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Kruppel-like factors (KLFs) are a family of broadly expressed zinc finger transcription factors. 再加上，我们可以发KLF2 试剂盒 (9) 和 KLF2 蛋白 (6)和数多这个蛋白质的别的产品。
Showing 10 out of 111 products:
Guinea Pig Polyclonal KLF2 Primary Antibody for ChIP, IHC - ABIN2777573
Saavedra, Patterson, West, Randell, Riches, Malcolm, Cool, Nick, Dinarello: Abrogation of anti-inflammatory transcription factor LKLF in neutrophil-dominated airways. in American journal of respiratory cell and molecular biology 2008
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Human Polyclonal KLF2 Primary Antibody for IF (p), IHC (p) - ABIN680473
Wang, Wang, Shen, Yin, Tang: Effects of artesunate and ursolic acid on hyperlipidemia and its complications in rabbit. in European journal of pharmaceutical sciences : official journal of the European Federation for Pharmaceutical Sciences 2013
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Human Polyclonal KLF2 Primary Antibody for IHC, IHC (p) - ABIN4329130
Ferraro, Mafalda Lopes da, Grimes, Lee, Ketteler, Kriston-Vizi, Cutler: Weibel-Palade body size modulates the adhesive activity of its von Willebrand Factor cargo in cultured endothelial cells. in Scientific reports 2016
Human Monoclonal KLF2 Primary Antibody for ELISA, WB - ABIN523760
Richardson, Jadlowsky, Didigu, Doms, Riley: Kruppel-like factor 2 modulates CCR5 expression and susceptibility to HIV-1 infection. in Journal of immunology (Baltimore, Md. : 1950) 2012
Human Monoclonal KLF2 Primary Antibody for FACS, ICC - ABIN4900476
Seifert, Sellmann, Bloehdorn, Wein, Stilgenbauer, Dürig, Küppers: Cellular origin and pathophysiology of chronic lymphocytic leukemia. in The Journal of experimental medicine 2012
Human Polyclonal KLF2 Primary Antibody for WB - ABIN1538992
Gan, Dang, Han, Ling, Duan, Liu, Xu: ERK5/HDAC5-mediated, resveratrol-, and pterostilbene-induced expression of MnSOD in human endothelial cells. in Molecular nutrition & food research 2015
Human Polyclonal KLF2 Primary Antibody for WB - ABIN2780291
Basu, Morris, Haar, Wani, Lingrel, Gaensler, Lloyd: KLF2 is essential for primitive erythropoiesis and regulates the human and murine embryonic beta-like globin genes in vivo. in Blood 2005
Transcription factor Kruppel-like factor 2 (KLF2) of the zinc finger family of DNA-binding proteins has recently emerged as a major molecular switch that controls endothelial homeostasis.
The findings point to KLF2 as a new gene involved in heritable pulmonary arterial hypertension.
we provide evidence that the tumor-suppressive microRNA miR (显示 MLXIP 抗体)-1825 controls KLF2 expression. Reporter gene analyses revealed that both microRNAs directly targeted the 3'-untranslated region of KLF2 messenger RNA. These data demonstrated that miR (显示 MLXIP 抗体)-1825 expression in serum of human glioma was associated with tumorigenesis and miR (显示 MLXIP 抗体)-1825 may be used as a biomarker for identification of the pathological grade of glioma.
Long noncoding RNA XIST acts as an oncogene (显示 RAB1A 抗体) in non-small cell lung cancer by epigenetically repressing KLF2 expression.
lysine-specific demethylase (显示 MBD2 抗体)-1 is an important oncogene (显示 RAB1A 抗体) in gastric cancer, and lysine-specific demethylase (显示 MBD2 抗体)-1-mediated epigenetic repression of KLF2 plays a critical role in gastric cancer development and progression.
KLF2 is shown to be downregulated in pancreatic ductal adenocarcinoma (PDAC) clinical samples and overexpression of KLF2 inhibits the growth, migration, and metastasis of PDAC cancer cells.
KLF2 expression was decreased in gastric cancer and negatively correlated with lymphatic metastasis. KLF2 overexpression inhibited cell proliferation and invasive potential and downregulated the protein expression of PCNA (显示 PCNA 抗体), Bcl-2 (显示 BCL2 抗体) and MMP-9 (显示 MMP9 抗体) in GC cells. KLF2 overexpression reduced xenograft tumor growth.
our data highlight an important role for KLF2/c-myc (显示 MYC 抗体) pathway in HCC (显示 FAM126A 抗体) development and progression.
Authors show that KLF2 induces the expression of CRABP2 (显示 CRABP2 抗体) and RARgamma and inhibits the expression FABP5 (显示 FABP5 抗体) and PPARbeta (显示 PPARD 抗体)/delta thereby shifting RA signaling from the pro-carcinogenic FABP5 (显示 FABP5 抗体)/PPARbeta (显示 PPARD 抗体)/delta to the growth-suppressing CRABP2 (显示 CRABP2 抗体)/RAR (显示 RARA 抗体) path.
MicroRNA-30 mediates anti-inflammatory effects of shear stress and KLF2 via repression of Ang2 (显示 ANGPT2 抗体).
miR (显示 MYLIP 抗体)-92a coregulates KLF4 (显示 KLF4 抗体) and KLF2 expression in arterial endothelium and contributes to phenotype heterogeneity associated with regional atherosusceptibility and protection in vivo.
Mouse embryos deficient in Orai1 (显示 TMEM132A 抗体), Klf2, or Klf4 (显示 KLF4 抗体) showed a significantly reduced lymphatic density and impaired lymphatic development.
this study shows that KLF2 governs the degree of dendritic cells activation and hence the intensity of proatherogenic T cell responses
A deficiency of KLF2 increased the expression of Notch ligand (显示 JAG2 抗体) Jagged2 (显示 JAG2 抗体) via hypoxia-inducible factor 1alpha (HIF-1alpha (显示 HIF1A 抗体)), which led to Th2 amplification.
our data demonstrate a protective role of KLF2 against glomerular endothelial cell injury and progression of chronic kidney disease in the model of compensatory renal hypertrophy
Under steady-state conditions, KLF2-deficient NK cells alter their expression of homeostatic homing receptors and subsequently undergo apoptosis due to IL-15 (显示 IL15 抗体) starvation.
KLF2 knockdown markedly reduced expression of miR (显示 MLXIP 抗体)-155 in macrophages and macrophage activation.
study therefore establishes the pivotal role of Klf2 and Tfcp2l1 (显示 TFCP2L1 抗体) in mediating ESC self-renewal promoted by Wnt (显示 WNT2 抗体)/beta-catenin (显示 CTNNB1 抗体) signaling.
endothelial-specific loss of Mekk3 (显示 MAP3K3 抗体), Klf2 or Klf4 (显示 KLF4 抗体) markedly prevents cerebral cavernous malformation lesion formation, reverses the increase in Rho activity, and rescues lethality
knockdown of Klf2 expression in endothelial cells induced more endothelial cell injury in early diabetic nephropathy
KLF2 and ERG (显示 ERG 抗体) associate in a physical complex and the two proteins synergistically activate transcription of Flk1 (显示 KDR 抗体).
Kruppel-like factors (KLFs) are a family of broadly expressed zinc finger transcription factors. KLF2 regulates T-cell trafficking by promoting expression of the lipid-binding receptor S1P1 (S1PR1\; MIM 601974) and the selectin CD62L (SELL\; MIM 153240) (summary by Weinreich et al., 2009
Kruppel-like factor 2 (lung)
, Krueppel-like factor 2
, Kruppel-like factor LKLF
, lung Kruppel-like zinc finger transcription factor
, lung krueppel-like factor
, lung Kruppel-like factor