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This locus encodes the transforming growth factor (TGF)-beta type III receptor. 再加上，我们可以发TGFBR3 抗体 (104) 和 TGFBR3 蛋白 (10)和数多这个蛋白质的别的产品。
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Inhibition of genes for betaglycan and FIBP (显示 FIBP ELISA试剂盒) in granulosa cells in vitro suggests that they inhibit estradiol production in regressing subordinate follicles.
describe a detailed characterization of TbetaRIII expression in lymphocyte subpopulations demonstrating that this co-receptor is significantly expressed in T but not B lymphocytes and among them, preferentially expressed on naive and central memory T cells
TGF-beta (显示 TGFB1 ELISA试剂盒) type I, II, and III receptors were all identified in pregnant serum; all were substantially elevated in early-onset but not late-onset PE. Endoglin (显示 ENG ELISA试剂盒) was increased in both subtypes.
TGFBR3 and/or MGEA5 (显示 MGEA5 ELISA试剂盒) rearrangements are much more common in hybrid hemosiderotic fibrolipomatous tumor-myxoinflammatory fibroblastic sarcomas than in classical myxoinflammatory fibroblastic sarcomas.
opposing functions for the different GAG modifications on TbetaRIII suggesting that Wnt (显示 WNT2 ELISA试剂盒) interactions with the TbetaRIII heparan sulfate chains result in inhibition of Wnt (显示 WNT2 ELISA试剂盒) signaling, likely via Wnt (显示 WNT2 ELISA试剂盒) sequestration, whereas the chondroitin sulfate GAG chains on TbetaRIII promote Wnt3a (显示 WNT3A ELISA试剂盒) signaling.
Results suggest that high expression levels of alpha-inhibin and beta-glycan transcripts in secretory phase endometrium are associated with a lower chance of achieving pregnancy with in vitro fertilization.
Results show decreased TbetaRIII expression with hepatocellullar carcinoma (HCC (显示 FAM126A ELISA试剂盒)) progression leading to the activation of Smad2 (显示 SMAD2 ELISA试剂盒) and suggest that TbetaRIII acts as a suppressive factor in regulating the migration and invasion of HCC (显示 FAM126A ELISA试剂盒), by inhibiting Smad2 (显示 SMAD2 ELISA试剂盒) and Akt (显示 AKT1 ELISA试剂盒) pathways.
Study shows that TbetaRIII expression is significantly decreased in salivary glands adenoid cystic carcinoma (ACC) patients and defines TbetaRIII as a biomarker exerting antitumor action on ACC progression.
No correlation of loss of heterozygosity at the TGFBR3 locus with clinicopathological parameters suggests that allelic imbalance may be an early genetic event during neoplastic transformation of human endometrium.
Study shows that GDF10 (显示 GDF10 ELISA试剂盒) is down-regulated in patients with oral squamous cell carcinoma, and is an independent risk factor for overall survival. Its expression is regulated by TGFBR3 which shares the signaling inhibiting epithelial-mesenchymal transition.
rs1192415 of TGFBR3 is associated with primary open angle glaucoma.
BMP2 (显示 BMP2 ELISA试剂盒) also requires Src (显示 SRC ELISA试剂盒) for filamentous actin polymerization in Tgfbr3(-/-) epicardial cells.
Study identified a novel major binding site for TGF-beta (显示 TGFB1 ELISA试剂盒)-like growth factors in TGFR-3. Findings potentially provide novel experimental insight into the general function of zona pellucida domain-containing proteins.
Retinoic acid enhanced TbetaRIII expression and that this increase contributed to LF-stimulated IgA production.
Pro-fibrotic cardiac effect of miR (显示 MLXIP ELISA试剂盒)-328 was mediated by down-regulating TGFbR3 and up-regulating TGFb1 (显示 TGFB1 ELISA试剂盒).
These data demonstrate that TbetaRIII regulates BMP-mediated signaling and biological effects, primarily through the ligand sequestration effects of sTbetaRIII in normal and cancerous mammary epithelial cells.
Glucocorticoids recruit Tgfbr3 and Smad1 (显示 SMAD1 ELISA试剂盒) to shift transforming growth factor-beta signaling from the Tgfbr1 (显示 TGFBR1 ELISA试剂盒)/Smad2 (显示 SMAD2 ELISA试剂盒)/3 axis to the Acvrl1 (显示 ACVRL1 ELISA试剂盒)/Smad1 (显示 SMAD1 ELISA试剂盒) axis in lung fibroblasts.
work reveals a 'seed and soil' mechanism where TGF-beta2 (显示 TGFB2 ELISA试剂盒) and TGF-beta (显示 TGFB1 ELISA试剂盒)-RIII signalling through p38alpha (显示 MAPK14 ELISA试剂盒)/beta regulates DTC dormancy and defines restrictive (BM) and permissive (lung) microenvironments for HNSCC metastasis
TGFBR3 mediated suppression of cancer progression includes effects on the tumor immune microenvironment.
Loss of Tgfbr3 compromises the functions of TGFbeta2 in the differentiating fetal testis.
This study provides evidence that the expressions of inhibin alpha subunit (显示 INHA ELISA试剂盒) and betaglycan are inferior in cystic follicles, and this may be caused by the decrease in FSH (显示 BRD2 ELISA试剂盒) in the presence of a cystic follicle.
This locus encodes the transforming growth factor (TGF)-beta type III receptor. The encoded receptor is a membrane proteoglycan that often functions as a co-receptor with other TGF-beta receptor superfamily members. Ectodomain shedding produces soluble TGFBR3, which may inhibit TGFB signaling. Decreased expression of this receptor has been observed in various cancers. Alternatively spliced transcript variants encoding different isoforms have been identified for this gene.
transforming growth factor, beta receptor III
, transforming growth factor, beta receptor III (betaglycan, 300kDa)
, transforming growth factor beta receptor type 3-like
, TGF-beta receptor type 3
, TGF-beta receptor type III
, betaglycan proteoglycan
, transforming growth factor beta receptor type 3
, transforming growth factor, beta receptor 3
, TGF-beta type III receptor
, transforming growth factor beta receptor III
, transforming growth factor-beta type III receptor