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activity; localization, and function is regulated by the C-terminal tail. 再加上，我们可以发Mitogen-Activated Protein Kinase 15 抗体 (86) 和 Mitogen-Activated Protein Kinase 15 蛋白 (8)和数多这个蛋白质的别的产品。
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CapZIP (显示 RCSD1 ELISA试剂盒), which has been shown to regulate ciliogenesis, is an ERK7 substrate, and that Dishevelled (显示 DVL2 ELISA试剂盒), which has also been shown to regulate ciliogenesis, facilitates ERK7 phosphorylation of CapZIP (显示 RCSD1 ELISA试剂盒) through binding to both ERK7 and CapZIP (显示 RCSD1 ELISA试剂盒).
In HeLa cells, phosphorylation of HuR (显示 ELAVL1 ELISA试剂盒) by ERK8 prevents it from binding to PDCD4 (显示 PDCD4 ELISA试剂盒) mRNA and allows miR (显示 MLXIP ELISA试剂盒)-21-mediated degradation of PDCD4 (显示 PDCD4 ELISA试剂盒).
depletion of endogenous MAPK15 expression inhibited BCR (显示 BCR ELISA试剂盒)-ABL1 (显示 ABL1 ELISA试剂盒)-dependent cell proliferation, in vitro
The present study suggests that MAPK15 overexpression may contribute to the malignant transformation of gastric mucosa by prolonging the stability of c-Jun (显示 JUN ELISA试剂盒).
ERK8 appears as a constitutive brake on N-Acetylgalactosaminyltransferase (显示 B4GALNT2 ELISA试剂盒) relocalisation, and the loss of its expression could drive cancer aggressivity through increased cell motility.
Data suggest that the model coulb be a tool for the development of specific ERK8 kinase inhibitors.
ATG8 (显示 GABARAPL2 ELISA试剂盒)-like proteins (MAP1LC3B (显示 MAP1LC3B ELISA试剂盒), GABARAP (显示 GABARAP ELISA试剂盒) and GABARAPL1 (显示 GABARAPL1 ELISA试剂盒)) are novel interactors of MAPK15/ERK8, a MAP kinase (显示 MAPK1 ELISA试剂盒) involved in cell proliferation and transformation.
a novel function for ERK8 as a bona fide ERRalpha (显示 ESRRA ELISA试剂盒) corepressor, involved in control of its cellular localization by nuclear exclusion, and suggest a key role for this MAP kinase (显示 MAPK1 ELISA试剂盒) in the regulation of the biological activities of this nuclear receptor.
Data show that ERK8 prevents HDM2-mediated PCNA (显示 PCNA ELISA试剂盒) destruction by inhibiting the association of PCNA (显示 PCNA ELISA试剂盒) with HDM2, and implicate ERK8 in the regulation of genomic stability.
Extracellular signal-regulated kinase 8-mediated c-Jun (显示 JUN ELISA试剂盒) phosphorylation increases tumorigenesis of human colon cancer
ERK8, a new member of the mitogen-activated protein kinase (显示 MAPK1 ELISA试剂盒) family.
Taken together, our results suggest that ERK8 plays an important role in spindle organization during mouse oocyte meiotic maturation and early embryo cleavage.
Erk8 has a role as a novel effector of RET (显示 RET ELISA试剂盒)/PTC3 (显示 NCOA4 ELISA试剂盒) and, therefore, RET (显示 RET ELISA试剂盒) biological functions
activity\; localization, and function is regulated by the C-terminal tail
mitogen-activated protein kinase 15
, extracellular signal-regulated kinase 7
, MAP kinase 15
, MAPK 15
, extracellular regulated kinase 8 delta
, extracellular signal regulated kinase 8
, extracellular signal-regulated kinase 8