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The protein encoded by BPIFB1 may be involved in the innate immune response to bacterial exposure in the mouth, nasal cavities, and lungs. 再加上，我们可以发BPI Fold Containing Family B, Member 1 蛋白 (7)和数多这个蛋白质的别的产品。
Showing 10 out of 48 products:
Human Monoclonal BPIFB1 Primary Antibody for ICC, FACS - ABIN969534
Larocque, Sabeti, Duggal, Chowdhury, Khan, Lebrun, Harris, Ryan, Qadri, Calderwood: A variant in long palate, lung and nasal epithelium clone 1 is associated with cholera in a Bangladeshi population. in Genes and immunity 2009
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Mouse (Murine) Monoclonal BPIFB1 Primary Antibody for ELISA, WB - ABIN1098135
Tang, Li, Tang, Li, Lin, Martin, Grant, Solloway, Parker, Ye, Forrest, Ghilardi, Oravecz, Platt, Rice, Hansen, Abuin, Eberhart, Godowski, Holt, Peterson, Zambrowicz, de Sauvage: A mouse knockout library for secreted and transmembrane proteins. in Nature biotechnology 2010
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Human Polyclonal BPIFB1 Primary Antibody for WB - ABIN530118
Liao, Zeng, Guo, Li, Wei, Zhang, Li, Chen, Liang, Xiang, Ma, Wu, Tang, Deng, Zeng, Tang, Xiong, Li: LPLUNC1 suppresses IL-6-induced nasopharyngeal carcinoma cell proliferation via inhibiting the Stat3 activation. in Oncogene 2014
The G allele of rs1078761 may be detrimental to lung function in cystic fibrosis (显示 S100A8 抗体) owing to decreased levels of BPIFA1 (显示 BPIFA1 抗体) and BPIFB1.
higher sputum concentrations of BPIFB1 were associated with changes of lung function over time, especially in current smokers with chronic obstructive pulmonary disease. BPIFB1 may be involved in the pathogenesis of smoking-related lung diseases.
Autoimmunity targeting the lung-specific antigen BPIFB1 may contribute to the pathogenesis of interstitial lung disease.
Results suggest that LPLUNC1 can inhibit inflammation and nasopharyngeal carcinoma (NPC (显示 NPC1 抗体)) growth by downregulating the Stat3 (显示 STAT3 抗体) pathway.
LPLUNC1 delays nasopharyngeal carcinoma cell growth by inhibiting the MAP Kinase (显示 MAPK1 抗体) and cyclin D1 (显示 CCND1 抗体)/E2F (显示 E2F1 抗体) pathways.
It was shown that there was a significant increase in LPLUNC1 in the airways of patients with cystic fibrosis (显示 S100A8 抗体), but it is unclear if this increase influences the development of lung disease.
These results demonstrate that LPLUNC1 is expressed in Paneth cells and likely plays a role in modulating host inflammatory responses to Vibrio cholerae infection.
LPLUNC1 is an abundant, secreted product of goblet cells and minor mucosal glands of the respiratory tract and oral cavity and suggest that the protein functions in the complex milieu that protects the mucosal surfaces in these locations.
LPLUNC1 is found to be down-expressed in 33 of 48 nasopharyngeal carcinoma biopsies.
We found a significant association of cholera with a marker in the promoter region of LPLUNC1 (rs11906665), a member of a family of evolutionarily conserved innate immunity proteins
Bpifb1 mRNA is upregulated in parotid acinar cells in NOD mice, but its expression is not related to the onset of diabetes. These findings suggest that high expression levels of Bpifb1 might predict disease traits before the onset of autoimmunity.
CETP (显示 CETP 抗体) inhibitor torcetrapib promotes reverse cholesterol transport in obese insulin (显示 INS 抗体)-resistant CETP (显示 CETP 抗体)-ApoB100 (显示 APOB 抗体) transgenic mice
a novel gene LPlunc1, a member of BPI (显示 BPI 抗体)/PLUNC (显示 BPIFA1 抗体) gene superfamily, which is asymmetrically expressed in the developing mouse node [LPlunc1 protein]
The protein encoded by this gene may be involved in the innate immune response to bacterial exposure in the mouth, nasal cavities, and lungs. The encoded protein is secreted and is a member of the BPI/LBP/PLUNC protein superfamily. This gene is found with other members of the superfamily in a cluster on chromosome 20.
BPI fold-containing family B member 1
, long palate, lung and nasal epithelium carcinoma associated 1
, long palate, lung and nasal epithelium carcinoma-associated protein 1
, von Ebner minor salivary gland protein
, von Ebner minor salivary protein
, fold containing family B, member 1