Forkhead Box G1 (FOXG1) ELISA试剂盒

This locus encodes a member of the forked-head transcription factor family. 再加上,我们可以发FOXG1 抗体 (87)FOXG1 蛋白 (8)和数多这个蛋白质的别的产品。

list all ELISA KIts 基因 基因ID UniProt
 FOXG1 FOXG1 2290 P55316
小鼠 FOXG1 FOXG1 15228 Q60987
大鼠 FOXG1 FOXG1 24370 Q00939
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适于 FOXG1 相互作用对的更多 ELISA 试剂盒

Zebrafish Forkhead Box G1 (FOXG1) interaction partners

  1. Results suggest that foxg1b and foxg1c have undergone expression pattern divergence during evolution that has resulted in functional specialization.

  2. These findings identify a key direct target of Foxg1, and uncover a simple molecular mechanism by which Foxg1 integrates two opposing signaling centers.

Human Forkhead Box G1 (FOXG1) interaction partners

  1. The genetic etiology of Rett syndrome (RTT) without MECP2, CDKL5 (显示 CDKL5 ELISA试剂盒), and FOXG1 mutations is heterogeneous, overlaps with other NDDs, and complicated by a high mutation burden. Dysregulation of chromatin structure and abnormal excitatory synaptic signaling may form two common pathological bases of RTT.

  2. FOXG1 and SOX2 (显示 SOX2 ELISA试剂盒) operate in complementary but distinct roles to fuel unconstrained self-renewal in Glioblastoma multiforme stem cells via transcriptional control of core cell cycle and epigenetic regulators.

  3. phenotypes associated with FOXG1 mutations in Chinese Rett syndrome or Rett syndrome-like patients.

  4. describe the initial design and characterizations of novel covalent BH3-based agents that potently target Bfl-1 (显示 BCL2A1 ELISA试剂盒)

  5. findings demonstrate clear phenotype differences between FOXG1 and MECP2 disorders.

  6. Abnormal involuntary movements are a major feature of FOXG1 mutations. Our study delineates the spectrum of movement disorders and confirms an expanding clinical phenotype. Symptomatic treatment may be considered for severe or disabling cases, although further research regarding potential treatment strategies is necessary.

  7. Report demonstrates the functional consequences of Foxg1 haploinsufficiency in the visual system of Foxg1+/Cre mice and a visual impairment in a cohort of Rett individuals presenting genetic alteration on FOXG1

  8. Upregulated miR (显示 MLXIP ELISA试剂盒)-200b in cervical cancer was proven to show positive regulation on cervical cancer development by directly targeting FoxG1.

  9. Rett syndrome with early epilepsy and the congenital variant are mainly due to variations in the CDKL5 (显示 CDKL5 ELISA试剂盒) and FOXG1 genes, respectively

  10. FOXG1 mutations are associated with familial recurrence in FOXG1-related disorders.

Mouse (Murine) Forkhead Box G1 (FOXG1) interaction partners

  1. Foxg1 was found to play a role in promoting the closure of optic fissure.

  2. Report demonstrates the functional consequences of Foxg1 haploinsufficiency in the visual system of Foxg1+/Cre mice and a visual impairment in a cohort of Rett individuals presenting genetic alteration on FOXG1

  3. The results presented here indicate that loss of Dlx5 (显示 DLX5 ELISA试剂盒) causes a down-modulation of miR (显示 MLXIP ELISA试剂盒)-9 and of miR (显示 MLXIP ELISA试剂盒)-200-class, which results in the over-expression of the Foxg1 protein.

  4. Foxg1-Cre mediated Lrp2 (显示 LRP2 ELISA试剂盒) inactivation in developing mouse neural retina, ciliary and retinal pigment epithelia is a model of congenital high myopia

  5. findings suggest that different subcellular localizations of Foxg1 control the machinery that brings about cell differentiation, replication, and bioenergetics, possibly linking mitochondrial functions to embryonic development and pathological conditions

  6. EGFR (显示 EGFR ELISA试剂盒) mutations remodel the activated enhancer landscape of glioblastoma multiforme, promoting tumorigenesis through a SOX9 (显示 SOX9 ELISA试剂盒) and FOXG1-dependent transcriptional regulatory network in vitro and in vivo.

  7. This study demonistrated that Celsr3 (显示 CELSR3 ELISA试剂盒)/Foxg1 mutation mice show the spinal motor network does not mature fully in the absence of corticofugal connections, and that some motor function is preserved despite congenital absence of the corticospinal tract.

  8. Foxg1 as a key coordinator of the early transcriptional network during the course of cortical development.

  9. target of Wnt (显示 WNT2 ELISA试剂盒)/beta-catenin (显示 CTNNB1 ELISA试剂盒) signalling, displayed significant upregulation of this pathway in Foxg1(-/-) nulls at embryonic days 12.5 and 14.5

  10. Targets of RBPJ (显示 RBPJ ELISA试剂盒)/N1ICD in cortical neural stem cell at a genome-wide level, were identified

FOXG1 抗原简介

Antigen Summary

This locus encodes a member of the forked-head transcription factor family. The encoded protein, which functions as a repressor, may play a role in brain development. Mutations at this locus have been associated with Rett syndrome.

Gene names and symbols associated with Forkhead Box G1 (FOXG1) ELISA试剂盒

  • forkhead box G1a (foxg1a) 抗体
  • forkhead box G1 (foxg1) 抗体
  • brain factor 1 (bf-1) 抗体
  • forkfead transcription factor G1 (Foxg1) 抗体
  • forkhead box G1b (foxg1b) 抗体
  • forkhead box G1 (FOXG1) 抗体
  • forkhead box G1 L homeolog (foxg1.L) 抗体
  • forkhead box G1 (Foxg1) 抗体
  • 2900064B05Rik 抗体
  • bf-1 抗体
  • Bf1 抗体
  • BF1A 抗体
  • BF2 抗体
  • BmFOXG1 抗体
  • CBF-1 抗体
  • FHKL3 抗体
  • FKH2 抗体
  • Fkhl1 抗体
  • FKHL2 抗体
  • FKHL3 抗体
  • FKHL4 抗体
  • FOXG1 抗体
  • FOXG1A 抗体
  • FOXG1B 抗体
  • foxg1b-a 抗体
  • FOXG1C 抗体
  • HBF-1 抗体
  • HBF-2 抗体
  • HBF-3 抗体
  • HBF-G2 抗体
  • HBF2 抗体
  • Hfh9 抗体
  • Hfhbf1 抗体
  • HFK1 抗体
  • HFK2 抗体
  • HFK3 抗体
  • KHL2 抗体
  • QIN 抗体
  • RATBF1A 抗体
  • XBF-1 抗体
  • XBf1 抗体
  • zgc:85969 抗体
  • zgc:109850 抗体

Protein level used designations for Forkhead Box G1 (FOXG1) ELISA试剂盒

brain factor 1 , forkhead box G1 , forkhead box protein G1 , forkfead transcription factor G1 , brain factor 2 , forkhead-like 1 , forkhead-like 2 , forkhead-like 3 , forkhead-like 4 , oncogene QIN , BF-1 , FKH-4 , forkhead protein 4 , winged-helix transcription factor , xFKH4 , xbf1 , HNF-3/forkhead homolog, brain factor 1 , forkhead-related protein FKHL1 , BF1 , forkhead-like transcription factor BF-1 , transcription factor BF-1 , CEQ 3-1 , N-62-5 , brain factor-1 , proto-oncogene C-QIN

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