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抗Human CEBPA 抗体:
抗Mouse (Murine) CEBPA 抗体:
抗Rat (Rattus) CEBPA 抗体:
Human Monoclonal CEBPA Primary Antibody for FACS, ICC - ABIN269606
Ferrari-Amorotti, Keeshan, Zattoni, Guerzoni, Iotti, Cattelani, Donato, Calabretta: Leukemogenesis induced by wild-type and STI571-resistant BCR/ABL is potently suppressed by C/EBPalpha. in Blood 2006
Show all 4 Pubmed References
Cow (Bovine) Polyclonal CEBPA Primary Antibody for IHC, WB - ABIN2787465
Singh, Trivedi, Behre: C/EBPalpha S248A mutation reduces granulocytic differentiation in human leukemic K562 cells. in Biochemical and biophysical research communications 2008
Show all 3 Pubmed References
Human Monoclonal CEBPA Primary Antibody for ICC, FACS - ABIN969048
Jin, Zhang, Yan, Liu, Wang, Ge, Zhai: C/EBPalpha regulates SIRT1 expression during adipogenesis. in Cell research 2010
Human Monoclonal CEBPA Primary Antibody for FACS, ELISA - ABIN969507
Geest, Buitenhuis, Vellenga, Coffer: Ectopic expression of C/EBPalpha and ID1 is sufficient to restore defective neutrophil development in low-risk myelodysplasia. in Haematologica 2009
Binding of C/EBPalpha was associated with increased deacetylation near the transcription start site (TSS (显示 RPL38 抗体)) of the PLK1 (显示 PLK1 抗体) promoter.
a MEF2C and CEBPA correlation in CML disease progression
CEBPA gene expression is significantly associated with long-term changes in Blood Pressure, providing a link between gene expression and Blood Pressure.
We provide evidence that CCAAT/enhancer-binding protein alpha directly binds the miR (显示 MLXIP 抗体)-203 gene within its hairpin region and thereby induces miR (显示 MLXIP 抗体)-203 transcription
High CEBP expression is associated with glioblastomas.
identified high frequencies of mutations in CEBPA (32.7%), GATA2 (显示 GATA2 抗体) (22.4%), NPM1 (显示 NPM1 抗体) (15.5%), SETBP1 (显示 SETBP1 抗体) (12.1%) and U2AF1 (显示 U2AF1 抗体)
our data show that excess p30 (显示 CENPV 抗体) cooperated with TRIB2 (显示 TRIB2 抗体) only in the presence of p42 (显示 EPB42 抗体) to accelerate acute myeloid leukaemia (AML (显示 RUNX1 抗体)), and the direct interaction and degradation of C/EBPa p42 (显示 EPB42 抗体) is required for TRIB2 (显示 TRIB2 抗体)-mediated AML (显示 RUNX1 抗体).
A single +42-kb enhancer is essential for CEBPA expression in myeloid cells only.
Co-occurrence of mutations in CSF3R (显示 CSF3R 抗体) and CEBPA in a well-defined acute myeloid leukemia (显示 BCL11A 抗体) subset, which uniformly responds to JAK (显示 JAK3 抗体) inhibitors; this paves the way to personalized clinical trials for this disease.
we established a reliable and straightforward screening method, based simply on the multidimensional analysis of widely available phenotypic parameters, suitable for large-scale detection of CEBPA-dm status and potentially able to overcome technical issues related to molecular methods.
Functional characterization of C/EBPa and C/EBPb (显示 CEBPB 抗体) proteomes suggests they can regulate novel pathways.
results indicate that JMJD2B (显示 KDM4B 抗体) regulates PPARgamma (显示 PPARG 抗体) and C/EBPalpha during adipogenesis
these data indicate that CycC (显示 CCNC 抗体) activates adipogenesis in part by stimulating the transcriptional activity of C/EBPalpha.
Taken together, these findings demonstrate that artesunate inhibits adipogenesis in 3T3-L1 preadipoytes through the reduced expression and/or phosphorylation levels of C/EBP-alpha, PPAR-gamma (显示 PPARG 抗体), FAS (显示 FAS 抗体), perilipin A (显示 PLIN1 抗体), and STAT-3 (显示 STAT3 抗体).
we show that SIX1 (显示 SIX1 抗体) binds to adipogenic and brown marker genes and interacts with C/EBPa, C/EBPb (显示 CEBPB 抗体) and EBF2 (显示 EBF2 抗体), suggesting their functional cooperation during adipogenesis.
these results indicate that C/EBPalpha functions throughout osteoclastogenesis as well as in Osteoclast function. This study provides additional understanding of the roles of C/EBPalpha in Osteoclast biology.
the DNA sequences to which EVI1 (显示 MECOM 抗体) binds at +35 and +37 kb and show that mutation of one of these releases Cebpa from EVI1 (显示 MECOM 抗体)-induced suppression.
The efficient repression of E2F (显示 E2F1 抗体) dependent S-phase genes and the activation of differentiation genes reside in the balanced DNA binding capacity of C/EBP alpha.
Cebpa enhancers and silencers in a transcriptional model have roles in hematopoietic lineage specification
C/ebpalpha plays a role in liver growth regulation via the p53 (显示 TP53 抗体) pathway.
Dnmt1 (显示 DNMT1 抗体) is required for hematopoietic stem and progenitor cells maintenance via cebpa regulation during definitive hematopoiesis in zebrafish
Data suggest that upregulation of 10-formyltetrahydrofolate dehydrogenase (FDH (显示 ALDH1L1 抗体)) involving CEBPalpha helps relieve embryonic oxidative stress induced (显示 SQSTM1 抗体) by ethanol exposure.
Bmi1 (显示 BMI1 抗体) acts immediately downstream of CCAAT enhancer binding protein-alpha to regulate the survival and self-renewal of hematopoietic stem cells and contribute to the erythropoietic dysplasia.
Results provide first evidence that sumoylation of Cebp-alpha (via SUMO1 (显示 SUMO1 抗体), SUMO2 (显示 SUMO2 抗体), and SUMO3 (显示 SUMO3 抗体)) might contribute to cell fate decision of myelo-erythroid progenitor cells in intermediate cell mass during primitive [extramedullary] hematopoiesis.
An evolutionarily conserved PTEN (显示 PTEN 抗体)-C/EBPalpha-CTNNA1 (显示 CTNNA1 抗体) axis controls myeloid development and transformation.
a C/EBP recognition sequence in the proximal promoter region of C/EBPalpha is essential for IL-6 (显示 IL6 抗体)-mediated repression
These results suggest that the CEBPA gene is a strong candidate gene that affects carcass traits in Qinchuan cattle.
Expressions of C-EBPalpha and myostatin (显示 MSTN 抗体) in muscles were higher in the concentrate-fed group than in the grass hay (显示 GTF2H5 抗体)-fed group.
C/EBPalpha is an essential regulatory factor for perilipin5 transcription and suggest that fasting stimulates perilipin5 transcription through influencing C/EBPalpha expression.
The differential expression of specific CEBPA/B isoforms observed in maturing follicles and CL may contribute to changes in follicular cell differentiation and increasing steroidogenic capacity.
The protein encoded by this intronless gene is a bZIP transcription factor which can bind as a homodimer to certain promoters and enhancers. It can also form heterodimers with the related proteins CEBP-beta and CEBP-gamma. The encoded protein has been shown to bind to the promoter and modulate the expression of the gene encoding leptin, a protein that plays an important role in body weight homeostasis. Also, the encoded protein can interact with CDK2 and CDK4, thereby inhibiting these kinases and causing growth arrest in cultured cells.
CCAAT/enhancer-binding protein alpha
, C/EBP alpha
, CAAT/enhancer-binding protein DNA-binding protein
, CAAT/enhancer-binding protein, DNA-binding protein
, CCAAT/enhancer binding protein, alpha
, c/EBP alpha
, CCAAT/enhancer binding protein alpha