental syringomyelia, MMP-9 plays an important role in causing edema in the presyrinx state.
Tongxinluo can inhibit the expression of MMP-3 (显示 MMP3 抗体) and 9 and increase the expression of PPARgamma (显示 PPARG 抗体) in atherosclerotic rabbits.
TGF-beta (显示 TGFB1 抗体) mediated MMP-9 induction may be regulated by the NF-kappaB (显示 NFKB1 抗体), Smad3 (显示 SMAD3 抗体), and JNK (显示 MAPK8 抗体) pathways, whereas the IL-1beta (显示 IL1B 抗体) mediated induction may be regulated by the NF-kappaB (显示 NFKB1 抗体) and p38 (显示 MAPK14 抗体) pathways.
The results showed that MMP-2 (显示 MMP2 抗体), MMP-9, and StAR were significantly expressed in the granulosa and thecal cells of the ovarian atretic follicles during proestrus, and were strongly expressed in the corpus luteum during metestrus.
Proteins of the matrix metalloproteinase (MMP) family are involved in the breakdown of extracellular matrix in normal physiological processes, such as embryonic development, reproduction, and tissue remodeling, as well as in disease processes, such as arthritis and metastasis. Most MMP's are secreted as inactive proproteins which are activated when cleaved by extracellular proteinases. The enzyme encoded by this gene degrades type IV and V collagens. Studies in rhesus monkeys suggest that the enzyme is involved in IL-8-induced mobilization of hematopoietic progenitor cells from bone marrow, and murine studies suggest a role in tumor-associated tissue remodeling.
92 kDa gelatinase
, 92 kDa type IV collagenase
, macrophage gelatinase
, matrix metalloproteinase 9 (gelatinase B, 92kDa gelatinase, 92kDa type IV collagenase)
, matrix metalloproteinase-9
, type V collagenase
, 92kD gelatinase
, 92kD type IV collagenase
, 92kDa gelatinase
, 92kDa type IV collagenase
, Gel B
, collagenase type IVB
, gelatinase B
, matrix metalloproteinase 9
, 92-kDa type IV collagenase
, matrix metalloproteinase 9 (gelatinase B 92-kDa type IV collagenase)
, matrix metalloproteinase 9 (gelatinase B, 92-kDa type IV collagenase)
, type IV collagenase MMP-9
, Matrix metalloproteinase-9
, matrix metalloproteinase 9 (gelatinase B, 92kDa matrix metalloproteinase 9 (gelatinase B, 92kDa gelatinase, 92kDa type IV collagenase)
, 75 kDa gelatinase