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AGGF1 encodes an angiogenic factor that promotes proliferation of endothelial cells. 再加上，我们可以发AGGF1 试剂盒 (30) 和 AGGF1 蛋白 (8)和数多这个蛋白质的别的产品。
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Human Polyclonal AGGF1 Primary Antibody for ICC, IF - ABIN151902
Tian, Kadaba, You, Liu, Timur, Yang, Chen, Szafranski, Rao, Wu, Housman, DiCorleto, Driscoll, Borrow, Wang: Identification of an angiogenic factor that when mutated causes susceptibility to Klippel-Trenaunay syndrome. in Nature 2004
Bmp-induced activation of beta-catenin (显示 CTNNB1 抗体) through Aggf1 regulates caudal (显示 CAD 抗体) vein development by promoting the Nr2f2 (显示 NR2F2 抗体)-dependent differentiation of venous endothelial cells and their survival.
aggf1 is involved in differentiation of both hematopoietic and endothelial lineages and acts upstream of scl (显示 TAL1 抗体), fli1 (显示 FLI1 抗体), and etsrp (显示 ETV2 抗体) in specification of hemangioblasts
Our study establishes AGGF1 as an angiogenic factor (显示 VEGFA 抗体) with an important role in the specification of vein identity and suggests that AGGF1-mediated AKT (显示 AKT1 抗体) signaling is responsible for establishing venous cell fate
Aggf1 has a role in regulating vascular injury.
High expression of AGGF1 predicts poor prognosis in gastric cancer patients.
AGGF1 gene polymorphism does not affect the risk of varicose veins of the legs in ethnic Russians.
Overexpression of AGGF1 is correlated with angiogenesis in hepatocellular carcinoma.
AGGF1 reduces myocardial apoptosis and inflammation and enhances angiogenesis, leading to decreased infarct size after I/R injury.
results of this study indicate that hypoxia down-regulates expression of the AGGF1 protein, but not AGGF1 mRNA, by inducing expression of miR (显示 MLXIP 抗体)-27a
AGGF1 potently attenuated TNF-alpha (显示 TNF 抗体) triggered NF-kappaB (显示 NFKB1 抗体) pathway, as indicated by the decreased promoter activity, nuclear distribution and phosphorylation of NF-kappaB (显示 NFKB1 抗体) p65 (显示 GORASP1 抗体) subunit as well as the increased protein level of IkappaBalpha (显示 NFKBIA 抗体).
analysis of sphingolipid modulation of angio (显示 VEGFA 抗体)genic factor expression in n (显示 ARHGEF16 抗体)euroblastoma
results define VG5Q as an angiogenic factor (显示 VEGFA 抗体), establish VG5Q as a susceptibility gene for Klippel-Trenaunay syndrome, and show that increased angiogenesis is a molecular pathogenic mechanism of KTS
VG5Q, E133K is a mutation that causes Klippel-Trenaunay and other overgrowth syndromes.
Together with recent identification of somatic mutations in p110a (encoded by PIK3CA (显示 PIK3CA 抗体)), our data establish a potential mechanistic link between AGGF1 and PIK3CA (显示 PIK3CA 抗体), the two genes identified for Klippel-Trenaunay syndrome (KTS)
Data indicate that angiogenic factor (显示 VEGFA 抗体) Aggf1+/- knockout (KO) mice show reduced autophagy, which was associated with inhibition of angiogenesis and larger infarct areas.
Mechanistically, Aggf1 activated the expression of gluconeogenesis gene and skewed the insulin (显示 INS 抗体) signaling pathway to induce insulin (显示 INS 抗体) resistance. Taken together, our data suggest that Aggf1 plays a role in steatosis in vivo and as such may be a new target in the development of therapeutics solutions against steatosis.
Sphingolipid modulation of angiogenic factor (显示 VEGFA 抗体) expression in neuroblastoma (显示 ARHGEF16 抗体)
This gene encodes an angiogenic factor that promotes proliferation of endothelial cells. Mutations in this gene are associated with a susceptibility to Klippel-Trenaunay syndrome. Pseudogenes of this gene are found on chromosomes 3, 4, 10 and 16.
angiogenic factor with G patch and FHA domains 1
, angiogenic factor VG5Q
, G patch domain-containing protein 7
, vasculogenesis gene on 5q protein