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High-affinity epithelial cell surface receptor for APF. 再加上，我们可以发CKAP4 蛋白 (5) 和 CKAP4 试剂盒 (1)和数多这个蛋白质的别的产品。
Showing 10 out of 83 products:
Human Polyclonal CKAP4 Primary Antibody for ICC, IF - ABIN439792
Agemy, Kotamraju, Friedmann-Morvinski, Sharma, Sugahara, Ruoslahti: Proapoptotic peptide-mediated cancer therapy targeted to cell surface p32. in Molecular therapy : the journal of the American Society of Gene Therapy 2013
Human Polyclonal CKAP4 Primary Antibody for ICC, IF - ABIN439793
Mandal, Mandal, Park: Global quantitative proteomics reveal up-regulation of endoplasmic reticulum stress response proteins upon depletion of eIF5A in HeLa cells. in Scientific reports 2016
Human Polyclonal CKAP4 Primary Antibody for IHC, IHC (p) - ABIN4298845
Ek, Andréasson, Hober, Kampf, Pontén, Uhlén, Merz, Borrebaeck: From gene expression analysis to tissue microarrays: a rational approach to identify therapeutic and diagnostic targets in lymphoid malignancies. in Molecular & cellular proteomics : MCP 2006
Cow (Bovine) Polyclonal CKAP4 Primary Antibody for IHC, WB - ABIN2782739
Ko, McNiff, Glusac: Squamous cell carcinomas with single cell infiltration: a potential diagnostic pitfall and the utility of MNF116 and p63. in Journal of cutaneous pathology 2008
Show all 2 Pubmed References
CKAP4 has a role as a Dickkopf1 (显示 DKK1 抗体) receptor and in pancreatic and lung tumor progression
Human Prostate Basal cell hyperplasia is an expansion of p63 (显示 RPE65 抗体).
APF binds specifically to sites within the cytoskeleton-associated protein 4 (CKAP4) extracellular domain
CLIMP-63 (also known as CKAP4), is the partner of triadin (显示 TRDN 抗体), is responsible for this association of triads and microtubules.
The still rudimentary information of how CLIMP-63 fulfills these different roles, what these are exactly and how post-translational modifications control them, will be discussed.
Although VIMP (显示 SELS 抗体) can interact with CLIMP-63 and Syn5L, it does not interact with MT-binding ER proteins (such as Reep1 (显示 REEP1 抗体)) that shape the tubular smooth ER
revealed that CKAP4 could associate with EGFR (显示 EGFR 抗体) at basal status and the complex was reduced upon EGF (显示 EGF 抗体) stimulation, leading to release EGFR (显示 EGFR 抗体) into cytoplasm
Single nucleotide polymorphisms in p63 (显示 RPE65 抗体) are implicated in the etiology of nonsyndromic bladder-exstrophy-epispadias complex.
Here we report that the combination of p63 (显示 RPE65 抗体), a master regulator of epidermal development and differentiation, and KLF4 (显示 KLF4 抗体), a regulator of epidermal differentiation, is sufficient to convert dermal fibroblasts to a keratinocyte phenotype.
CKAP4 was correlated with favorable clinical outcome and was an independent predictor for overall survival in hepatic cholangiocarcinoma patients.
p63 positively regulates desmosome adhesion by directly controlling the expression of several desmosome genes, including Dsp (显示 DSP 抗体), Dsc3 (显示 DSC3 抗体) and Dsg1 (显示 DSG1 抗体).
Thus p63 closely interacts with SP-A (显示 SFTPA1 抗体) and may play a role in the trafficking or the biological function of the surfactant protein.
High-affinity epithelial cell surface receptor for APF. Mediates the anchoring of the endoplasmic reticulum to microtubules.
cytoskeleton-associated protein 4
, 63 kDa membrane protein
, 63-kDa cytoskeleton-linking membrane protein
, transmembrane protein (63kD), endoplasmic reticulum/Golgi intermediate compartment
, type-II transmembrane protein p63
, late passage cDNA-1