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LGALS9 was initially thought to represent a pseudogene of galectin 9; however, this transcript has good exon-intron structure and encodes a predicted protein of the same size as and highly similar to galectin 9. 再加上，我们可以发Lectin, Galactoside-Binding, Soluble, 9 试剂盒 (58) 和 Lectin, Galactoside-Binding, Soluble, 9 蛋白 (35)和数多这个蛋白质的别的产品。
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Human Polyclonal LGALS9 Primary Antibody for ELISA, WB - ABIN561667
Bellac, Coimbra, Simon, Imboden, Leib: Gene and protein expression of galectin-3 and galectin-9 in experimental pneumococcal meningitis. in Neurobiology of disease 2007
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Mouse (Murine) Polyclonal LGALS9 Primary Antibody for CyTOF, FACS - ABIN4899254
Iqbal, Sampaio, Maione, Greco, Niki, Hirashima, Perretti, Cooper: Endogenous galectin-1 and acute inflammation: emerging notion of a galectin-9 pro-resolving effect. in The American journal of pathology 2011
in a model of experimental autoimmune encephalomyelitis, we show that an agonist of DR3 (显示 TNFRSF25 抗体) suppressed disease, correlating with expansion of CD4 (显示 CD4 抗体)+Foxp3 (显示 FOXP3 抗体)+ Tregs, and this protective effect was lost in Galectin-9-/- mice
Galectin 9 is a novel dectin 1 (显示 CLEC7A 抗体) ligand in pancreatic ductal adenocarcinoma. Upon disruption of the dectin 1 (显示 CLEC7A 抗体)-galectin 9 axis, CD4 (显示 CD4 抗体)(+) and CD8 (显示 CD8A 抗体)(+) T cells, which are dispensable for PDA progression in hosts with an intact signaling axis, become reprogrammed into indispensable mediators of anti-tumor immunity.
The Gal-9/Tim-3 (显示 HAVCR2 抗体) signal is important for the regulation of decidua NK cells function, which is beneficial for the maintenance of a normal pregnancy.
These results indicate the possibility that cooperative binding of oligosaccharide and neighboring polypeptide structures of monoclonal IgE to galectin-9 affects the overall affinity and specificity of the IgE-lectin interaction.
galectin-9 functions as a novel alarmin by augmenting the inflammatory response in sepsis development during pulmonary F. novicida infection.
Gal-9 has a role in the maintenance of hepatic homeostasis and preventing liver ischemia-reperfusion injury
Gal-9, a natural TIM-3 (显示 HAVCR2 抗体) ligand, was produced primarily by and released from ischemia-reperfusion-stressed hepatocytes, both in vivo and in vitro.
Galectin-9 functions as an astrocyte-microglia communication signal and promotes cytokine production from microglia in a Tim-3 (显示 HAVCR2 抗体) independent manner.
galectin-9, in addition to being an effector molecule for regulatory T cells cells, acts synergistically with TGF-beta (显示 TGFB1 抗体) to enforce iTreg cell differentiation and maintenance.
Galectin-9 is a suppressor of T and B cells and predicts the immune modulatory potential of mesenchymal stromal cell preparations.
Significantly elevated Gal-9 levels were found in both minimal-mild (I-II) and moderate-severe (III-IV) stages of endometriosis in comparison with healthy controls.
We investigated tumor-infiltrating CD4 (显示 CD4 抗体) and CD8 (显示 CD8A 抗体) T cells and the expression of PD-L1 (显示 CD274 抗体), Galectin-9, and XAGE1 (显示 XAGE1A 抗体) in stage I to IIIA lung adenocarcinomas using a tissue microarray to deduce their contribution to overall survival, and our data showed that PD-L1 (显示 CD274 抗体) expression was a positive indicator, whereas Galectin-9 and XAGE1 (显示 XAGE1A 抗体) expression was negative.
Tim3 (显示 HAVCR2 抗体)/Gal-9 alleviates the inflammation of TAO patients via suppressing Akt (显示 AKT1 抗体)/NF-kappaB (显示 NFKB1 抗体) signaling pathway.
Low Gal-9 expression is associated with pancreatic and ampullary cancer.
X-ray structure of a protease-resistant mutant form of human galectin-9 having two carbohydrate recognition domains with a metal-binding site has been presented.
These findings may indicate that an increase in the Gal-9 level, a novel immune checkpoint molecule, can reflect immune-related adverse effects of various biotherapies.
Our data suggest a role of galectin-9 in regulating HIV transcription and viral production in vivo during therapy
these data suggest that the high Tim-3 (显示 HAVCR2 抗体) expression in monocytes could be utilized by tumor-promoting Gal-9 expression on CD4 (显示 CD4 抗体)(+) T cells
this study shows that in osteosarcoma patients, Tim3 (显示 HAVCR2 抗体) expression did not directly mediate immune suppression, but the interaction between Tim3 (显示 HAVCR2 抗体)+ T cells and monocytes, naive CD4 (显示 CD4 抗体)+ T cells, and Gal9-expressing CD4 (显示 CD4 抗体)+ CD25 (显示 IL2RA 抗体)+ Tregs could resulting in progressive suppression of Th1 (显示 TH1L 抗体) responses
The Tim-3 (显示 HAVCR2 抗体)/Gal-9 pathway might link disease progression with T cell exhaustion during bovine leukemia virus infection.
The relative abundance of PIBF (显示 PIBF1 抗体), LGALS1 (显示 LGALS1 抗体), LGALS3 (显示 LGALS2 抗体), LGALS3BP (显示 LGALS3BP 抗体), and LGALS9 mRNA would display a differential expression pattern in the endometrium.
The galectins are a family of beta-galactoside-binding proteins implicated in modulating cell-cell and cell-matrix interactions. The protein encoded by this gene is an S-type lectin. It is overexpressed in Hodgkin's disease tissue and might participate in the interaction between the H&RS cells with their surrounding cells and might thus play a role in the pathogenesis of this disease and/or its associated immunodeficiency. Multiple alternatively spliced transcript variants have been found for this gene.
lectin, galactoside-binding, soluble, 9
, galactoside-binding soluble lectin 9
, galectin 9
, lectin, galactoside-binding, soluble, 9 (galectin 9)
, lectin, galactose binding, soluble 5
, tumor antigen HOM-HD-21
, urate transporter/channel protein
, 36 kDa beta-galactoside-binding lectin
, Lectin galactose binding soluble 9 (Galectin-9)
, Lectin, galactose binding, soluble 9 (Galectin-9)
, urate transporter/channel
, urate transporter
, urate transporter/channel protein, isoform (UATp,i)