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The Rap1 (显示 RABGEF1 ELISA试剂盒)-RIAM (显示 APBB1IP ELISA试剂盒)-talin axis of integrin activation and blood cell function
Rap1 (显示 RABGEF1 ELISA试剂盒) activation was dependent on PKA and required Src (显示 SRC ELISA试剂盒) family kinases and the Rap1 (显示 RABGEF1 ELISA试剂盒) exchanger C3G (显示 RAPGEF1 ELISA试剂盒).
RAP1 (显示 RABGEF1 ELISA试剂盒) promotes colorectal cell migration through the regulation of Vimentin (显示 VIM ELISA试剂盒) and RAP1 (显示 RABGEF1 ELISA试剂盒) may act as a potential target for the diagnosis and therapy of CRC (显示 CALR ELISA试剂盒).
Data show that isoform beta2 of the heregulin (HRGbeta2) localizes at telomeres with the telomere-associated proteins TRF2 and RAP1.
Data indicate telomere-binding protein RAP1 (显示 RABGEF1 ELISA试剂盒) as an interacting partner of isoform beta2 of the heregulin (显示 NRG1 ELISA试剂盒) (HRGbeta2).
In pro-inflammatory macrophages, Rap1 promotes cytokine production via NFkappaB activation favoring a pro-inflammatory environment which may contribute to the development and progression of atherosclerosis.
Through a combination of biochemical, biophysical and structural approaches, we unveiled a unique mode of assembly between RAP1 (显示 RABGEF1 ELISA试剂盒) and TRF2 (显示 TERF2 ELISA试剂盒)
the conservation of Rap1 reflects its role in transcriptional regulation rather than a function at telomeres.
These findings reveal Pkp3 as a coordinator of desmosome and adherens junction assembly and maturation through its functional association with Rap1.
Data show that full-length repressor activator protein 1 (显示 FOSB ELISA试剂盒) (Rap1 (显示 RABGEF1 ELISA试剂盒)) binds to full-length telomeric repeat binding factor 2 (TRF2 (显示 TERF2 ELISA试剂盒)) with high affinity and equimolar ratio.
HS1 as an important regulator of proper Rac1 and Rap1 activation and neutrophil extravasation.
Rap1 activation was dependent on PKA and required Src (显示 SRC ELISA试剂盒) family kinases and the Rap1 exchanger C3G (显示 RAPGEF1 ELISA试剂盒).
Collectively the observations uncover a requirement for Rap1 in maintenance of lens epithelial phenotype and morphogenesis.
our results indicate that Rasa3 catalytic activity controls Rap1 activation and integrin signaling during megakaryocyte differentiation in mouse.
RASA3, inhibits platelet activation and provides a link between P2Y12 and activation of the RAP1 signaling pathway.
we summarize recent developments in understanding the small G protein RAP1 and its effector RASIP1 as critical mediators of endothelial junction stabilization.
Molecular investigation revealed that deletion of RAP1 reduced upregulation of inflammatory cytokine (IL1A (显示 IL1A ELISA试剂盒)), finely regulated the expression of angiogenic factor (VEGF (显示 VEGFA ELISA试剂盒)), and antiangiogenic factor (PEDF (显示 SERPINF1 ELISA试剂盒)), following injury for better corneal recovery.
Cdk5-mediated serine-phosphorylation of C3G may control Rap1 stability and activity
Mesenchymal high-grade glioma is maintained by the ID-RAP1 axis.
Rap1-Rac1 circuits potentiate platelet activation.
The gene encodes a protein that is part of a complex involved in telomere length regulation. Pseudogenes are present on chromosomes 5 and 22.
, TERF2-interacting telomeric protein 1
, TRF2-interacting telomeric RAP1 protein
, TRF2-interacting telomeric protein 1
, dopamine receptor interacting protein 5
, dopamine receptor-interacting protein 5
, repressor/activator protein 1 homolog
, telomeric repeat-binding factor 2-interacting protein 1
, TRF2-interacting telomeric protein Rap1