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FBP2 encodes a gluconeogenesis regulatory enzyme which catalyzes the hydrolysis of fructose 1,6-bisphosphate to fructose 6-phosphate and inorganic phosphate. 再加上，我们可以发Fructose-1,6-Bisphosphatase 2 蛋白 (11) 和 Fructose-1,6-Bisphosphatase 2 试剂盒 (6)和数多这个蛋白质的别的产品。
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IFN-gamma (显示 IFNG 抗体) production was increased in spleen cells of KSRP (显示 KHSRP 抗体)(-/-) mice.
Resveratrol causes a shift from the mesenchymal-specific forms of these factors to the respective epithelial forms and increases the expression of the RNA-binding proteins KHSRP (显示 KHSRP 抗体) and hnRNPA1 (显示 HNRNPA1 抗体).
KSRP (显示 KHSRP 抗体) is critical in governing hepatic lipid metabolism through controlling Per2 (显示 PER2 抗体) expression
KSRP (显示 KHSRP 抗体) modulation of GAP-43 (显示 GAP43 抗体) mRNA stability restricts axonal outgrowth in embryonic hippocampal neurons.
This study reveals the in vivo function of KSRP (显示 KHSRP 抗体) in controlling brown-like transformation of subcutaneous/inguinal white adipose tissue through post-transcriptional regulation of miR (显示 MLXIP 抗体)-150 expression.
This study reveals a novel in vivo function of KSRP (显示 KHSRP 抗体) in controlling adipose lipolysis through posttranscriptional regulation of miR (显示 MLXIP 抗体)-145 expression.
KSRP (显示 KHSRP 抗体) is required to maintain P19 (显示 CDKN2D 抗体) cells in undifferentiated state and that its inactivation can orient cells towards neural differentiation.
Our results outline a critical role for KSRP (显示 KHSRP 抗体) in regulating pro-inflammatory mediators and have implications for a wide range of CNS inflammatory and autoimmune diseases.
We have identified a post-transcriptional regulatory network active during mouse pituitary development in which the expression of the hormone alphaGSU (显示 CGA 抗体) is increased by let7b/c through downregulation of KSRP (显示 KHSRP 抗体)
data suggest that miR (显示 MLXIP 抗体)-27b targets KSRP (显示 KHSRP 抗体) and modulates iNOS (显示 NOS2 抗体) mRNA stability following C. parvum infection, a process that may be relevant to the regulation of epithelial anti-microbial defense in general
Results demonstrate that truncation of the evolutionarily conserved N-terminal residues of FBP2 (显示 KHSRP 抗体) results in a loss of its mitochondria-protective functions.
FBP2 (显示 KHSRP 抗体) does negatively regulate cell growth, and reduced expression of FBP2 (显示 KHSRP 抗体) may contribute to carcinogenesis for gastric cancer.
The key role in strong AMP (显示 APRT 抗体) binding to muscle isozyme play K20 (显示 KRT20 抗体), T177 and Q179.
The existence of highly AMP (显示 APRT 抗体)-sensitive muscle-like FBPase (显示 FBP1 抗体), activity of which is regulated by metabolite-dependent interaction with aldolase (显示 ALD 抗体) enables the precise regulation of muscle energy expenditures.
This gene encodes a gluconeogenesis regulatory enzyme which catalyzes the hydrolysis of fructose 1,6-bisphosphate to fructose 6-phosphate and inorganic phosphate.
, fructose-1,6-bisphosphatase isozyme 2
, D-fructose-1,6-bisphosphate 1-phosphohydrolase 2
, FBPase 2
, FBPase muscle
, muscle fructose-bisphosphatase
, fructose bisphosphatase 2
, FUSE-binding protein 2
, KH type-splicing regulatory protein
, far upstream element-binding protein 2